Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Ivan C. Alcantara"'
Autor:
Lukas Anneser, Anja Gemmer, Tim Eilers, Ivan C. Alcantara, Anett-Yvonn Loos, Soojin Ryu, Erin M. Schuman
Publikováno v:
iScience, Vol 25, Iss 3, Pp 103868- (2022)
Summary: Behavior is context-dependent and often modulated by an animal’s internal state. In particular, different social contexts can alter anxiety levels and modulate social behavior. The vertebrate-specific neuropeptide parathyroid hormone 2 (pt
Externí odkaz:
https://doaj.org/article/77826a777e504b769292716e133ccbd7
Publikováno v:
Nature Metabolism. 4:836-847
Publikováno v:
Nature
Species that depend on membership in social groups for survival exhibit changes in neuronal gene expression and behaviour when they face restricted social interactions or isolation1–3. Here we show that, across the lifespan of zebrafish (Danio reri
Publikováno v:
PLoS ONE, Vol 11, Iss 12, p e0167963 (2016)
The NIH Undiagnosed Diseases Program admitted a male patient with unclassifiable late-onset ataxia-like symptoms. Exome sequencing revealed a heterozygous de novo mutation converting glycine 316 to serine in ATP1A3, which might cause disease. ATP1A3
Externí odkaz:
https://doaj.org/article/3e9661f72b414ab2ae561c2012a34499
Publikováno v:
Nature metabolism. 4(7)
The overconsumption of highly caloric and palatable foods has caused a surge in obesity rates in the past half century, thereby posing a healthcare challenge due to the array of comorbidities linked to heightened body fat accrual. Developing treatmen
Autor:
Anja Gemmer, Anett-Yvonn Loos, Lukas Anneser, Soojin Ryu, Tim Eilers, Ivan C. Alcantara, Erin M. Schuman
SummaryAnimal behavior is strongly context-dependent and behavioral performance is often modulated by internal state. In particular, different social contexts can alter anxiety levels and modulate social behavior. The vertebrate-specific neuropeptide
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::84fa2eddc896735c9700497497df0262
https://doi.org/10.1101/2021.07.28.454115
https://doi.org/10.1101/2021.07.28.454115
Publikováno v:
PLoS ONE
PLoS ONE, Vol 11, Iss 12, p e0167963 (2016)
PLoS ONE, Vol 11, Iss 12, p e0167963 (2016)
The NIH Undiagnosed Diseases Program admitted a male patient with unclassifiable late-onset ataxia-like symptoms. Exome sequencing revealed a heterozygous de novo mutation converting glycine 316 to serine in ATP1A3, which might cause disease. ATP1A3