Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Hussain Mubarak Al-Aamri"'
Publikováno v:
BMC Cancer, Vol 21, Iss 1, Pp 1-10 (2021)
Abstract Background Daunorubicin is used clinically in the treatment of myeloma, acute lymphatic and myelocytic leukaemia. The toxic lesions caused by daunorubicin induce various modes of cell death, including apoptosis. Apoptosis is highly regulated
Externí odkaz:
https://doaj.org/article/f923535fdba041a0aef22c9238e7b9df
Publikováno v:
BMC Research Notes, Vol 12, Iss 1, Pp 1-5 (2019)
Abstract Objective DNA double strand breaks (DNA-DSBs) are among the most lethal DNA lesions leading to genomic instability and repaired by either homologous recombination (HR) or the non-homologous end joining (NHEJ) mechanisms. The purpose of this
Externí odkaz:
https://doaj.org/article/27a64ef37e5d4e9fab638d340d14171e
Autor:
Hussain Mubarak Al-Aamri, Heng Ku, Helen R. Irving, Joseph Tucci, Terri Meehan-Andrews, Christopher Bradley
Publikováno v:
BMC Cancer, Vol 19, Iss 1, Pp 1-12 (2019)
Abstract Background Daunorubicin is commonly used in the treatment of acute lymphoblastic leukaemia (ALL). The aim of this study was to explore the kinetics of double strand break (DSB) formation of three ALL cell lines following exposure to daunorub
Externí odkaz:
https://doaj.org/article/dc6db11dc2ea4364aad787ab59778e63
Publikováno v:
BMC Cancer
BMC Cancer, Vol 21, Iss 1, Pp 1-10 (2021)
BMC Cancer, Vol 21, Iss 1, Pp 1-10 (2021)
BackgroundDaunorubicin is used clinically in the treatment of myeloma, acute lymphatic and myelocytic leukaemia. The toxic lesions caused by daunorubicin induce various modes of cell death, including apoptosis. Apoptosis is highly regulated programme
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7cffa7ad6c815de1add85680e340350d
https://doi.org/10.1186/s12885-021-08167-y
https://doi.org/10.1186/s12885-021-08167-y
Publikováno v:
BMC Research Notes
BMC Research Notes, Vol 12, Iss 1, Pp 1-5 (2019)
BMC Research Notes, Vol 12, Iss 1, Pp 1-5 (2019)
Objective DNA double strand breaks (DNA-DSBs) are among the most lethal DNA lesions leading to genomic instability and repaired by either homologous recombination (HR) or the non-homologous end joining (NHEJ) mechanisms. The purpose of this study was
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::535b8706a20f7b0643aebe479ca8d3a1
https://doi.org/10.1186/s13104-019-4663-8
https://doi.org/10.1186/s13104-019-4663-8
Autor:
Terri Meehan-Andrews, Christopher Bradley, Heng Ku, Helen Irving, Joseph Tucci, Hussain Mubarak Al-Aamri
Publikováno v:
BMC Cancer
BMC Cancer, Vol 19, Iss 1, Pp 1-12 (2019)
BMC Cancer, Vol 19, Iss 1, Pp 1-12 (2019)
Background Daunorubicin is commonly used in the treatment of acute lymphoblastic leukaemia (ALL). The aim of this study was to explore the kinetics of double strand break (DSB) formation of three ALL cell lines following exposure to daunorubicin and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d672ef715b846532d9993ca3f98f10c0
https://pubmed.ncbi.nlm.nih.gov/30813936
https://pubmed.ncbi.nlm.nih.gov/30813936