Zobrazeno 1 - 7
of 7
pro vyhledávání: '"I B, Schweitzer"'
Autor:
D. D. Myers, Beverly Paigen, Roderick T. Bronson, Wesley G. Beamer, C. K. Reeb-Whitaker, I. B. Schweitzer, Gary A. Churchill
Publikováno v:
Laboratory Animals. 35:58-73
Our purpose in this investigation was to determine if we could reduce cage changing frequency without adversely affecting the health of mice. We housed mice at three different cage changing frequencies: 7, 14, and 21 days, each at three different cag
Autor:
L D Shultz, P A Schweitzer, S W Christianson, B Gott, I B Schweitzer, B Tennent, S McKenna, L Mobraaten, T V Rajan, D L Greiner
Publikováno v:
The Journal of Immunology. 154:180-191
The scid mutation was backcrossed ten generations onto the NOD/Lt strain background, resulting in an immunodeficient stock (NOD/LtSz-scid/scid) with multiple defects in adaptive as well as nonadaptive immunologic function. NOD/LtSz-scid/scid mice lac
Autor:
Michael A. Lynes, J. C. Lee, L. D. Shultz, I. B. Schweitzer, K. C. Crowthers, J. Youn, R. McCabe, C. A. Richardson
Publikováno v:
Metallothionein IV ISBN: 9783034897990
Cells under stress produce metallothionein as one way of coping with the biological consequences of that stress. A broad array of conditions are known to elicit increases in metallothionein (MT) synthesis, including exposure to various toxic chemical
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b21e006af058b94d70f392d89cdf3bfa
https://doi.org/10.1007/978-3-0348-8847-9_63
https://doi.org/10.1007/978-3-0348-8847-9_63
Autor:
S C, Pelsue, P A, Schweitzer, I B, Schweitzer, S W, Christianson, B, Gott, J P, Sundberg, W G, Beamer, L D, Shultz
Publikováno v:
European journal of immunology. 28(4)
The autosomal recessive mutation "flaky skin" (fsn) causes pleiotropic abnormalities in the immune and hematopoietic systems accompanied by pathologic changes in the skin. Homozygotes (fsn/fsn) showed increased size and histological alterations in th
Autor:
S W, Christianson, D L, Greiner, R A, Hesselton, J H, Leif, E J, Wagar, I B, Schweitzer, T V, Rajan, B, Gott, D C, Roopenian, L D, Shultz
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 158(8)
Genetic crosses produced NOD/LtSz mice doubly homozygous for the severe combined immunodeficiency (scid) mutation and the beta2m (B2m) null allele. Both NOD/LtSz-scid/scid and NOD/LtSz-scid/scid B2m(null) mice lacked mature lymphocytes and serum Ig.
Autor:
L D, Shultz, P A, Schweitzer, S W, Christianson, B, Gott, I B, Schweitzer, B, Tennent, S, McKenna, L, Mobraaten, T V, Rajan, D L, Greiner
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 154(1)
The scid mutation was backcrossed ten generations onto the NOD/Lt strain background, resulting in an immunodeficient stock (NOD/LtSz-scid/scid) with multiple defects in adaptive as well as nonadaptive immunologic function. NOD/LtSz-scid/scid mice lac
Autor:
I. B. Schweitzer, M.A. Lynes, L. D. Shultz, J. C. Lee, C.A. Ricardson, R. McCabe, K. C. Crowthers, J. Youn
Publikováno v:
Toxicology Letters. 95:179