Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Ingmar Sören Meyer"'
Autor:
Ingmar Sören Meyer, Xue Li, Carina Meyer, Oksana Voloshanenko, Susann Pohl, Michael Boutros, Hugo Albert Katus, Norbert Frey, Florian Leuschner
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 20, p 12252 (2022)
Wnt (a portmanteau of Wingless and Int-1) signaling in the adult heart is largely quiescent. However, there is accumulating evidence that it gets reactivated during the healing process after myocardial infarction (MI). We here tested the therapeutic
Externí odkaz:
https://doaj.org/article/ac409f8a8daf433cb2f50130e45aeeea
Autor:
Ingmar Sören Meyer, Andreas Jungmann, Christoph Dieterich, Min Zhang, Felix Lasitschka, Susann Werkmeister, Jan Haas, Oliver J Müller, Michael Boutros, Matthias Nahrendorf, Hugo A Katus, Stefan E Hardt, Florian Leuschner
Publikováno v:
EMBO Molecular Medicine, Vol 9, Iss 9, Pp 1279-1293 (2017)
Abstract A disturbed inflammatory response following myocardial infarction (MI) is associated with poor prognosis and increased tissue damage. Monocytes are key players in healing after MI, but little is known about the role of the cardiac niche in m
Externí odkaz:
https://doaj.org/article/5849c9afcb14451aae01f76bf3c5ab64
Autor:
Ingmar Sören Meyer, Carl Christoph Goetzke, Meike Kespohl, Martina Sauter, Arnd Heuser, Volker Eckstein, Hans-Peter Vornlocher, Daniel G. Anderson, Jan Haas, Benjamin Meder, Hugo Albert Katus, Karin Klingel, Antje Beling, Florian Leuschner
Publikováno v:
Frontiers in Immunology, Vol 9 (2018)
Myocarditis is an inflammatory disease of the heart muscle most commonly caused by viral infection and often maintained by autoimmunity. Virus-induced tissue damage triggers chemokine production and, subsequently, immune cell infiltration with pro-in
Externí odkaz:
https://doaj.org/article/48b682ae3bcc48c7b00ba3d1e47c0c08
Autor:
Christoph Hofmann, Adrian Serafin, Ole M Schwerdt, Fereshteh S Younesi, Florian Sicklinger, Ingmar Sören Meyer, Ellen Malovrh, Clara Sandmann, Lonny Jürgensen, Verena Kamuf-Schenk, Claudia Stroh, Zoe Löwenthal, Mandy Rettel, Frank Stein, Hugo A. Katus, Tobias Jakobi, Norbert Frey, Florian Leuschner, Mirko Völkers
RationaleRapid reperfusion is the most effective treatment for attenuating cardiac injury caused by myocardial ischemia. Yet, reperfusion itself elicits damage to the myocardium through incompletely understood mechanisms, known as ischemia/reperfusio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b525252149d2c5bbda14b71af7c2e4ad
https://doi.org/10.1101/2022.07.25.501397
https://doi.org/10.1101/2022.07.25.501397
Autor:
Xue Li, Johannes Backs, Felix Lasitschka, Hugo A. Katus, Florian Sicklinger, Tim Christian Kuhn, Andreas Jungmann, Florian Leuschner, Jeroen Krijgsveld, Oliver J. Müller, Ingmar Sören Meyer, Sonja Burkert-Rettenmaier, Johannes Knobel
Publikováno v:
Circulation
Background: Acute occlusion of a coronary artery results in swift tissue necrosis. Bordering areas of the infarcted myocardium can also experience impaired blood supply and reduced oxygen delivery, leading to altered metabolic and mechanical processe
Autor:
Ingmar Sören Meyer, Xue Li, David Voehringer, Norbert Frey, Hugo A. Katus, Christina Mertens, Kory J. Lavine, Julia K. Meier, Severin Dicks, Moritz P. Kornadt, Melanie Boerries, Florian Leuschner, Daniel Radtke, Florian Sicklinger, Y. Zhang, Gabriele Schramm, Tobias Terzer, Tim Christian Kuhn, Jyoti Patel
Publikováno v:
J Clin Invest
The inflammatory response after myocardial infarction (MI) is a precisely regulated process that greatly affects subsequent remodeling. Here, we show that basophil granulocytes infiltrated infarcted murine hearts, with a peak occurring between days 3
Autor:
Ingmar Sören Meyer, Florian Leuschner
Publikováno v:
Basic research in cardiology. 113(6)
Various cell types are involved in the healing process after myocardial infarction (MI). Besides cardiac resident cells (such as cardiomyocytes, fibroblasts and endothelial cells) already present at the lesion site, a massive influx of leukocytes (ma
Autor:
Hugo A. Katus, Ingmar Sören Meyer, Stefan E. Hardt, Susann Werkmeister, Michael Boutros, Jan Haas, Felix Lasitschka, Matthias Nahrendorf, Florian Leuschner, Oliver J. Müller, Min Zhang, Andreas Jungmann, Christoph Dieterich
Publikováno v:
EMBO Molecular Medicine
A disturbed inflammatory response following myocardial infarction (MI) is associated with poor prognosis and increased tissue damage. Monocytes are key players in healing after MI, but little is known about the role of the cardiac niche in monocyte a