Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Ian J. Sutton"'
Autor:
Malini Visweswaran, Kevin Hendrawan, Jennifer C. Massey, Melissa L. Khoo, Carole D. Ford, John J. Zaunders, Barbara Withers, Ian J. Sutton, David D. F. Ma, John J. Moore
Publikováno v:
Annals of Clinical and Translational Neurology, Vol 9, Iss 2, Pp 206-220 (2022)
Abstract Objective Autologous haematopoietic stem cell transplantation (AHSCT) has the potential to induce sustained periods of disease remission in multiple sclerosis (MS), which is an inflammatory disease of the central nervous system (CNS) charact
Externí odkaz:
https://doaj.org/article/8d61bbe5a2804e2db6416507495ffc03
Publikováno v:
Frontiers in Immunology, Vol 9 (2018)
Multiple sclerosis (MS) is an inflammatory disorder of the central nervous system where evidence implicates an aberrant adaptive immune response in the accrual of neurological disability. The inflammatory phase of the disease responds to immunomodula
Externí odkaz:
https://doaj.org/article/e75c4de6cb94405991274b107d303878
Autor:
Malini Visweswaran, Kevin Hendrawan, Jennifer C. Massey, Melissa L. Khoo, Carole D. Ford, John J. Zaunders, Barbara Withers, Ian J. Sutton, David D. F. Ma, John J. Moore
Publikováno v:
Annals of clinical and translational neurology. 9(2)
Autologous haematopoietic stem cell transplantation (AHSCT) has the potential to induce sustained periods of disease remission in multiple sclerosis (MS), which is an inflammatory disease of the central nervous system (CNS) characterised by demyelina
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::84bcffe3fc33488fd54c39de4e4e82e3
https://pubmed.ncbi.nlm.nih.gov/35106961
https://pubmed.ncbi.nlm.nih.gov/35106961
Autor:
Ian J. Sutton, James I. K. Last, Susan J. Ritchie, Hugh J. Harrington, Philip J. Byrd, A. Malcolm R. Taylor
Publikováno v:
Annals of Neurology. 55:891-895
Ataxia telangiectasia (A-T) is an autosomal recessive neurodegenerative disorder that arises because of mutations in the ATM gene. The 5762ins137 A→G point mutation activates a cryptic splice donor site resulting in a 137bp intronic insert being ab
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2d8332252c9dac782573ed9f00711ad8
https://doi.org/10.1002/ana.20139
https://doi.org/10.1002/ana.20139