Zobrazeno 1 - 10
of 28
pro vyhledávání: '"Irma M. Sainz"'
Autor:
Junya Fukuoka, Jaishree Jagirdar, Steven S. Shen, Irma M. Sainz, Bradley M. Turner, Philip T. Cagle
Publikováno v:
Archives of Pathology & Laboratory Medicine. 136:163-171
Context.—Differentiation of non–small cell carcinoma into histologic types is important because of new, successful therapies that target lung adenocarcinoma (ACA). TTF-1 is a favored marker for lung ACA but has limited sensitivity and specificity
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::040e02f79773c5b2c963f338f91e6145
https://doi.org/10.5858/arpa.2011-0320-oa
https://doi.org/10.5858/arpa.2011-0320-oa
Autor:
Irma Isordia-Salas, Elba Reyes-Maldonado, Alfredo Leaños-Miranda, Irma M. Sainz, Gabriela Borrayo-Sánchez
Publikováno v:
Revista Española de Cardiología (English Edition). 62:365-372
To investigate the role of the 4G/5G polymorphism in the plasminogen activator inhibitor-1 (PAI-1) gene in patients with ST-elevation myocardial infarction (STEMI) agedor =45 years and its influence on regulation of the plasma PAI-1 concentration.Thi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::90d9e716eac67778cb3ad70a4be6ab4a
https://doi.org/10.1016/s1885-5857(09)71663-9
https://doi.org/10.1016/s1885-5857(09)71663-9
Autor:
Gabriela Borrayo-Sánchez, Irma Isordia-Salas, Elba Reyes-Maldonado, Alfredo Leaños-Miranda, Irma M. Sainz
Publikováno v:
Revista Española de Cardiología. 62:365-372
Introduccion y objetivos Determinar la participacion del polimorfismo 4G/5G en el gen del inhibidor del activador del plasminogeno tipo 1 (PAI-1) en pacientes con infarto agudo de miocardio con elevacion del segmento ST y edad ≤ 45 anos y su influe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0d0890cedc4707e6bd59a38675f4d0cb
https://doi.org/10.1016/s0300-8932(09)70893-0
https://doi.org/10.1016/s0300-8932(09)70893-0
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 27:1968-1975
Objective— The cleaved form of high molecular weight kininogen (HKa) is a potent inhibitor of angiogenesis and tumor growth in vivo; the functional domain has been identified as domain 5 (D5, named as kininostatin). We now identify the subcellular
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e2f1f8b8a7187eb51a1e2d60d4b0af6b
https://doi.org/10.1161/atvbaha.107.148759
https://doi.org/10.1161/atvbaha.107.148759
Autor:
Irma Isordia-Salas, Robert W. Colman, Leo M. Albert, Yelena Leathurby, Robin A. Pixley, James C. Keith, Irma M. Sainz
Publikováno v:
Arthritis Research & Therapy
The human leukocyte antigen B27 (HLA-B27) transgenic rat is a model of human inflammatory bowel disease, rheumatoid arthritis and psoriasis. Studies of chronic inflammation in other rat models have demonstrated activation of the kallikrein–kinin sy
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cb782cdde112e3090e436904f5b373f3
http://europepmc.org/articles/PMC1175023
http://europepmc.org/articles/PMC1175023
Autor:
Irma Isordia-Salas, Robert W. Colman, Robin A. Pixley, Raul A. DeLa Cadena, Julian L. Castaneda, Albert Adam, Irma M. Sainz, Alexis Agelan, Bo Liu, R. Balfour Sartor
Publikováno v:
Arthritis & Rheumatism. 52:2549-2552
Objective To compare inflammatory peripheral arthritis in wild-type and high molecular weight kininogen (HK)–deficient rats, both on the genetically susceptible Lewis background. Methods By backcrossing Brown-Norway HK-deficient rats with Lewis rat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6944960ebc0993cdc99fa82a43d99986
https://doi.org/10.1002/art.21202
https://doi.org/10.1002/art.21202
Publikováno v:
Thrombosis Research. 116:533-543
Introduction Thrombospondin 1 (TSP1) has the ability to bind to HL-60 cells and to reversibly inhibit human neutrophil elastase (HNE). Human factor V (FV) can be cleaved by HNE thereby providing FV with cofactor activity (FVa HNE ). Experiments were
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6eaca88bd0bb8899246bed5f2bd23794
https://doi.org/10.1016/j.thromres.2005.02.012
https://doi.org/10.1016/j.thromres.2005.02.012
Autor:
Irma M. Sainz, Yan-Lin Guo, Harlan N. Bradford, E. Premkumar Reddy, James S. Song, Abdel Bior, Stephen C. Cosenza, Irma Isordia-Salas, Robert W. Colman, Robin A. Pixley
Publikováno v:
Blood. 104:2065-2072
We have shown that human high molecular weight kininogen is proangiogenic due to release of bradykinin. We now determined the ability of a murine monoclonal antibody to the light chain of high molecular weight kininogen, C11C1, to inhibit tumor growt
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b32d4b9b702642c2cbe1615be9e78191
https://doi.org/10.1182/blood-2004-02-0449
https://doi.org/10.1182/blood-2004-02-0449
Autor:
Irma M. Sainz, Carlos Martínez-Murillo, Irma Isordia-Salas, Robin A. Pixley, Robert W. Colman
Publikováno v:
Archives of Medical Research. 35:369-377
Inflammation is accompanied by activation of the plasma kallikrein-kinin system (KKS). KKS activation has been demonstrated in a variety of inflammatory human diseases. To further explore the participation of KKS in arthritis and inflammatory bowel d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::88a57d663b7262e74c01587d624a6834
https://doi.org/10.1016/j.arcmed.2004.05.004
https://doi.org/10.1016/j.arcmed.2004.05.004
Autor:
Irma M. Sainz, Audrey B. Uknis, Robert W. Colman, Albert Adam, Walter K. Long, Robin A. Pixley, Alexis Agelan, John P. Gaughan, Irma Isordia-Salas, Raul A. DeLa Cadena, Ricardo G. Espinola
Publikováno v:
The American Journal of Pathology. 165:969-976
We reported that high-molecular weight kininogen is proangiogenic by releasing bradykinin and that a monoclonal antibody to high-molecular weight kininogen, C11C1, blocked its binding to endothelial cells. We now test if this antibody can prevent art
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::adfb24aa48e4683fd6902045f03da771
https://doi.org/10.1016/s0002-9440(10)63358-5
https://doi.org/10.1016/s0002-9440(10)63358-5