Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Iohanan Daniel García-Marín"'
Autor:
Raúl Horacio Camarillo-López, Maricarmen Hernández Rodríguez, Mónica Adriana Torres-Ramos, Ivonne Maciel Arciniega-Martínez, Iohanan Daniel García-Marín, José Correa Basurto, Juan Vicente Méndez Méndez, Martha Cecilia Rosales-Hernández
Publikováno v:
Molecules, Vol 25, Iss 21, p 5009 (2020)
Alzheimer’s disease (AD) is a neurodegenerative disease with no cure nowadays; there is no treatment either to prevent or to stop its progression. In vitro studies suggested that tert-butyl-(4-hydroxy-3-((3-(2-methylpiperidin-yl)propyl)carbamoyl)ph
Externí odkaz:
https://doaj.org/article/cf5f6be911c848929a40165450205ebd
Autor:
José Correa-Basurto, Iohanan Daniel García-Marín, Maricarmen Hernández-Rodríguez, I. Arciniega-Martinez, Martha Cecilia Rosales-Hernández
Publikováno v:
Molecular Neurobiology. 57:3979-3988
The increase of amyloid beta (Aβ) release and hyperphosphorylation of Tau protein represents the main events related to Alzheimer's disease (AD). Furthermore, the sporadic type represents the most common form of AD. Therefore, the establishment of a
Autor:
Iohanan Daniel García Marín, Raúl Horacio Camarillo López, Oscar Aurelio Martínez, Itzia Irene Padilla-Martínez, José Correa-Basurto, Martha Cecilia Rosales-Hernández
Publikováno v:
PloS one. 17(6)
The preset neurodegenerations in Alzheimer disease (AD) are due to several mechanisms such as amyloidogenic proteolysis, neuroinflammation, mitochondrial dysfunction, neurofibrillary tangles, cholinergic dysfunction, among others. The aim of this wor
Autor:
José Correa Basurto, Juan Vicente Méndez Méndez, Raúl Horacio Camarillo-López, Iohanan Daniel García-Marín, Martha Cecilia Rosales-Hernández, I. Arciniega-Martinez, Mónica Adriana Torres-Ramos, Maricarmen Hernández Rodríguez
Publikováno v:
Molecules
Volume 25
Issue 21
Molecules, Vol 25, Iss 5009, p 5009 (2020)
Volume 25
Issue 21
Molecules, Vol 25, Iss 5009, p 5009 (2020)
Alzheimer&rsquo
s disease (AD) is a neurodegenerative disease with no cure nowadays
there is no treatment either to prevent or to stop its progression. In vitro studies suggested that tert-butyl-(4-hydroxy-3-((3-(2-methylpiperidin-yl)propyl
s disease (AD) is a neurodegenerative disease with no cure nowadays
there is no treatment either to prevent or to stop its progression. In vitro studies suggested that tert-butyl-(4-hydroxy-3-((3-(2-methylpiperidin-yl)propyl
Autor:
Iohanan Daniel García Marín, Raúl Horacio Camarillo López, Oscar Aurelio Martínez, Itzia Irene Padilla-Martínez, José Correa-Basurto, Martha Cecilia Rosales-Hernández
Publikováno v:
PLoS ONE, Vol 17, Iss 6 (2022)
The preset neurodegenerations in Alzheimer disease (AD) are due to several mechanisms such as amyloidogenic proteolysis, neuroinflammation, mitochondrial dysfunction, neurofibrillary tangles, cholinergic dysfunction, among others. The aim of this wor
Externí odkaz:
https://doaj.org/article/10264a7698fd4adeb98fdb0cc35b9110