Zobrazeno 1 - 10
of 138
pro vyhledávání: '"I. Bednar"'
Autor:
I. Bednarczyk
Publikováno v:
Archives of Metallurgy and Materials, Vol vol. 67, Iss No 3, Pp 1179-1184 (2022)
The article presents the results of tests of plastic shaping of magnesium alloy Mg-Li. Magnesium alloy for an extrusion process was obtained with the method of vacuum smelting and casting into graphite moulds. The materials for tests were slabs cast
Externí odkaz:
https://doaj.org/article/eb9f7b5138d94363996e31dc3653eb6e
Publikováno v:
Journal of Neural Transmission. 112:255-268
Transgenic mice over-expressing human acetylcholinesterase (hAChE-Tg) display memory impairments, cholinergic deficits and reduced dendritic branching. In this study, we found a reduced number of N-Methyl-D-Aspartate (NMDA) binding sites and reduced
Publikováno v:
Neuropharmacology. 47:558-571
Overexpression of acetylcholinesterase (AChE) in mice causes cholinergic deficits with memory impairment. In this study, AChE overexpressing (hAChE-Tg) and control (FVB/N) mice were treated with the AChE inhibitor (AChEI) galantamine (4 mg/kg/day) fo
Publikováno v:
International Journal of Developmental Neuroscience. 22:545-549
In this study, we investigated regulatory mechanisms and plasticity of the nicotinic acetylcholine receptors (nAChRs) in the brain and adrenal glands of two transgenic mice models over-expressing human beta-amyloid precursor protein (APP(SWE)Tg) and
Autor:
Jennifer A. Court, Elaine K. Perry, Agneta Nordberg, Amelia Marutle, Alan J. Thomas, I. Bednar, M. Lee, Ewa Hellström-Lindahl, Marie Svedberg, Jessica Keverne
Publikováno v:
European Journal of Neuroscience. 19:2703-2710
Ten days treatment with nicotine reduced insoluble amyloid A beta 1-40 and Alpha beta 1-42 peptides by 80% in the cortex of 9-month-old APPsw mice, which is more than that observed in 14.5-month-old mice following nicotine treatment for 5.5 months. A
Publikováno v:
Neuroscience Letters. 340:148-152
Subtypes of nicotinic (alpha4 and alpha7) as well as muscarinic (M1 and M2) receptor binding sites were quantified in the brain of transgenic mice overexpressing human acetylcholinesterase (AChE) at different ages using selective radioligands. A sign
Autor:
M. Lee, Jennifer A. Court, Elaine K. Perry, Mary Johnson, Ros Hall, Agneta Nordberg, I. Bednar, Malahat Mousavi, Ewa Hellström-Lindahl
Publikováno v:
Journal of Neurochemistry. 81:655-658
Alzheimer's disease neuropathology is characterised by beta-amyloid plaques and neurofibrillary tangles. Inhibition of beta-amyloid accumulation may be essential for effective therapy in Alzheimer's disease. In this study we have treated transgenic m
Publikováno v:
International Journal of Developmental Neuroscience. 16:661-668
The basal forebrain cholinergic neurons are implicated in the pathogenesis of neurodegenerative diseases including Alzheimer's disease (AD). The nicotinic acetylcholine receptors (nAChRs) have been found to be significantly afflicted in AD. To study
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Publikováno v:
Europe PubMed Central
A dopamine D1 (SKF-38393, 1 mg)- or D2 (LY-171555, 0.1 mg)-receptor agonist inhibited intake of an intraorally infused solution of sucrose by male rats, a test of consummatory ingestive behavior. Treatment with a D1 (SCH-39166, 0.1 mg) or D2 (raclopr