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Autor:
A. V. Meza Sánchez, C. Hernandez-Diaz, C. Duarte-Salazar, R. E. Barbosa-Cobos, I. Romero-Vazquez, L. Ventura, G. Lugo-Zamudio
Publikováno v:
Annals of the Rheumatic Diseases. 81:1257.1-1257
BackgroundRheumatoid arthritis (RA) is an autoimmune, progressive disease characterized by an inflammatory process mediated by CD4+T lymphocytes leading to damage of the synovial membrane1. Its prevalence is estimated between 0.2 and 1.2% worldwide,