The Interaction between Macrophages and Triple‐negative Breast Cancer Cells Induces ROS‐Mediated Interleukin 1α Expression to Enhance Tumorigenesis and Metastasis

Autor: Meng Hao, Bin Huang, Renfei Wu, Zheng Peng, Kathy Qian Luo

Publikováno v: Advanced Science, Vol 10, Iss 29, Pp n/a-n/a (2023)

Abstract Triple‐negative breast cancer (TNBC) has higher mortality than non‐TNBC because of its stronger metastatic capacity. Increasing studies reported that TNBC tumors had more macrophage infiltration than non‐TNBC tumors, which promoted the

Externí odkaz: https://doaj.org/article/6afbccf4fa9a4714bf3d63402314ccc4

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Host-derived Interleukin 1α induces an immunosuppressive tumor microenvironment via regulating monocyte-to-macrophage differentiation.

Autor: Raja MRK; Department of Biological Sciences, University of South Carolina, Columbia, SC 29208, USA., Gupta G; Department of Biological Sciences, University of South Carolina, Columbia, SC 29208, USA., Atkinson G; Department of Biological Sciences, University of South Carolina, Columbia, SC 29208, USA., Kathrein K; Department of Biological Sciences, University of South Carolina, Columbia, SC 29208, USA., Armstrong A; Department of Biological Sciences, University of South Carolina, Columbia, SC 29208, USA., Gower M; Department of Chemical Engineering and Biomedical Engineering, University of South Carolina, Columbia, SC 29108, USA., Roninson I; Department of Drug Discovery & Biomedical Sciences (DDBS), College of Pharmacy, University of South Carolina, Columbia, SC 29208, USA., Broude E; Department of Drug Discovery & Biomedical Sciences (DDBS), College of Pharmacy, University of South Carolina, Columbia, SC 29208, USA., Chen M; Department of Drug Discovery & Biomedical Sciences (DDBS), College of Pharmacy, University of South Carolina, Columbia, SC 29208, USA., Ji H; Department of Drug Discovery & Biomedical Sciences (DDBS), College of Pharmacy, University of South Carolina, Columbia, SC 29208, USA., Lim C; Department of Drug Discovery & Biomedical Sciences (DDBS), College of Pharmacy, University of South Carolina, Columbia, SC 29208, USA., Wang H; Department of Biomedical Engineering, Stevens Institute of Technology, Hoboken, NJ 07030, USA., Fan D; Department of Cell Biology and Anatomy, University of South Carolina School of Medicine, Columbia, SC 29209, USA., Xu P; Department of Drug Discovery & Biomedical Sciences (DDBS), College of Pharmacy, University of South Carolina, Columbia, SC 29208, USA., Li J; Department of Chemistry and Biochemistry, University of South Carolina, Columbia, SC 29201, USA., Zhou G; Georgia Cancer Center, Department of Medicine, Medical College of Georgia, Augusta, GA 30912, USA., Chen H; Department of Biological Sciences, University of South Carolina, Columbia, SC 29208, USA.

Publikováno v: BioRxiv : the preprint server for biology [bioRxiv] 2024 May 05. Date of Electronic Publication: 2024 May 05.

Heightened Expression of Tumor Necrosis Factor α , Interleukin 1α , and Glial Fibrillary Acidic Protein in Experimental Creutzfeldt-Jakob Disease in Mice

Autor: Kordek, Radzislaw, Nerurkar, Vivek R., Liberski, Pawel P., Isaacson, Stuart, Yanagihara, Richard, Gajdusek, D. Carleton

Publikováno v: Proceedings of the National Academy of Sciences of the United States of America, 1996 Sep 01. 93(18), 9754-9758.

Externí odkaz: https://www.jstor.org/stable/39783

The N-Terminal Propiece of Interleukin 1α is a Transforming Nuclear Oncoprotein

Autor: Stevenson, Frazier T., Turck, Johanna, Locksley, Richard M., Lovett, David H.

Publikováno v: Proceedings of the National Academy of Sciences of the United States of America, 1997 Jan . 94(2), 508-513.

Externí odkaz: https://www.jstor.org/stable/41233