Zobrazeno 1 - 10
of 32
pro vyhledávání: '"ITSUO KATOH"'
Autor:
Takaki Hori, Kazuma Maisawa, Taisuke Nakayama, Hirotsugu Kurobe, Itsuo Katoh, Hiroshi Ishitoya, Hitoshi Sogabe, Tetsuya Kitagawa
Publikováno v:
Japanese Journal of Cardiovascular Surgery. 38:160-164
Viability of cryopreserved semilunar valves: an evaluation of cytosolic and mitochondrial activities
Autor:
Takaki Hori, Tetsuya Kitagawa, Masashi Kano, Osamu Yasuta, Yutaka Masuda, Itsuo Katoh, Takashi Tominaga
Publikováno v:
The Annals of Thoracic Surgery. 70:792-795
Background . Despite long-standing, widespread use of cryopreserved allografts, the basic cellular biology of these tissues is still yet unknown. The present investigation was undertaken to study cryopreserved heart valves from the standpoint of cyto
Publikováno v:
The Japanese Journal of Thoracic and Cardiovascular Surgery. 46:1329-1333
We experienced a case of a 38-year-old woman with a persistent cough, 24 years after repair of absent pulmonary valve syndrome with anomalous origin of the left pulmonary artery. The right pulmonary artery was massively dilated, thus it caused the co
Autor:
Takaki Hori, Hitoshi Houchi, Tetsuya Kitagawa, Takashi Kitaichi, Yutaka Masuda, Itsuo Katoh, Masanori Yoshizumi, Y. Ogawa
Publikováno v:
The Japanese Journal of Thoracic and Cardiovascular Surgery. 46:651-656
It has been suggested that plasma sulfoconjugated dopamine (DA) may serve as a source or reservoir for free DA in plasma. Moreover, it has also been reported that the plasma levels of conjugated DA may be used as an index predicting heart failure in
Autor:
Tetsuya Kitagawa, Itsuo Katoh, Kenzo Itoh, Yasushi Fukuta, Hitoshi Houchi, Toshiaki Tamaki, Takaki Hori, Masanori Yoshizumi
Publikováno v:
Basic Research in Cardiology. 93:23-29
It has previously been reported that ischemia and reperfusion of the heart cause accumulation of lysophosphatidylcholine (LPC) within the myocardium. While it is known that LPC causes the transient increase of intracellular free Ca2+ concentration ([
Autor:
Tetsuya Kitagawa, Hitoshi Houchi, Itsuo Katoh, Takaki Hori, Toshiaki Tamaki, Yasushu Fukuta, Masanori Yoshizumi
Publikováno v:
Biochemical Pharmacology. 55:481-487
In the present study, we examined the effect of angiotensin II on Ca2+ efflux from freshly isolated adult rat cardiomyocytes. Angiotensin II stimulated the efflux of 45Ca2+ from the cells in a concentration-dependent manner, at least in pharmacologic
Autor:
Masanori Yoshizumi, Tetsuya Kitagawa, Kazuya Horike, Itsuo Katoh, Yutaka Masuda, Y. Ogawa, Y. Suzuki, Toshiaki Tamaki
Publikováno v:
Scandinavian Cardiovascular Journal. 32:167-172
The effect of amiloride, a potent inhibitor of Na+/H+ exchange, on ischaemic reperfused rat hearts was studied in order to investigate whether Na+/H+ exchange or Na+/Ca2+ exchange is involved in ischaemia-reperfusion injury, When hearts were pre-isch
Autor:
Kazuo Minakuchi, Tetsuya Kitagawa, Koichiro Tsuchiya, Itsuo Katoh, Hitoshi Houchi, Masanori Yoshizumi, Kazuya Horike, Toshiaki Tamaki
Publikováno v:
FEBS Letters. 419:255-258
In the present study, we examined the effect of atrial natriuretic peptide (ANP) on Ca2+ efflux from freshly isolated adult rat cardiomyocytes. Rat ANP(1–28) stimulated the efflux of 45Ca2+ from the cells in a concentration-dependent manner (10−8
Autor:
Hitoshi Houchi, Kazuya Horike, Masanori Yoshizumi, Kenzo Itoh, Tetsuya Kitagawa, Toshiaki Tamaki, Itsuo Katoh
Publikováno v:
Naunyn-Schmiedeberg's Archives of Pharmacology. 356:756-762
Several physiological stimuli cause a rise in intracellular Ca2+ concentration ([Ca2+]i) in cardiomyocytes. This increased [Ca2+]i must be restored to physiological resting level to ensure response to further stimuli. In the present study, we examine
Autor:
Toshiaki Tamaki, Takaki Hori, Hitoshi Houchi, Tetsuya Kitagawa, Itsuo Katoh, Masanori Yoshizumi
Publikováno v:
Life Sciences. 61:1679-1689
In the present study, we examined the effect of extracellular adenosine 5′-triphosphate (ATP) on Ca2+ efflux from freshly isolated adult rat cardiomyocytes. ATP at 1 mM caused a release of 3.6 ± 0.08% of the total cellular content. The 45Ca2+ effl