Zobrazeno 1 - 10
of 21
pro vyhledávání: '"Hyeseon Cha"'
Autor:
Chang Sik Park, Hyeseon Cha, Eun Jeong Kwon, Dongtak Jeong, Roger J Hajjar, Evangelia G Kranias, Chunghee Cho, Woo Jin Park, Do Han Kim
Publikováno v:
PLoS ONE, Vol 7, Iss 8, p e43282 (2012)
Histidine-rich calcium binding protein (HRC) is located in the lumen of sarcoplasmic reticulum (SR) that binds to both triadin (TRN) and SERCA affecting Ca(2+) cycling in the SR. Chronic overexpression of HRC that may disrupt intracellular Ca(2+) hom
Externí odkaz:
https://doaj.org/article/0b519156a7014028a1c1127c55f20273
Autor:
Nuri Yun, Il Seon Park, Chiho Kim, Hirohiko Shibayama, Hyeseon Cha, Woo Jin Park, Young J. Oh
Publikováno v:
Biochemical and Biophysical Research Communications. 432:533-538
Mammalian protein kinase C-interacting cousin of thioredoxin (PICOT) is a multi-domain mono-thiol glutaredoxin that is involved in several signal transduction pathways and is necessary for cell growth and metastasis. Here, we demonstrate that PICOT i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::71a132fce83cc93c8b29532a534ec7b7
https://doi.org/10.1016/j.bbrc.2013.02.017
https://doi.org/10.1016/j.bbrc.2013.02.017
Autor:
Jihwa Kim, Soonyong Park, Chang Sik Park, Dong Kwon Yang, Do Han Kim, Changwon Kho, Yung Joon Yoo, Jae Gyun Oh, Ji Myoung Kim, Ekaterina Lifirsu, Roger J. Hajjar, Hyeseon Cha, Jaetaek Kim, Woo Jin Park, Dongtak Jeong
Publikováno v:
Journal of Molecular and Cellular Cardiology. 53:53-63
Protein kinase C (PKC)-interacting cousin of thioredoxin (PICOT) has distinct anti-hypertrophic and inotropic functions. We have previously shown that PICOT exerts its anti-hypertrophic effect by inhibiting calcineurin-NFAT signaling through its C-te
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0531f478fefe68f2f75ac4dcbe219f1b
https://doi.org/10.1016/j.yjmcc.2012.03.005
https://doi.org/10.1016/j.yjmcc.2012.03.005
Autor:
Pyoung Oh Yoon, Roger J. Hajjar, Dongtak Jeong, Bo Youn Choi, Moon Hee Jeong, Dong Kwon Yang, Jooyeon Kim, Min-Ah Lee, Woo Jin Park, Seung Pil Jang, Hyeseon Cha
Publikováno v:
Journal of Molecular and Cellular Cardiology. 49:294-303
CCN family members are matricellular proteins with diverse roles in cell function. The differential expression of CCN2 and CCN5 during cardiac remodeling suggests that these two members of the CCN family play opposing roles during the development of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dc83e41879766a84130297b9b7c72551
https://doi.org/10.1016/j.yjmcc.2010.04.010
https://doi.org/10.1016/j.yjmcc.2010.04.010
Autor:
Eun-Seon Ju, Ji Myoung Kim, Eun-Seok Jeon, Dongtak Jeong, Woo Jin Park, Jaeho Park, Roger J. Hajjar, Hyeseon Cha, Soo-Hyeon Yun, Jae Gyun Oh
Publikováno v:
Circulation Research. 102:711-719
PICOT (protein kinase C–interacting cousin of thioredoxin) was previously shown to inhibit pressure overload-induced cardiac hypertrophy, concomitant with an increase in ventricular function and cardiomyocyte contractility. The combined analyses of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bcce160f9bb19e4ed93fde2380d6e96b
https://doi.org/10.1161/circresaha.107.165985
https://doi.org/10.1161/circresaha.107.165985
PICOT Inhibits Cardiac Hypertrophy and Enhances Ventricular Function and Cardiomyocyte Contractility
Autor:
Ji Myoung Kim, Susumu Sakata, Woo Jin Park, Eun Young Kim, Dong Kwon Yang, Sun-Hee Woo, Roger J. Hajjar, Dongtak Jeong, Hyeseon Cha, Lei Cui, Young-Hoon Lee, Le Thi Thu, Jae Gyun Oh, Do Han Kim, Ronglih Liao, Oliver Y. Bernecker, Misuk Kang, Pyoung Oh Yoon
Publikováno v:
Circulation Research. 99:307-314
Multiple signaling pathways involving protein kinase C (PKC) have been implicated in the development of cardiac hypertrophy. We observed that a putative PKC inhibitor, PICOT ( P KC- I nteracting C ousin O f T hioredoxin) was upregulated in response t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fe2a3f114a7f8ede7c4f215b0a317b6e
https://doi.org/10.1161/01.res.0000234780.06115.2c
https://doi.org/10.1161/01.res.0000234780.06115.2c
Autor:
Hong-Sheng Wang, Shan Chen, Inju Park, Peidong Han, Donald M. Bers, Evangelia G. Kranias, Chang Sik Park, Chunghee Cho, Hoyong Lee, Kenneth S. Ginsburg, Sora Jin, Do Han Kim, Clara Franzini-Armstrong, Woo Jin Park, Vivek P. Singh, Hyeseon Cha, Sunghee Hong, Jae Gyun Oh
Publikováno v:
Basic Research in Cardiology. 108
The histidine-rich Ca(2+)-binding protein (HRC) is located in the lumen of the sarcoplasmic reticulum (SR) and exhibits high-capacity Ca(2+)-binding properties. Overexpression of HRC in the heart resulted in impaired SR Ca(2+) uptake and depressed re
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca52bd3d61d48d6c5b07504524a440cb
https://doi.org/10.1007/s00395-013-0344-2
https://doi.org/10.1007/s00395-013-0344-2
Publikováno v:
Biochemical and biophysical research communications. 421(3)
Evidence has shown that endoplasmic reticulum stress (ERS) is associated with the pathogenesis of cardiac hypertrophy. The aim of this study was to investigate whether direct alleviation of ER stress by 4-phenylbutyric acid (PBA), a known chemical ch
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::510adb803897dc9ae472b2dd48576699
https://pubmed.ncbi.nlm.nih.gov/22525677
https://pubmed.ncbi.nlm.nih.gov/22525677
Autor:
Dong Kwon Yang, Jae Gyun Oh, Joo Yeon Kim, Hyeseon Cha, Woo Jin Park, Hyeon Joo Jeong, Seung Pil Jang
Publikováno v:
Experimentalmolecular medicine. 42(1)
Parathyroid hormone (PTH) treatment was previously shown to improve cardiac function after myocardial infarction by enhancing neovascularization and cell survival. In this study, pressure overload-induced left ventricular hypertrophy (LVH) was induce
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f8d7398b821c6966871c1f4d0f13d89b
https://pubmed.ncbi.nlm.nih.gov/19887893
https://pubmed.ncbi.nlm.nih.gov/19887893
Autor:
Dongtak Jeong, Chang Sik Park, Hyeon Joo Jeong, Dong Kwon Yang, Byung Keon Park, Moon Hee Jeong, Hyeseon Cha, Roger J. Hajjar, Do Han Kim, Jae Gyun Oh, Ji Myoung Kim, Young-Hoon Lee, Woo Jin Park, Jaeho Park, Oliver Y. Bernecker
Publikováno v:
Journal of molecular and cellular cardiology. 45(6)
PICOT (PKC-interacting cousin of thioredoxin) was previously shown to inhibit the development of cardiac hypertrophy, concomitant with an increase in cardiomyocyte contractility. To explore the physiological function of PICOT in the hearts, we genera
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ebf5d8a94d6e6a828c2fb2ef63e903b1
https://pubmed.ncbi.nlm.nih.gov/18929570
https://pubmed.ncbi.nlm.nih.gov/18929570