Zobrazeno 1 - 10
of 33
pro vyhledávání: '"Irmela Sulzer"'
Autor:
Rahel Froehlich-Zahnd, James N. George, Sara K. Vesely, Deirdra R. Terrell, Khatira Aboulfatova, Jing-Fei Dong, Brenda M. Luken, Jan Voorberg, Ulrich Budde, Irmela Sulzer, Bernhard Lämmle, Johanna A. Kremer Hovinga
Publikováno v:
Haematologica, Vol 97, Iss 2 (2012)
Background Severe ADAMTS13 deficiency is a critical component of the pathogenesis of idiopathic thrombotic thrombocytopenic purpura but is found only in about 60% of patients clinically diagnosed with this disease.Design and Methods Over a period of
Externí odkaz:
https://doaj.org/article/a0b8fecb958348f5be4719c9e1a632ad
Publikováno v:
Haematologica, Vol 97, Iss 1 (2012)
Background The current gold-standard for diagnosing heparin-induced thrombocytopenia is the detection of platelet-activating antibodies by means of functional assays which, since they are time consuming and not widely available, are not suited to gui
Externí odkaz:
https://doaj.org/article/7efe398d0f5f485bb6e209bc7bc320f6
Autor:
Tiziano Moccetti, Irmela Sulzer, Adriana Anesini, Giovanni Pedrazzini, Luigi Biasco, Johanna A. Kremer Hovinga, Lorenzo Alberio
Publikováno v:
Blood
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::274a09217708faea1bef2ad7b3c8f258
https://www.zora.uzh.ch/id/eprint/128534/
https://www.zora.uzh.ch/id/eprint/128534/
Autor:
Ulrich Budde, Irmela Sulzer, Brenda M. Luken, Rahel Froehlich-Zahnd, Bernhard Lämmle, Jan Voorberg, Deirdra R. Terrell, Khatira Aboulfatova, James N. George, Jing-fei Dong, Sara K. Vesely, Johanna A. Kremer Hovinga
Publikováno v:
Haematologica, 97(2), 297-303. Ferrata Storti Foundation
Background Severe ADAMTS13 deficiency is a critical component of the pathogenesis of idiopathic thrombotic thrombocytopenic purpura but is found only in about 60% of patients clinically diagnosed with this disease. Design and Methods Over a period of
Publikováno v:
Haematologica. 97:89-97
Background The current gold-standard for diagnosing heparin-induced thrombocytopenia is the detection of platelet-activating antibodies by means of functional assays which, since they are time consuming and not widely available, are not suited to gui
Publikováno v:
International Journal of Laboratory Hematology. 33:168-175
Introduction: The aim of the present work was to verify whether calculating a ratio between clotting times obtained with the sensitive PTT-LA and a less sensitive activated partial thromboplastin time (aPTT)-reagent may represent a valuable aPTT-base
Variability of anti‐PF4/heparin antibody results obtained by the rapid testing system ID‐H/PF4‐PaGIA
Autor:
Irmela Sulzer, Lorenzo Alberio, Giuseppe Colucci, S. Schneiter, Bernhard Lämmle, Gabriela Barizzi
Publikováno v:
Journal of Thrombosis and Haemostasis. 7:1649-1655
Summary. Background: Recent studies have shown that a low clinical pretest probability may be adequate for excluding heparin-induced thrombocytopenia. However, for patients with intermediate or high pretest probability, laboratory testing is essentia
Autor:
Walter A. Wuillemin, Irmela Sulzer, C. Erik Hack, A. J. M. Eerenberg-Belmer, Christoph Caliezi, Gerard van Mierlo, Sacha Zeerleder
Publikováno v:
Clinical and diagnostic laboratory immunology, 10(4), 529-535. American Society for Microbiology
Forty patients with severe sepsis or septic shock recently received C1 inhibitor. In the present study we studied the effect of C1 inhibitor therapy on circulating elastase-α1-antitrypsin complex (EA) and lactoferrin (LF) levels in these patients to
Publikováno v:
Thrombosis Research. 105:463-470
Hereditary plasma prekallikrein (PK) deficiency was diagnosed in a 71-year-old man with an 8-year history of osteomyelofibrosis. PK deficiency was suspected in view of a severely prolonged activated partial thromboplastin time (aPTT) that nearly norm
Publikováno v:
Therapeutische Umschau. 56:509-512
Bei einer Patientin ohne Blutungsneigung und ohne anamnestische Thromboembolien wurde auf Grund einer isoliert stark verlängerten aPTT bei normaler Thromboplastinzeit (Quick) und normaler Thrombinzeit ein schwerer Faktor XII (FXII)-Mangel diagnostiz