Zobrazeno 1 - 6
of 6
pro vyhledávání: '"IGF 1R"'
Publikováno v:
Journal of the Serbian Chemical Society, Vol 68, Iss 11, Pp 811-818 (2003)
The biological activities of IGF-I and II are mediated mainly by the type 1 IGF receptor (IGF 1R) and controlled by their interaction with soluble proteins, the IGF binding proteins (IGFBPs). Although there is a growing body of evidence that some IGF
Externí odkaz:
https://doaj.org/article/27a069178286443cbeaf2887eed9bb1d
Publikováno v:
Journal of the Serbian Chemical Society, Vol 68, Iss 11, Pp 811-818 (2003)
The biological activities of IGF-I and –II are mediated mainly by the type 1 IGF receptor (IGF 1R) and controlled by their interaction with soluble proteins, the IGF binding proteins (IGFBPs). Although there is a growing body of evidence that some
Externí odkaz:
https://doaj.org/article/09abec8c03774aab94353db723412c5e
Autor:
María C. Sánchez, Paula Virginia Subirada Caldarone, Maria Constanza Paz, Valeria E. Lorenc, Gustavo A. Chiabrando, Darío G. Ferrer, J. D. Luna
Publikováno v:
Molecular Neurobiology. 55:1123-1135
In ischemic proliferative diseases such as retinopathies, persistent hypoxia leads to the release of numerous neovascular factors that participate in the formation of abnormal vessels and eventually cause blindness. The upregulation and activation of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8e5749723861a8183010aa497bfc3593
https://doi.org/10.1007/s12035-017-0386-9
https://doi.org/10.1007/s12035-017-0386-9
Publikováno v:
Journal of the Serbian Chemical Society, Vol 68, Iss 11, Pp 811-818 (2003)
Scopus-Elsevier
Scopus-Elsevier
The biological activities of IGF-I and ?II are mediated mainly by the type 1 IGF receptor (IGF 1R) and controlled by their interaction with soluble proteins, the IGF binding proteins (IGFBPs). Although there is a growing body of evidence that some IG
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3cafb01133cc32ab10b92561057558cc
https://doi.org/10.2298/jsc0311811m
https://doi.org/10.2298/jsc0311811m
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