Insulin, CCAAT/enhancer-binding proteins and lactate regulate the human 11β-hydroxysteroid dehydrogenase type 2 gene expression in colon cancer cell lines

Autor: Andrieu, Thomas, Fustier, Pierre, Alikhani-Koupaei, Rasoul, Ignatova, Irena D, Guettinger, Andreas, Frey, Felix J, Frey, Brigitte M, Alikhani, Rasoul, Ignatova, Irena Darieva, Güttinger, Andreas, Frey, Felix, Frey, Brigitte

Publikováno v: PLoS ONE
PLoS ONE, Vol 9, Iss 8, p e105354 (2014)
Andrieu, Thomas; Fustier, Pierre; Alikhani-Koupaei, Rasoul; Ignatova, Irena D; Guettinger, Andreas; Frey, Felix J; Frey, Brigitte M; Andrieu, Thomas; Fustier, Pierre; Alikhani, Rasoul; Ignatova, Irena Darieva; Güttinger, Andreas; Frey, Felix; Frey, Brigitte (2014). Insulin, CCAAT/enhancer-binding proteins and lactate regulate the human 11β-hydroxysteroid dehydrogenase type 2 gene expression in colon cancer cell lines. PLoS ONE, 9(8), e105354. Public Library of Science 10.1371/journal.pone.0105354

11β-Hydroxysteroid dehydrogenases (11beta-HSD) modulate mineralocorticoid receptor transactivation by glucocorticoids and regulate access to the glucocorticoid receptor. The isozyme 11beta-HSD2 is selectively expressed in mineralocorticoid target ti

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::85b77cf3e4d239e3068c777035b037d1
https://pubmed.ncbi.nlm.nih.gov/25133511

Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
K zobrazení výsledku je třeba se přihlásit.

Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
K zobrazení výsledku je třeba se přihlásit.

Tumor necrosis factor-α upregulates 11 β-hydroxysteroid dehydrogenase type 1 expression by CCAAT/enhancer binding protein-β in HepG2 cells.

Autor: Ignatova, Irena D., Kostadinova, Radina M., Goldring, Christopher E., Nawrocki, Andrea R., Frey, Felix J., Frey, Brigitte M.

Publikováno v: American Journal of Physiology: Endocrinology & Metabolism; Feb2009, Vol. 296, pE367-E377, 11p, 8 Graphs

Impaired protein stability of 11beta-hydroxysteroid dehydrogenase type 2: a novel mechanism of apparent mineralocorticoid excess.

Autor: Atanasov AG; Department of Nephrology and Hypertension, University of Berne, Freiburgstrasse 15, 3010 Berne, Switzerland., Ignatova ID, Nashev LG, Dick B, Ferrari P, Frey FJ, Odermatt A

Publikováno v: Journal of the American Society of Nephrology : JASN [J Am Soc Nephrol] 2007 Apr; Vol. 18 (4), pp. 1262-70. Date of Electronic Publication: 2007 Feb 21.