Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Inge Snoeren"'
Autor:
Hélène Gleitz, Inge Snoeren, Stijn Fuchs, James Nagai, Aurélien Dugourd, Charlotte Boys, Vanessa Klöker, Jessica Pritchard, Eric Bindels, Peter André Willem Te Boekhorst, Martina Crysandt, Julio Saez-Rodriguez, Thomas Vogl, Rafael Kramann, Ivan Costa, Rebekka Schneider
Publikováno v:
HemaSphere, Vol 7, p e36177f7 (2023)
Externí odkaz:
https://doaj.org/article/0db4256c0e524b6dbd91b7fab558df1f
Autor:
Fiona Kerr, Oyinkan Sofola-Adesakin, Dobril K Ivanov, Jemma Gatliff, Beatriz Gomez Perez-Nievas, Hélène C Bertrand, Pedro Martinez, Rebecca Callard, Inge Snoeren, Helena M Cochemé, Jennifer Adcott, Mobina Khericha, Jorge Iván Castillo-Quan, Geoffrey Wells, Wendy Noble, Janet Thornton, Linda Partridge
Publikováno v:
PLoS Genetics, Vol 13, Iss 3, p e1006593 (2017)
Nrf2, a transcriptional activator of cell protection genes, is an attractive therapeutic target for the prevention of neurodegenerative diseases, including Alzheimer's disease (AD). Current Nrf2 activators, however, may exert toxicity and pathway ove
Externí odkaz:
https://doaj.org/article/0da1b265333b4d3d89cda4194d8f09d6
Autor:
Bella Banjanin, James Nagai, Yevin Mun, Inge Snoeren, Stijn Fuchs, Marek Weiler, Fabian Kiessling, Twan Lammers, Kishor Sivaraj, Ralf Adams, Cesar Nombela-Arrieta, Ivan Costa, Rebekka K. Schneider
Publikováno v:
Blood. 140:3856-3857
Autor:
Jessica E. Pritchard, Juliette Pearce, Inge Snoeren, Stijn Fuchs, Katrin Götz, Silke Wagner, Kathrin Olschok, Nicolas Chatain, Steffen Koschmieder, Marie-Luise Berres, Ivan Costa, Helene Gleitz, Rafael Kramann, Rebekka K. Schneider
Publikováno v:
Blood. 140:6728-6729
Autor:
Dobril Ivanov, Teresa Niccoli, Linda Partridge, Benjamin Aleyakpo, Inge Snoeren, Daniel K. Fabian, Fiona Kerr, Janet M. Thornton, Oyinkan Sofola-Adesakin, Adam Cryar, Jennifer Adcott
Publikováno v:
Brain Commun
Brain Communications
Brain Communications
Accumulation of amyloid beta peptides is thought to initiate the pathogenesis of Alzheimer's disease. However, the precise mechanisms mediating their neurotoxicity are unclear. Our microarray analyses show that, in Drosophila models of amyloid beta 4
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::786bea2882571eb2415fc70aaeca055e
https://hdl.handle.net/21.11116/0000-000B-2D76-F
https://hdl.handle.net/21.11116/0000-000B-2D76-F