Zobrazeno 1 - 10
of 22
pro vyhledávání: '"Imen Darragi"'
Autor:
Ines Cherif, Ghassen Kharroubi, Imen Darragi, Soumaya El Benna, Adel Gharbi, Amor Baccouche, Cyrine Souissi, Olfa Bahri, Melika Ben Ahmed, Jihene Bettaieb
Publikováno v:
Libyan Journal of Medicine, Vol 19, Iss 1 (2024)
ABSTRACTThis study aimed to assess the kinetics of antibodies against the SARS-CoV-2, following natural infection in a cohort of employees of the Institut Pasteur de Tunis (IPT) and to assess the risk of reinfection over a 12-months follow-up period.
Externí odkaz:
https://doaj.org/article/6b218e47a94943a2afb814a59bd90aa2
Autor:
Nawel Trabelsi, Ghada Bouguerra, Faten Haddad, Monia Ouederni, Imen Darragi, Imen Boudrigua, Dorra Chaouachi, Mbarka Barmat, Chaker Fouzai, Mohamed Bejaoui, Samia Menif, Imen Kraiem, Salem Abbes
Publikováno v:
Cellular Physiology and Biochemistry, Vol 55, Iss 1, Pp 117-129 (2021)
Externí odkaz:
https://doaj.org/article/dfaaabcdd6f94502879de79c424c2da2
Autor:
Leila Chaouch, Miniar Kalai, Manel Ben Jbara, Arij Ben Chaabene, Imen Darragi, Dorra Chaouachi, Fethi Mallouli, Raouf Hafsia, Abderraouf Ghanem, Salem Abbes
Publikováno v:
Biomedical Papers, Vol 159, Iss 1, Pp 145-149 (2015)
Aims: The skeletal manifestations of sickle cell disease are the result of changes in bone and bone marrow caused by chronic tissue hypoxia that is exacerbated by episodic occlusion of the microcirculation by the abnormal sickle cells. Furthermore, t
Externí odkaz:
https://doaj.org/article/ff6c76f4151f4f91bc425bf5c7cf1453
Autor:
Mouna, Jaouani, Nadia, Hamdi, Leila, Chaouch, Miniar, Kalai, Fethi, Mellouli, Imen, Darragi, Imen, Boudriga, Dorra, Chaouachi, Mohamed, Bejaoui, Salem, Abbes
Publikováno v:
In Egyptian Journal of Medical Human Genetics July 2016 17(3):265-270
Autor:
Mohamed Bejaoui, Samia Menif, Mbarka Barmat, Salem Abbes, Imen Boudrigua, Imen Darragi, Imen Kraiem, Nawel Trabelsi, Dorra Chaouachi, Monia Ouederni, Faten Haddad, Ghada Bouguerra, Chaker Fouzai
Publikováno v:
Cellular Physiology and Biochemistry, Vol 55, Iss 1, Pp 117-129 (2021)
Background/Aims: Hereditary Spherocytosis (HS) is the most common erythrocyte membrane disorder causing hemolytic anemia. The wide heterogeneity of both clinical and laboratory manifestations of HS contributes to difficulties associated with the diag
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::30b3c518fb926b1127e15a7e77018cf9
https://pubmed.ncbi.nlm.nih.gov/33667330
https://pubmed.ncbi.nlm.nih.gov/33667330
Autor:
Nawel Trabelsi, Aminetou Abba, Mohamed Lemine Ould Salem, Sidi M. Ghaber, Faten Haddad, Salem Abbes, Imen Darragi
Publikováno v:
Hemoglobin. 42:7-10
Unstable hemoglobins (Hbs) are a group of Hb disorders that could be the origin of chronic hemolytic anemia. Most of these disorders are caused by point mutations taking place in the globin genes and affecting the stability of the Hb molecule. They a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cd571ec90b2100d1516b4feffe5f8f7c
https://doi.org/10.1080/03630269.2018.1428620
https://doi.org/10.1080/03630269.2018.1428620
Autor:
Houssem Sellami, Imen Darragi, Samia Mnif, Dorra Chaouachi, Leila Chaouch, Miniar Kalai, Imen Boudrigua, Salem Abbes
Publikováno v:
Journal of pediatric hematology/oncology. 42(1)
OBJECTIVES The 5' upstream region of the HBG1 gene plays a very important role in the expression of fetal hemoglobin (HbF). In contrast, increased HbF levels can inhibit the deoxygenation-induced polymerization of sickle hemoglobin (α2βS2), which l
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::37848dc001c29eeb5464935a43f6a4da
https://pubmed.ncbi.nlm.nih.gov/31688634
https://pubmed.ncbi.nlm.nih.gov/31688634
Autor:
Emna Barkaoui, Imen Boudrigua, Faten Haddad, Nawel Trabelsi, Samia Menif, Salem Abbes, Dorra Chaouachi, Imen Darragi, Leila Chaouch, Mouna Jaouani
Publikováno v:
European Journal of Medical Genetics. 64:104139
Introduction Unconjugated hyperbilirubinemia (UCB) is a feature of Gilbert's syndrome (GS) and Crigler-Najjar's syndrome (CNS), which are two hereditary defects in bilirubin metabolism. Both syndromes are linked to mutations in the UGT1A1 gene, which
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::96b0a87fcea4a07002eaf1003abb35ce
https://doi.org/10.1016/j.ejmg.2021.104139
https://doi.org/10.1016/j.ejmg.2021.104139
Autor:
Imen Darragi, Salem Abbes, Imen Boudrigua, Miniar Kalai, Houyem Ouragini, Mouna Ben Sassi, Raouf Hafsia, Leila Chaouch, Imen Moumni, Dorra Chaouachi
Publikováno v:
Acta Haematologica Polonica. 47:242-247
Background and aim Sickle cell anemia (SCA) is characterized by variable patterns of clinical expression. Polymorphisms linked to different genes have been associated with specific complications of the disease. Herein, we focused on the study of the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::706def2ca8b8575c97ae2fd0edec4602
https://doi.org/10.1016/j.achaem.2016.10.004
https://doi.org/10.1016/j.achaem.2016.10.004