Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Isaí Martínez-Torres"'
Autor:
Azael Torales-Cardeña, Isaí Martínez-Torres, Sandra Rodríguez-Martínez, Fernando Gómez-Chávez, Juan C. Cancino-Díaz, Ernesto A. Vázquez-Sánchez, Mario E. Cancino-Díaz
Publikováno v:
Mediators of Inflammation, Vol 2015 (2015)
Psoriasis is a chronic inflammatory skin disease where the altered regulation in angiogenesis, inflammation, and proliferation of keratinocytes are the possible causes of the disease, and the transcription factor “hypoxia-inducible factor 1-alpha
Externí odkaz:
https://doaj.org/article/c3b7b83da11543fb8bb11daaf00e31cd
Autor:
Isaí Martínez-Torres, Araceli Tepale-Segura, Octavio Castro-Escamilla, Juan Carlos Cancino-Diaz, Sandra Rodríguez-Martínez, Sonia Mayra Perez-Tapia, Laura C. Bonifaz, Mario Eugenio Cancino-Diaz
Publikováno v:
International Journal of Molecular Sciences; Volume 23; Issue 9; Pages: 5226
Psoriasis is a chronic inflammatory disease distinguished by an excessive proliferation and abnormal differentiation of keratinocytes. Immune cells, such as T lymphocytes and neutrophils, and inflammatory cytokines, such as Tumor Necrosis Factor-α (
Autor:
Mario E. Cancino-Diaz, Sandra Rodríguez-Martínez, Fernando Gómez-Chávez, Edith Sierra-Mondragon, Isaí Martínez-Torres, Martín Murrieta-Coxca, José Luis Reyes-Sánchez, Oscar Rojas-Espinosa, Juan C. Cancino-Diaz, Rafael Rodríguez-Muñoz, Ernesto Antonio Vázquez-Sánchez
Publikováno v:
Molecular Immunology. 68:333-340
The dynamic regulation of NF-κB activity in the uterus maintains a favorable environment of cytokines necessary to prepare for pregnancy throughout the estrous cycle. Recently, the mechanisms that directly regulate the NF-κB transcriptional activit
Autor:
Fernando Gómez-Chávez, Isaí Martínez-Torres, Azael Torales-Cardeña, Juan C. Cancino-Diaz, Sandra Rodríguez-Martínez, Ernesto Antonio Vázquez-Sánchez, Mario E. Cancino-Diaz
Publikováno v:
Mediators of Inflammation, Vol 2015 (2015)
Mediators of Inflammation
Mediators of Inflammation
Psoriasis is a chronic inflammatory skin disease where the altered regulation in angiogenesis, inflammation, and proliferation of keratinocytes are the possible causes of the disease, and the transcription factor “hypoxia-inducible factor 1-alpha