Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Ingvild Tronstad Moe"'
CTGF/CCN2 Postconditioning Increases Tolerance of Murine Hearts towards Ischemia-Reperfusion Injury.
Autor:
Ole Jørgen Kaasbøll, Ingvild Tronstad Moe, Mohammad Shakil Ahmed, Espen Stang, Else Marie Valbjørn Hagelin, Håvard Attramadal
Publikováno v:
PLoS ONE, Vol 11, Iss 2, p e0149000 (2016)
BACKGROUND AND PURPOSE:Previous studies of ischemia-reperfusion injury (IRI) in hearts from mice with cardiac-restricted overexpression of CCN2 have shown that CCN2 increases tolerance towards IRI. The objectives of this study were to investigate to
Externí odkaz:
https://doaj.org/article/477267d77ca8411d812f4adf71f2331b
Autor:
Tor Skomedal, Kurt A. Krobert, Thor Edvardsen, Ingvild Tronstad Moe, Eirik Qvigstad, M. Shakil Ahmed, Finn Olav Levy, Guro Valen, Jørgen Gravning, Håvard Attramadal, Else Marie Valbjørn Hagelin, Jan-Bjørn Osnes, Julia Sagave
Publikováno v:
Molecular Pharmacology. 84:372-383
Myocardial connective tissue growth factor (CTGF/CCN2) is induced in heart failure, a condition associated with diminution of β-adrenergic receptor (β-AR) responsiveness. Accordingly, we aimed to investigate whether CTGF could play a mechanistic ro
Autor:
Else Marie Valbjørn Hagelin, Mohammad Shakil Ahmed, Ole Jørgen Kaasbøll, Håvard Attramadal, Ingvild Tronstad Moe, Espen Stang
Publikováno v:
PLoS ONE
PLoS ONE, Vol 11, Iss 2, p e0149000 (2016)
PLoS ONE, Vol 11, Iss 2, p e0149000 (2016)
BACKGROUND AND PURPOSE:Previous studies of ischemia-reperfusion injury (IRI) in hearts from mice with cardiac-restricted overexpression of CCN2 have shown that CCN2 increases tolerance towards IRI. The objectives of this study were to investigate to
Autor:
Gabor Czibik, Håvard Attramadal, Thomas G. von Lueder, Vladimir N. Martinov, Erik Øie, Thor Edvardsen, Leif Erik Vinge, M. Shakil Ahmed, Guro Valen, Ingvild Tronstad Moe, Jørgen Gravning
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 300:H1291-H1302
CCN2/connective tissue growth factor (CTGF), a CCN family matricellular protein repressed in healthy hearts after birth, is induced in heart failure of various etiologies. Multiple cellular and biological functions have been assigned to CCN2/CTGF dep
Autor:
Ingvild Tronstad Moe
Publikováno v:
Tidsskrift for Den norske legeforening. 135:1393-1393
Autor:
Tuyet A.T. Pham, Håvard Attramadal, Mohammad Shakil Ahmed, Else Marie Valbjørn Hagelin, Ingvild Tronstad Moe
Publikováno v:
Journal of Cell Communication and Signaling
We recently reported that transgenic mice with cardiac-restricted overexpression of CCN2/CTGF have substantially increased tolerance towards ischemia/reperfusion injury. The purpose of this study was to investigate to what extent fully differentiated