Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Inka Lindner"'
Autor:
Jürgen Schrezenmeir, Alexandra Fischer, Berit Marten, Inka Lindner, Frank Döring, Stefan Schreiber, Ulf Helwig, Diana Rubin
Publikováno v:
Molecular Nutrition & Food Research. 51:1447-1451
The prostaglandin E synthase 2 (PTGES2) gene maps to a locus linked to obesity and is involved in the synthesis of the antilipolytic compound prostaglandin E(2). In a recent study, we found an association of the minor PTGES2 Arg298His allele and lowe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9cf793db34d846cff2f844c828025d01
https://doi.org/10.1002/mnfr.200700144
https://doi.org/10.1002/mnfr.200700144
Autor:
Inka Lindner, Stefan Dahm, Henning Gohlke, Barbara Burwinkel, Wolfgang Rathmann, Harald Grallert, Eva Fisher, Heiner Boeing, Thomas Illig, H.-Erich Wichmann, Jürgen Schrezenmeir, Frank Döring, Christian Gieger, Inke Nitz
Publikováno v:
Molecular Nutrition & Food Research. 51:178-184
The human acyl-CoA-binding protein (ACBP) is a potential candidate gene of type 2 diabetes (T2D), since it plays a central role in determining the intracellular concentration of activated fatty acids which contribute to insulin resistance. The aim of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c576f7d9bddad28da4f2704a37dfd826
https://doi.org/10.1002/mnfr.200600163
https://doi.org/10.1002/mnfr.200600163
Publikováno v:
IUBMB Life. 59:628-633
Coenzyme Q10 (CoQ10, ubiquinone) is an essential cofactor in the electron transport chain, serves as a potent antioxidant in mitochondria and lipid membranes, and is often used as a dietary supplement for a number of diseases including cardiovascular
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dc28933a99388b23a9176d24d677b280
https://doi.org/10.1080/15216540701545991
https://doi.org/10.1080/15216540701545991
Autor:
Inka Lindner, Gerald Rimbach, Thomas Menke, Gerti Lorenz, Frank Döring, Constance Schmelzer, Petra Niklowitz
Publikováno v:
BioFactors. 31:35-41
Studies in humans and cell culture as well as bioinformatics suggested that Coenzyme Q(10) (CoQ10) functions as an anti-inflammatory molecule. Here we studied the influence of CoQ10 (Kaneka Q10) on secretion of the pro-inflammatory cytokine tumor nec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ae69a0731e6bcc8c8be9d22b0355634c
https://doi.org/10.1002/biof.5520310104
https://doi.org/10.1002/biof.5520310104
Autor:
Inka Lindner, Stefan Schreiber, Ulf Helwig, Inke Nitz, Jochen Hampe, Jürgen Schrezenmeir, Frank Döring, Diana Rubin
Publikováno v:
Molecular Nutrition & Food Research. 50:270-274
Enzymes of the medium-chain acyl-CoA synthetase (MACS) family catalyze the ligation of medium chain fatty acids with CoA to produce medium-chain-acyl-CoA. At least four members of the MACS gene family are clustered on human chromosome 16p12. Associat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b2432e7ce5945db2bfe041f7edba597f
https://doi.org/10.1002/mnfr.200500241
https://doi.org/10.1002/mnfr.200500241
Autor:
Stefan Schreiber, Matthias Möhlig, Ulf Helwig, Heiner Boeing, Diana Rubin, Yun Li, Jürgen Schrezenmeir, Frank Döring, Joachim Spranger, Inka Lindner, Jochen Hampe, Andreas Pfeiffer, Eva Fisher
Publikováno v:
Molecular Nutrition & Food Research. 49:972-976
The protein encoded by the pancreatic colipase (CLPS) gene is an essential cofactor needed by pancreatic triglyceride lipase (PNLIP) for efficient dietary lipid hydrolysis. Since the inhibition of lipase activity was shown to reduce the incidence of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d2c85f1b8bb7aeed2247b75b5af63daa
https://doi.org/10.1002/mnfr.200500087
https://doi.org/10.1002/mnfr.200500087
Autor:
Thomas Menke, Gerald Rimbach, Frank Döring, Inka Lindner, Constance Schmelzer, Petra Niklowitz
Publikováno v:
BioFactors (Oxford, England). 32(1-4)
Clinical studies demonstrated the efficacy of Coenzyme Q(10) (CoQ(10)) as an adjuvant therapeutic in cardiovascular diseases, mitochondrial myopathies and neurodegenerative diseases. More recently, expression profiling revealed that Coenzyme Q(10) (C
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::88d7d87b8a3fe9fa315154a2a3abb456
https://pubmed.ncbi.nlm.nih.gov/19096114
https://pubmed.ncbi.nlm.nih.gov/19096114
Autor:
Jürgen Schrezenmeir, Henning Gohlke, Stefan Schreiber, H.-Erich Wichmann, Eva Fisher, Frank Döring, Wolfgang Rathmann, Angela Döring, Yun Li, Thomas Illig, Christian Gieger, Heiner Boeing, Diana Rubin, Harald Grallert, Inka Lindner, Joachim Spranger, Inke Nitz
Publikováno v:
The Journal of clinical endocrinology and metabolism. 92(8)
Context: On the basis of its chromosomal localization and its role in the synthesis of the antilipolytic compound prostaglandin E2, the prostaglandin E synthase 2 (PTGES2) is a candidate gene for type 2 diabetes.Objective: The aim of the present stud
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a4ea1cca52cd8cb9388e80ff4e537834
https://pubmed.ncbi.nlm.nih.gov/17566096
https://pubmed.ncbi.nlm.nih.gov/17566096
Autor:
Jochen Hampe, Stefan Schreiber, Inke Nitz, Diana Rubin, Matthias Möhlig, Heiner Boeing, Eva Fisher, Inka Lindner, Jürgen Schrezenmeir, Frank Döring
Publikováno v:
Molecular nutritionfood research. 51(2)
To search for common variants etiological for type 2 diabetes, we screened 15 genes involved in fat assimilation for sequence variants. Approximately 55 kb in promoter and coding regions, and intron/ splice sites were sequenced by cycle sequencing. I
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5cffeddc9d5212bbdc302106449bba15
https://pubmed.ncbi.nlm.nih.gov/17266179
https://pubmed.ncbi.nlm.nih.gov/17266179
Autor:
Eva Fisher, Maja Klapper, Jürgen Schrezenmeir, Cornelia Weikert, Heiner Boeing, Matthias Möhlig, Inka Lindner, Frank Döring, Joachim Spranger
Publikováno v:
Molecular genetics and metabolism. 91(3)
To determine the possible role of the common FABP1 T94A polymorphism in modulating susceptibility to traits of the metabolic syndrome, we analysed a random sample of 826 subjects from the European Prospective Investigation into Cancer and Nutrition (
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::745ecf492700880d0ac89524fc2d064c
https://pubmed.ncbi.nlm.nih.gov/17485234
https://pubmed.ncbi.nlm.nih.gov/17485234