Zobrazeno 1 - 10 of 85 pro vyhledávání: '"Inhak Choi"'
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Akademický článek
VSIG4(+) peritoneal macrophages induce apoptosis of double-positive thymocyte via the secretion of TNF-α in a CLP-induced sepsis model resulting in thymic atrophy
Autor: Hae-Yun Cho, Yun Gyeong Yang, Youkyoung Jeon, Chae-Kwan Lee, InHak Choi, Soo-Woong Lee
Publikováno v: Cell Death and Disease, Vol 12, Iss 6, Pp 1-14 (2021)
Abstract Thymic atrophy in sepsis is a critical disadvantage because it induces immunosuppression and increases the mortality rate as the disease progresses. However, the exact mechanism of thymic atrophy has not been fully elucidated. In this study,
Externí odkaz: https://doaj.org/article/f567aba303204726877a31fe9d1e3ccd
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VSIG4-expressing tumor-associated macrophages impair anti-tumor immunity
Autor: Keunok Jung, You-kyoung Jeon, Dae Hoon Jeong, Jung Mi Byun, Bjarne Bogen, Inhak Choi
Publikováno v: Biochemical and Biophysical Research Communications. 628:18-24
VSIG4, a newly identified co-inhibitory molecule belonging to the B7-related family, is exclusively expressed on tissue-resident macrophages and is involved in the suppression of T cell proliferation and cytokine production. We sought to characterize
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c7c6969b3d2f43313f71dd8f3bd6c880
https://doi.org/10.1016/j.bbrc.2022.08.055
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Data from Myeloma Drug Resistance Induced by Binding of Myeloma B7-H1 (PD-L1) to PD-1
Autor: Koiti Inokuchi, Koji Tamada, Inhak Choi, Keiichi Moriya, Yasuko Hamada, Namiko Okuyama, Asaka Kondo-Onodera, Mika Sunakawa, Hideto Tamura, Mariko Ishibashi
B7 homolog 1 (B7-H1)–expressing myeloma cells not only inhibit myeloma-specific cytotoxic T lymphocytes (CTL), but also confer a proliferative advantage: resistance to antimyeloma chemotherapy. However, it remains unknown whether B7-H1 expressed on
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::b44c7f9302111a8db44f05ec01e8f1ee
https://doi.org/10.1158/2326-6066.c.6548588.v1
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Supplementary Table 1 and Supplementary Figures 1 through 3 from Myeloma Drug Resistance Induced by Binding of Myeloma B7-H1 (PD-L1) to PD-1
Autor: Koiti Inokuchi, Koji Tamada, Inhak Choi, Keiichi Moriya, Yasuko Hamada, Namiko Okuyama, Asaka Kondo-Onodera, Mika Sunakawa, Hideto Tamura, Mariko Ishibashi
Supplementary Table S1 Primer Sequence. Supplementary Figure S1. (A) Surface antigen expression and (B) Cell morphology of MOSTI-1 cells, and (C) drug sensitivity to melphalan and bortezomib of MOSTI-1, U266, and RPMI8226 cells. Supplementary Figure
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::91798621635efee147057d1e41e6c5d4
https://doi.org/10.1158/2326-6066.22537886.v1
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9
VSTM5 is a novel immune checkpoint that promotes oral tolerance of cell-mediated and antibody responses
Autor: Opeyemi Ernest Oludada, Temitayo Opemipo Idowu, Youkyoung Jeon, Inhak Choi
Publikováno v: Biochemical and biophysical research communications. 635
The V-set and transmembrane domain-containing protein (VSTM) family is a newly discovered immunoglobulin (Ig) superfamily that shares structural similarities with the B7-like transmembrane proteins. Although most VSTM5 members have been reported to e
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0ca87b2f95cb1a21a331f15dc881bb70
https://pubmed.ncbi.nlm.nih.gov/36308908
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Coexpression of lymphocyte-activation gene 3 and programmed death ligand-1 in tumor infiltrating immune cells predicts worse outcome in renal cell carcinoma
Autor: Chan Ho Lee, Soo Jin Jung, Won Ik Seo, Jae Il Chung, Dae Sim Lee, Dae Hoon Jeong, Youkyoung Jeon, Inhak Choi
Publikováno v: International journal of immunopathology and pharmacology. 36
Objectives Lymphocyte-activation gene 3 (LAG-3) represents a potential immune checkpoint target for cancer treatment. We investigated LAG-3 expression and its prognostic value in patients with surgically treated clear cell renal cell carcinoma (RCC)
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b423cfb938d598c2a4acd645113a56a6
https://pubmed.ncbi.nlm.nih.gov/36083857
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