Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Ibuki Harada"'
Autor:
Hiroshi Okuda, Ryo Miyamoto, Satoshi Takahashi, Takeshi Kawamura, Juri Ichikawa, Ibuki Harada, Tomohiko Tamura, Akihiko Yokoyama
Publikováno v:
Nature Communications, Vol 13, Iss 1, Pp 1-15 (2022)
The establishment of a mouse model of MLL-AF4-induced leukaemia is a challenge as the introduction of fusion gene of human MLL-AF4 does not cause leukaemia in mice. Here the authors reveal that MLL-AF4 is post-transcriptionally regulated by RNA-bindi
Externí odkaz:
https://doaj.org/article/87bc68820b034fe487fc80b5531a4e43
Autor:
Ibuki Harada, Haruka Sasaki, Koichi Murakami, Akira Nishiyama, Jun Nakabayashi, Motohide Ichino, Takuya Miyazaki, Ken Kumagai, Kenji Matsumoto, Maki Hagihara, Wataru Kawase, Takayoshi Tachibana, Masatsugu Tanaka, Tomoyuki Saito, Heiwa Kanamori, Hiroyuki Fujita, Shin Fujisawa, Hideaki Nakajima, Tomohiko Tamura
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-10 (2021)
Abstract Chronic myeloid leukemia (CML) is a form of myeloproliferative neoplasm caused by the oncogenic tyrosine kinase BCR-ABL. Although tyrosine kinase inhibitors have dramatically improved the prognosis of patients with CML, several problems such
Externí odkaz:
https://doaj.org/article/ca1582ae26944192807940863f8aa2cc
Autor:
Hiroyuki Fujita, Ibuki Harada, Koichi Murakami, Hideaki Nakajima, Akira Nishiyama, Heiwa Kanamori, Maki Hagihara, Takuya Miyazaki, Shin Fujisawa, Jun Nakabayashi, Motohide Ichino, Wataru Kawase, Tomoyuki Saito, Takayoshi Tachibana, Tomohiko Tamura, Masatsugu Tanaka, Haruka Sasaki, Kenji Matsumoto, Ken Kumagai
Publikováno v:
Scientific Reports
Scientific Reports, Vol 11, Iss 1, Pp 1-10 (2021)
Scientific Reports, Vol 11, Iss 1, Pp 1-10 (2021)
Chronic myeloid leukemia (CML) is a form of myeloproliferative neoplasm caused by the oncogenic tyrosine kinase BCR-ABL. Although tyrosine kinase inhibitors have dramatically improved the prognosis of patients with CML, several problems such as resis