Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Ian M Dixon"'
Autor:
Matthew D. Martens, Nivedita Seshadri, Lucas Nguyen, Donald Chapman, Elizabeth S. Henson, Bo Xiang, Landon Falk, Arielys Mendoza, Sunil Rattan, Jared T. Field, Philip Kawalec, Spencer B. Gibson, Richard Keijzer, Ayesha Saleem, Grant M. Hatch, Christine A. Doucette, Jason M. Karch, Vernon W. Dolinsky, Ian M. Dixon, Adrian R. West, Christof Rampitsch, Joseph W. Gordon
Publikováno v:
Cell Death and Disease, Vol 12, Iss 12, Pp 1-16 (2021)
Abstract Systemic hypoxia is a common element in most perinatal emergencies and is a known driver of Bnip3 expression in the neonatal heart. Bnip3 plays a prominent role in the evolution of necrotic cell death, disrupting ER calcium homeostasis and i
Externí odkaz:
https://doaj.org/article/e1343dcb2c5b484c91d04a2bde3e5a8e
Autor:
Navid, Koleini, Barbara E, Nickel, Raghu S, Nagalingam, Natalie M, Landry, Robert R, Fandrich, David Y C, Cheung, Ian M, Dixon, Michael P, Czubryt, Davinder S, Jassal, Peter A, Cattini, Elissavet, Kardami
Publikováno v:
Cell and tissue research. 385(3)
Fibroblast growth factor 2 (FGF2), produced as high (Hi-) and low (Lo-) molecular weight isoforms, is implicated in cardiac response to injury. The role of endogenous FGF2 isoforms during chronic stress is not well defined. We investigated the effect
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::59e8067b0143dede31da8264751d36a7
https://pubmed.ncbi.nlm.nih.gov/34057573
https://pubmed.ncbi.nlm.nih.gov/34057573
Autor:
Krista L Filomeno, Sunil G Rattan, Sheri Bage, Matthew Zeglinski, Michael P Czubryt, Ian M Dixon
Publikováno v:
Circulation Research. 117
Introduction: Coronary heart disease is causal to myocardial infarction (MI) and cardiac fibrosis. Upon ischemic myocardial injury, resident cardiac fibroblasts phenoconvert to myofibroblasts and synthesize large amounts of fibrillar collagens to pro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::bf16775fcf308aee41b7f41e8b2a2e1b
https://doi.org/10.1161/res.117.suppl_1.372
https://doi.org/10.1161/res.117.suppl_1.372