Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Imtiyaz Najar"'
Autor:
Sameer Ahmad Guru, Rashid Mir, Musadiq Bhat, Imtiyaz Najar, Mariyam Zuberi, Mamta Sumi, Mirza Masroor, Naresh Gupta, Alpana Saxena
Publikováno v:
Tumor Biology, Vol 39 (2017)
Platelet-derived growth factor receptor has been implicated in many malignant and non-malignant diseases. Platelet-derived growth factor receptor-α is a tyrosine kinase and a side target for imatinib, a revolutionary drug for the treatment of chroni
Externí odkaz:
https://doaj.org/article/9e1d493e05514523a86ef51212ac3534
Autor:
Sameer Ahmad Guru, Rashid Mir, Imtiyaz Najar, Gauri Gandhi, Nita Khurana, Prakash Chandra Ray, Jamsheed Javid, Prasant Yadav, Sagar Dholariya, Alpana Saxena, Masroor Mirza, Mariyam Zuberi
Publikováno v:
Clinical Ovarian and Other Gynecologic Cancer. 7:33-39
Background To investigate the methylation pattern in promoter region of RASSF1A and PTEN genes in epithelial ovarian cancer patients in North India. Patients and Methods Fifty patients and 20 healthy controls were studied. Isolation of genomic DNA fr
Autor:
Sameer Ahmad Guru, Alpana Saxena, Rashid Mir, Pramod Lali, Imtiyaz Najar, Mariyam Zuberi, Naresh Gupta, Mamta P Sumi
Publikováno v:
Annals of Oncology. 30:vi110
Background Mutations in Janus kinase 2 (JAK2) genes are the genetic characteristic of BCR-ABL1-negative myeloproliferative neoplasms (MPN). JAK2 V617F mutation is frequently found in Polycythemia Vera (PV) and Essential Thrombocythemia (ET). We aimed
Autor:
Alpana Saxena, Mariyam Zuberi, Rashid Mir, Naresh Gupta, Pramod Lali, Sameer Ahmad Guru, Imtiyaz Najar, Mamta P. Alpana
Publikováno v:
Annals of Oncology. 30:vi110
Background Escalated PD-L1 expression is reported in many cancer types initiating an immune escape mechanism. This led to the development of checkpoint inhibitors against PD-1/PD-L1. However, the mechanisms underlying escalated production of PD-L1 in
Publikováno v:
Cancer Research. 79:831-831
Chronic myelogenous leukemia (CML) has a typical progressive course with transition from a less aggressive chronic phase to a terminal more aggressive blast crisis. The molecular mechanisms leading to CML disease progression remain to be elucidated.
Autor:
Alpana Saxena, P. C. Ray, Rashid Mir, Prasant Yadav, Shazia Farooq, Imtiyaz Najar, Musadiq Bhat, Mariyam Zuberi, Naresh Gupta, Mirza Masroor, Sameer Ahmad Guru, Jamsheed Javaid
Publikováno v:
Leukemialymphoma. 58(7)
RIZ1 encodes a retinoblastoma (Rb)-interacting zinc finger protein, is commonly lost or expressed at reduced levels in cancer cells. The RIZ1 gene locus commonly undergoes LOH in many cancers. Here, we analyzed Proline insertion-deletion polymorphism