Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Ian Baudi"'
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
Hepatitis B virus (HBV) remains a leading cause of liver-related morbidity and mortality through chronic hepatitis that may progress to liver cirrhosis and cancer. The central role played by HBV-specific CD8+ T cells in the clearance of acute HBV inf
Externí odkaz:
https://doaj.org/article/6550abec135044728b6a76643b74f741
Autor:
Ian Baudi, Masanori Isogawa, Federica Moalli, Masaya Onishi, Keigo Kawashima, Yuji Ishida, Chise Tateno, Yusuke Sato, Hideyoshi Harashima, Hiroyasu Ito, Tetsuya Ishikawa, Takaji Wakita, Matteo Iannacone, Yasuhito Tanaka
Publikováno v:
PLoS Pathogens, Vol 17, Iss 5, p e1009228 (2021)
Virus infection, such as hepatitis B virus (HBV), occasionally causes endoplasmic reticulum (ER) stress. The unfolded protein response (UPR) is counteractive machinery to ER stress, and the failure of UPR to cope with ER stress results in cell death.
Externí odkaz:
https://doaj.org/article/e016bc46b1164d8e848ca52ceee39270
Autor:
Keigo Kawashima, Masanori Isogawa, Masaya Onishi, Ian Baudi, Satoru Saito, Atsushi Nakajima, Takashi Fujita, Yasuhito Tanaka
Publikováno v:
JCI Insight, Vol 6, Iss 3 (2021)
Hepatitis B virus–specific (HBV-specific) CD8+ T cells fail to acquire effector functions after priming in the liver, but the molecular basis for the dysfunction is poorly understood. By comparing the gene expression profile of intrahepatically pri
Externí odkaz:
https://doaj.org/article/fedaafc8bd0c4c418a48b78e34ffe45a
Autor:
Masaya Onishi, Masanori Isogawa, Yasuhito Tanaka, Takashi Fujita, Ian Baudi, Satoru Saito, Keigo Kawashima, Atsushi Nakajima
Publikováno v:
JCI Insight, Vol 6, Iss 3 (2021)
JCI Insight
JCI Insight
Hepatitis B virus–specific (HBV-specific) CD8+ T cells fail to acquire effector functions after priming in the liver, but the molecular basis for the dysfunction is poorly understood. By comparing the gene expression profile of intrahepatically pri
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6ebda26f1ff8422fbccd02e056edef05
https://doaj.org/article/fedaafc8bd0c4c418a48b78e34ffe45a
https://doaj.org/article/fedaafc8bd0c4c418a48b78e34ffe45a
Autor:
Hideyoshi Harashima, Ian Baudi, Hiroyasu Ito, Federica Moalli, Matteo Iannacone, Masaya Onishi, Masanori Isogawa, Tetsuya Ishikawa, Keigo Kawashima, Yasuhito Tanaka, Takaji Wakita, Yusuke Sato
Virus infection, such as hepatitis B virus (HBV), often causes endoplasmic reticulum (ER) stress. The unfolded protein response (UPR) is counteractive machinery to ER stress, and the failure of UPR to cope with ER stress results in cell death. Mechan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::993753d1f79a4b35d19ea7717a4cfc82
https://doi.org/10.1101/2020.12.22.423938
https://doi.org/10.1101/2020.12.22.423938
Autor:
Keigo Kawashima, Chise Tateno, Federica Moalli, Hideyoshi Harashima, Ian Baudi, Yasuhito Tanaka, Masanori Isogawa, Hiroyasu Ito, Masaya Onishi, Matteo Iannacone, Yuji Ishida, Takaji Wakita, Tetsuya Ishikawa, Yusuke Sato
Publikováno v:
PLoS Pathogens, Vol 17, Iss 5, p e1009228 (2021)
PLoS Pathogens
PLoS Pathogens
Virus infection, such as hepatitis B virus (HBV), occasionally causes endoplasmic reticulum (ER) stress. The unfolded protein response (UPR) is counteractive machinery to ER stress, and the failure of UPR to cope with ER stress results in cell death.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6f7b8eac449f0d4ad81cd955e1ab505e
https://doi.org/10.1371/journal.ppat.1009228
https://doi.org/10.1371/journal.ppat.1009228
Autor:
Masanori Isogawa, Sayuki Iijima, Shuko Murakami, Sekesai Mtapuri-Zinyowera, Ian Baudi, Yasumasa Iwatani, Atsuko Hachiya, Nyasha Chin'ombe, Yasuhito Tanaka
Publikováno v:
Journal of Medical Virology. 89:257-266
The objective of this study was to determine the prevalence of co-infection with hepatitis B virus (HBV) and human immunodeficiency virus (HIV) and the genetic characteristics of both viruses among pre-HIV-treatment patients in Harare, Zimbabwe. This
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::bd2e8a1e89bce0061ef719405ddc6b6a
https://doi.org/10.1002/jmv.24641
https://doi.org/10.1002/jmv.24641
Publikováno v:
International Journal of Molecular Sciences, Vol 21, Iss 3, p 949 (2020)
International Journal of Molecular Sciences
International Journal of Molecular Sciences
The hepatitis B virus (HBV) cannot be removed completely from infected hepatocytes, owing to the presence of intrahepatic covalently closed circular DNA (cccDNA). As chronic hepatitis B (CHB) can progress to cirrhosis and hepatocellular carcinoma (HC
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::eaa400065975bd92ad9300eb6496f319
https://doi.org/10.3390/ijms21030949
https://doi.org/10.3390/ijms21030949