Zobrazeno 1 - 10
of 43
pro vyhledávání: '"Irene J Andersson"'
Publikováno v:
PLoS ONE, Vol 10, Iss 3, p e0121897 (2015)
Danshen, in particular its derivative tanshinone IIA (TS), is a promising compound in the treatment of cardiovascular diseases and has been used for many years in traditional Chinese medicine. Although many actions of TS have been researched, its vas
Externí odkaz:
https://doaj.org/article/94cd8c51514c4a7ca17bd0e27c8609ec
Autor:
Rajan Poudel, Joanna L Stanley, Christian F Rueda-Clausen, Irene J Andersson, Colin P Sibley, Sandra T Davidge, Philip N Baker
Publikováno v:
PLoS ONE, Vol 8, Iss 5, p e64401 (2013)
Preeclampsia (PE) and fetal growth restriction (FGR) contribute significantly to fetal and maternal morbidity and mortality. Although the causes of PE and FGR are not fully understood, both conditions are known to be associated with impaired uterine
Externí odkaz:
https://doaj.org/article/9e159fba021044c0b3508a46e1b622a7
Autor:
Sandra T. Davidge, Fred A. English, Irene J. Andersson, Louise C. Kenny, Joanna L. Stanley, Sarah K. Walsh, Philip N. Baker, Fergus P. McCarthy
Publikováno v:
Reproductive Sciences. 19:806-813
A low-resistance/high-flow uteroplacental circulation is integral to a healthy pregnancy. Impaired flow in the uterine circulation is associated with intrauterine growth restriction (IUGR) and preeclampsia (PE). Poly(ADP-ribose) polymerase (PARP) is
Autor:
Cassandra J. Hirt, Mark Wareing, Mark Dilworth, Bernadette Baker, Colin P. Sibley, Irene J. Andersson, Jocelyn D. Glazier, Joanna L. Stanley, Lewis Renshall, Philip N. Baker, L.C. Kusinski
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 303:R86-R93
Fetal growth restriction (FGR) is the inability of a fetus to reach its genetically predetermined growth potential. In the absence of a genetic anomaly or maternal undernutrition, FGR is attributable to “placental insufficiency”: inappropriate ma
Autor:
Rajan Poudel, Christian F. Rueda-Clausen, Sandra T. Davidge, Irene J. Andersson, Joanna L. Stanley, Philip N. Baker, Colin P. Sibley
Publikováno v:
Hypertension. 59:1021-1028
Preeclampsia and fetal growth restriction are responsible for the majority of maternal and perinatal morbidity and mortality associated with complicated pregnancies. Although their etiologies are complex and multifactorial, both are associated with i
Autor:
Camilla Alexanderson, Göran Bergström, Agneta Holmäng, Ole Skøtt, Maria E. Johansson, Irene J. Andersson
Publikováno v:
Johansson, M E, Andersson, I J, Alexanderson, C, Skøtt, O, Holmang, A & Bergstrom, G 2008, ' Hyperinsulinemic rats are normotensive but sensitized to angiotensin II ', American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, vol. 294, pp. R1240-R1247 . https://doi.org/10.1152/ajpregu.00493.2007
The effect of insulin on blood pressure (BP) is debated, and an involvement of an activated renin-angiotensin aldosterone system (RAAS) has been suggested. We studied the effect of chronic insulin infusion on telemetry BP and assessed sympathetic act
Autor:
Göran Bergström, Irene J. Andersson, Evelina Bernberg, Maria E. Johansson, Li-Ming Gan, Andrew S. Naylor
Publikováno v:
Stress. 11:381-389
Social support and a stimulating environment have been suggested to reduce stress reactions and cardiovascular risk. The aim of this study was to assess the role of environmental enrichment and social interaction for development of atherosclerosis in
Autor:
Emil Egecioglu, Britt G. Gabrielsson, Jörgen Isgaard, Mohammad Bohlooly-Y, Ole Skøtt, John J. Kopchick, Vilborg Palsdottir, Entela Bollano, Göran Bergström, Anna Wickman, Peter Bie, Irene J. Andersson
Publikováno v:
Egecioglu, E, Andersson, I J, Bollano, E, Palsdottir, V, Gabrielsson, B G, Kopchick, J J, Skøtt, O, Bie, P, Isgaard, J, Bohlooly-Y, M, Bergström, G & Wickman, A 2007, ' Growth hormone receptor deficiency in mice results in reduced systolic blood pressure and plasma renin, increased aortic eNOS expression, and altered cardiovascular structure and function ', American Journal of Physiology: Endocrinology and Metabolism, vol. 292, no. 5, pp. E1418-E1425 . https://doi.org/10.1152/ajpendo.00335.2006
To study the role of the growth hormone receptor (GHR) in the development of cardiovascular structure and function, female GHR gene-disrupted or knockout (KO) and wild-type (WT) mice at age 18 wk were used. GHR KO mice had lower plasma renin levels (
Autor:
Joanna L. Stanley, Philip N. Baker, David S. Wishart, Rupasri Mandal, Sandra T. Davidge, Colin P. Sibley, Louise C. Kenny, David Broadhurst, Karolina Sulek, Irene J. Andersson
Publikováno v:
Scientific Reports
Preeclampsia (PE) and fetal growth restriction (FGR) are serious complications of pregnancy, associated with greatly increased risk of maternal and perinatal morbidity and mortality. These complications are difficult to diagnose and no curative treat
Autor:
Jan Borén, Anna Wickman, Natalia Klintland, Göran Bergström, Maria E. Johansson, Irene J. Andersson, Kenneth Caidahl, Maria Gustafsson, Mohammad Bohlooly-Y, Li-Ming Gan
Publikováno v:
Clinical Science. 110:217-225
Acromegaly [overproduction of GH (growth hormone)] is associated with cardiovascular disease. Transgenic mice overexpressing bGH (bovine GH) develop hypertension and hypercholesterolaemia and could be a model for cardiovascular disease in acromegaly.