Zobrazeno 1 - 10
of 36
pro vyhledávání: '"Iris Kistner"'
Autor:
Agnes J. Bosch, Joanna M. Harazny, Iris Kistner, Stefanie Friedrich, Joanna Wojtkiewicz, Roland E. Schmieder
Publikováno v:
BMC Cardiovascular Disorders, Vol 17, Iss 1, Pp 1-11 (2017)
Abstract Background Microvascular rarefaction influences peripheral vascular resistance, perfusion and metabolism by affecting blood pressure and flow pattern. In hypertension microvascular rarefaction has been described in experimental animal studie
Externí odkaz:
https://doaj.org/article/53d288e1f3df466f9c2f2389bb10c740
Autor:
Christian Ott, Iris Kistner, Agnes Jumar, Stephanie Friedrich, Peter Bramlage, Roland Schmieder
Publikováno v:
Artery Research, Vol 16 (2016)
Externí odkaz:
https://doaj.org/article/b2e8af3a80de4476a086317d642aa218
Autor:
Roland E. Schmieder, Christian Ott, Axel Schmid, Stefanie Friedrich, Iris Kistner, Tilmann Ditting, Roland Veelken, Michael Uder, Stefan W. Toennes
Publikováno v:
Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, Vol 5, Iss 2 (2016)
BackgroundAdherence to medication has been repeatedly proposed to represent a major cause of treatment‐resistant hypertension (TRH); however, treatment decisions such as treating TRH with renal denervation depend on accurate judgment of adherence.
Externí odkaz:
https://doaj.org/article/e3faaf66b3b54a34970a33baa780c41b
Autor:
Agnes Jumar, Joanna M Harazny, Christian Ott, Stefanie Friedrich, Iris Kistner, Kristina Striepe, Roland E Schmieder
Publikováno v:
PLoS ONE, Vol 11, Iss 12, p e0162608 (2016)
In diabetes mellitus type 2, capillary rarefaction plays a pivotal role in the pathogenesis of end-organ damage. We investigated retinal capillary density in patients with early disease.This cross-sectional study compares retinal capillary rarefactio
Externí odkaz:
https://doaj.org/article/51d9217ed1d8438aadc429bbf46cd5dc
Autor:
Iris Kistner, Stefanie Friedrich, Joanna Harazny, Christian Ott, Agnes Jumar, Roland E. Schmieder, Georg Michelson
Publikováno v:
Microcirculation. 23:447-455
Background Eutrophic and hypertrophic remodeling are major vascular hallmarks for hypertension and diabetes-associated microvascular end-organ damage in peripheral arterioles. The aim of this study is to compare retinal arterioles of diabetic, hypert
Autor:
Christian Ott, Roland E. Schmieder, Agnes Jumar, Iris Kistner, Joanna Harazny, Stefanie Friedrich
Publikováno v:
J Clin Hypertens (Greenwich)
Decreased capillary density influences vascular resistance and perfusion. The authors aimed to investigate the influence of the renin‐angiotensin receptor blocker valsartan on retinal capillary rarefaction in hypertensive patients. Retinal vascular
Autor:
Joanna Harazny, Roland E. Schmieder, Iris Kistner, Agnes Jumar, Stephanie Schmidt, Christian Ott, Stefanie Friedrich
Publikováno v:
Journal of Hypertension. 33:2491-2499
In hypertension, changes in small arterial structure are characterized by an increased wall-to-lumen ratio (WLR). These adaptive processes are modulated by the rennin-angiotensin system. It is unclear whether direct renin inhibitors exert protective
Autor:
Michael Uder, Felix Mahfoud, Roland Veelken, Roland E. Schmieder, Christian Ott, Michael Böhm, Ertan Akarca, Axel Schmid, Iris Kistner, Tilmann Ditting
Publikováno v:
Journal of the American College of Cardiology. 72(25)
A bidirectional relationship is known between increased sympathetic activity inducing insulin resistance and hyperinsulinemia producing sympathetic overactivity, resulting in a vicious circle [(1)][1]. Thus, it seems likely that renal denervation (RD
Autor:
S. Friedrich, Christian Ott, Roland E. Schmieder, T. Ditting, R. Veelken, R. Schutte, Ulrike Raff, S. Schmidt, Iris Kistner, B. Deutsch
Publikováno v:
Journal of Nephrology. 28:165-172
Background Hyperkalemia is a common and life–threatening complication frequently seen in patients with acute kidney injury, end–stage renal disease and chronic heart failure. Cardiac arrest and ventricular fibrillation are possible consequences.
Autor:
Andreas Schneider, Christian Ott, Markus P. Schneider, Roland E. Schmieder, Iris Kistner, Agnes Jumar
Publikováno v:
Clinical Science. 127:499-505
Endothelial dysfunction has been shown to promote podocyte injury and albuminuria in diabetes, highlighting the importance of the interaction between renal endothelial cells and podocytes. Folic acid (FA) improves nitric oxide synthase (NOS) function