Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Ines Zaaber"'
Autor:
Hela Marmouch, Oumaima Inoubli, Souhir Mestiri, Hana Nejehi, Nabil Abid, Ines Zaaber, Abir Omrani
Publikováno v:
International Journal of Immunogenetics. 47:294-308
Autoimmune thyroid diseases (AITD) including Graves' disease (GD) and Hashimoto's thyroiditis (HT) are complex genetic diseases. Th2 cytokines act on the development of AITD. This study was conducted on Tunisian patients with AITD to investigate the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::99c850bbd4ad9ac92f81fd116c413b31
https://doi.org/10.1111/iji.12472
https://doi.org/10.1111/iji.12472
Publikováno v:
Acta Endocrinol (Buchar)
Background Autoimmune thyroid diseases (AITD) including Graves' disease (GD) and Hashimoto's thyroiditis (HT) are complex genetic diseases. TSHR is considered as candidate gene in GD. This finding prompted us to investigate the association of TSHR ge
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5cfd5c5c973555e29048fdb0b615dbe4
https://pubmed.ncbi.nlm.nih.gov/32685031
https://pubmed.ncbi.nlm.nih.gov/32685031
Autor:
Besma Bel Haj Jrad Tensaout, Souhir Mestiri, Helena Murray, Sophie Visvikis-Siest, Silvia Mahjoub, Marc Rancier, Abdelsalam Saleh, Maria G. Stathopoulou, Jérôme Chatelin, Ines Zaaber, Peter Fitzgerald, Said El Shamieh, Hela Marmouch, John Victor Lamont, Khaled Said, Christine Masson
Publikováno v:
Autoimmunity
Autoimmunity, Taylor & Francis, 2016, 49 (6), pp.366-372. ⟨10.1080/08916934.2016.1199019⟩
Autoimmunity, Taylor & Francis, 2016, 49 (6), pp.366-372. ⟨10.1080/08916934.2016.1199019⟩
International audience; The aim of this study was to assess the relationships between five different splice isoforms of VEGF mRNA and its plasma levels in individuals treated for autoimmune thyroid diseases (AITD); mainly Graves' disease (GD) and Has
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::958d1528fcc3648843a00c8e47d45e0c
https://pubmed.ncbi.nlm.nih.gov/27494076
https://pubmed.ncbi.nlm.nih.gov/27494076
Autor:
Hounayda Hammedi, Hela Marmouch, Souhir Mestiri, Khaled Said, Besma Bel Hadj Jrad Tensaout, Ines Zaaber, Silvia Mahjoub
Publikováno v:
Immunological investigations. 45(4)
Autoimmune thyroid diseases (AITD) including Graves' disease (GD) and Hashimoto's thyroiditis (HT) are complex genetic diseases. Cytokines IL-1B and IL-4 play a role in the pathogenesis of AITD. This study was conducted on Tunisian patients with GD o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f226d9561c8a9b0e9c83f9fcc3822f72
https://pubmed.ncbi.nlm.nih.gov/27100882
https://pubmed.ncbi.nlm.nih.gov/27100882
Autor:
Marc Rancier, Christine Masson, Souhir Mestiri, Maria G. Stathopoulou, Ines Zaaber, Selvia Mahjoub, Mary Jo Kurth, Khaled Said, John Victor Lamont, Sophie Visvikis-Siest, Peter Fitzgerald, Besma Bel Hadj Jrad Tensaout, Abdelsalam Saleh, Helena Murray, Hela Marmouch
Publikováno v:
Autoimmunity
Autoimmunity, Taylor & Francis, 2016, 49 (4), pp.229-235. ⟨10.3109/08916934.2016.1151005⟩
Autoimmunity, Taylor & Francis, 2016, 49 (4), pp.229-235. ⟨10.3109/08916934.2016.1151005⟩
International audience; Autoimmune thyroid diseases (AITD), including Graves' disease (GD) and Hashimoto thyroiditis (HT), are complex multifactorial diseases. Vascular endothelial growth factor (VEGF) is implicated in various inflammatory diseases,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3118a945d26dbee22e3cb1973f44b809
https://hal.univ-lorraine.fr/hal-01708503
https://hal.univ-lorraine.fr/hal-01708503
Publikováno v:
Biomedical Research and Therapy. 2
Graves’ disease (GD) is a complex genetic autoimmune thyroid diseases (AITD). TSHR is considered as candidate gene in GD. IL1RN gene shown to be related to the pathogenesis of a number of autoimmune diseases. These finding prompted us to investigat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8bb84f95332a3b520981ed241e1cd81c
https://doi.org/10.7603/s40730-015-0030-0
https://doi.org/10.7603/s40730-015-0030-0