Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Huiqiong HE"'
Publikováno v:
Pifu-xingbing zhenliaoxue zazhi, Vol 28, Iss 5, Pp 352-356 (2021)
Objective To explore the antibacterial effect of artemether on P. acnes in vitro and its effect on the bacterial morphology in order to provide a rationale for its use in the treatment of acne. Methods The minimal inhibitory concentrations (MIC) of a
Externí odkaz:
https://doaj.org/article/ae436f6163a24e4f8fd1ee03850ed6ce
Autor:
Lei Sun, Qiong Wu, Yunjuan Nie, Ni Cheng, Rui Wang, Gang Wang, Dan Zhang, Huiqiong He, Richard D. Ye, Feng Qian
Publikováno v:
Frontiers in Immunology, Vol 9 (2018)
Increased inflammatory responses and enhanced reactive oxygen species contribute to hepatic ischemia/reperfusion (I/R) injury, however the modulatory mechanisms haven't been completely unveiled. Here, we report that genetic deficiency of MAPK-activat
Externí odkaz:
https://doaj.org/article/155c9ec35c27444991152fa84662ecbc
Publikováno v:
PLoS ONE, Vol 6, Iss 1, p e14510 (2011)
BACKGROUND: BmK IT2 is regarded as a receptor site-4 modulator of sodium channels with depressant insect toxicity. It also displays anti-nociceptive and anti-convulsant activities in rat models. In this study, the potency and efficacy of BmK IT2 were
Externí odkaz:
https://doaj.org/article/22d7cd02fb5d4ff2a497a8ca5dfd4eca
Autor:
Yaxian Wu, Hongchao Jiang, Feng Qian, Yudong Hu, Depeng Zhang, Tian-hong Yu, Xiaozong Li, Huiqiong He, Yunhe Ding, Yunduan Song, Lei Sun, Kaikai Yu
Publikováno v:
Acta Pharmacologica Sinica. 40:64-74
Isoalantolactone (IAL) is a sesquiterpene lactone extracted from roots of Inula helenium L and has shown anti-inflammatory effects. In this study we investigated the therapeutic effects of IAL on acute lung injury (ALI) and elucidated the mechanisms
Autor:
Richard D. Ye, Gang Wang, Yunjuan Nie, Qiong Wu, Dan Zhang, Ni Cheng, Huiqiong He, Lei Sun, Rui Wang, Feng Qian
Publikováno v:
Frontiers in Immunology, Vol 9 (2018)
Frontiers in Immunology
Frontiers in Immunology
Increased inflammatory responses and enhanced reactive oxygen species contribute to hepatic ischemia/reperfusion (I/R) injury, however the modulatory mechanisms haven't been completely unveiled. Here, we report that genetic deficiency of MAPK-activat
Autor:
Hongchao Jiang, Sirui Liu, Depeng Zhang, Huiqiong He, Jun Wang, Yunhe Ding, Yaxian Wu, Lei Sun, Feng Qian, Richard D. Ye, Dan Zhang
Publikováno v:
American journal of physiology. Lung cellular and molecular physiology. 315(3)
MAPK-activated protein kinase 2 (MK2) plays a critical role in the development of inflammation. However, the modulatory mechanisms in macrophage activation and acute lung injury (ALI) have not been completely defined. Here, we reported that MK2-defic
Autor:
Huiqiong He, Depeng Zhang, Jingsong Xu, Xiaozong Li, Hongchao Jiang, Yunhe Ding, Kaikai Yu, Yudong Hu, Lei Sun, Feng Qian, Yaxian Wu
Publikováno v:
Biochemical pharmacology. 154
Sepsis caused by Gram-negative bacteria is one of major causes for the progression of acute lung injury (ALI) with limited treatment and effective medicines. Tabersonine is an indole alkaloid mainly isolated from Catharanthus roseus, and a potential
Toll-like receptor 4 (TLR4)-mediated signaling plays a critical role in sepsis-induced acute lung injury (ALI). LYRM03 (3-amino-2-hydroxy-4-phenyl-valyl-isoleucine) is a novel derivative of ubenimex, a widely used antineoplastic medicine. We previous
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e3669692af01ac929658966b714d475d
https://europepmc.org/articles/PMC5342666/
https://europepmc.org/articles/PMC5342666/
Autor:
John W. Christman, Lei Sun, Qing Liang, Yanhua Chang, Yaxian Wu, Jianwei Zhu, Jun Wang, Huiqiong He, Yaoyao Yang, Feng Qian, Yudong Hu, Richard D. Ye, Yunjuan Nie
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 198(11)
Idiopathic pulmonary fibrosis (IPF) is a highly lethal pathological process that is characterized by inflammation, fibroblast accumulation, and excessive collagen deposition. Although AKT2-mediated signaling pathways modulate inflammatory responses,
Publikováno v:
Journal of Alzheimer's disease : JAD. 43(1)
Amyloid-β peptides such as Aβ1-42 (Aβ42) play a pivotal role in the progression of Alzheimer's disease (AD). Aβ42 is neurotoxic and can activate microglial cells. These cells in turn migrate toward senile (neuritic) plaques and help to clear Aβ