Zobrazeno 1 - 10
of 44
pro vyhledávání: '"Ian D Haidl"'
Autor:
Sharon A. Oldford, Suzanne P. Salsman, Liliana Portales‐Cervantes, Raidan Alyazidi, Robert Anderson, Ian D Haidl, Jean S. Marshall
Publikováno v:
Immunity, Inflammation and Disease, Vol 6, Iss 1, Pp 176-189 (2018)
Abstract Background Mast cells are resident immune effector cells, often studied in the context of allergic disease. Found in substantial numbers at sites of potential infection they are increased at sites of angiogenesis and can be pivotal for the s
Externí odkaz:
https://doaj.org/article/068a1d98bb8b47b595d32e0d4620df7d
Autor:
Michael G Brown, Sarah M McAlpine, Yan Y Huang, Ian D Haidl, Ayham Al-Afif, Jean S Marshall, Robert Anderson
Publikováno v:
PLoS ONE, Vol 7, Iss 3, p e34055 (2012)
Dengue hemorrhagic fever and/or dengue shock syndrome represent the most serious pathophysiological manifestations of human dengue virus infection. Despite intensive research, the mechanisms and important cellular players that contribute to dengue di
Externí odkaz:
https://doaj.org/article/83932672101b43b29516d14dcfe88f64
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
IntroductionChlamydia trachomatis (C. trachomatis) is a Gram-negative obligate intracellular bacterium that causes reproductive tract complications in women, including ectopic pregnancies and tubal factor infertility. We hypothesized that mast cells,
Externí odkaz:
https://doaj.org/article/f01588e5dca842319608163330d3eeb4
Autor:
Jean-Francois Légaré, Stephanie Legere, Impart Investigator Team Canada, Ian D. Haidl, Keith R. Brunt, Mathieu Castonguay, Jean S. Marshall
Publikováno v:
Journal of Molecular and Cellular Cardiology. 149:15-26
Fibrotic remodelling of the atria is poorly understood and can be regulated by myocardial immune cell populations after injury. Mast cells are resident immune sentinel cells present in the heart that respond to tissue damage and have been linked to f
Publikováno v:
Immunology and Cell Biology
Natural killer (NK) cells are innate effector cells with critical roles not only in tumor immunosurveillance and viral immunity, but also in bacterial and fungal infections. Toll‐like receptor 2 (TLR2) can be important in the early and sustained im
Autor:
Drew Slauenwhite, J G Hanly, Thomas B. Issekutz, Jean S. Marshall, Aniko Malik, Sarah M. McAlpine, Ian D. Haidl
Publikováno v:
Arthritis & Rheumatology. 72:1091-1102
OBJECTIVE Rheumatoid arthritis (RA) is a chronic inflammatory disease mediated through complex immunologic pathways. Among RA patients receiving low-dose methotrexate (MTX) monotherapy, approximately one-half exhibit a meaningful clinical response wi
Autor:
Judit Fazekas-Singer, Ina Herrmann, Giulia Pellizzari, Rodolfo Bianchini, Karin Hufnagl, Sophia N. Karagiannis, Erika Jensen-Jarolim, Jean S. Marshall, Jelena Gotovina, Ian D. Haidl
Publikováno v:
Allergy
Publikováno v:
Scientific Reports
Scientific Reports, Vol 11, Iss 1, Pp 1-10 (2021)
Scientific Reports, Vol 11, Iss 1, Pp 1-10 (2021)
Histamine receptor 2 (H2R) blockade is commonly used in patients with gastric, duodenal ulcers or gastroesophageal reflux disease. Beyond the gastrointestinal tract, H2R is expressed by multiple immune cells, yet little is known about the immunomodul
Autor:
Suzanne P. Salsman, Sharon A. Oldford, Liliana Portales-Cervantes, Jean S. Marshall, Ian D. Haidl, Robert Anderson, Raidan Alyazidi
Publikováno v:
Immunity, Inflammation and Disease
Immunity, Inflammation and Disease, Vol 6, Iss 1, Pp 176-189 (2018)
Immunity, Inflammation and Disease, Vol 6, Iss 1, Pp 176-189 (2018)
Background Mast cells are resident immune effector cells, often studied in the context of allergic disease. Found in substantial numbers at sites of potential infection they are increased at sites of angiogenesis and can be pivotal for the sensing an
Autor:
Manoj K. Saraswat, Ian D. Haidl, Alec Falkenham, Karen J. Buth, Chloe Wong, Tanya Myers, Jean S. Marshall, Jahanara Begum, Kareem Gawdat, Jean-Francois Légaré
Publikováno v:
ESC Heart Failure. 5:107-114
Aims Previous studies have examined risk factors for the development of heart failure (HF) subsequent to acute coronary syndrome (ACS). Our study seeks to clarify the clinical variables that best characterize patients who remain free from HF after co