Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Huimiao Xu"'
Calcium alleviates fluoride-induced kidney damage via FAS/FASL, TNFR/TNF, DR5/TRAIL pathways in rats
Autor:
Haojie Li, Junjiang Fan, Yangfei Zhao, Jiarong Yang, Huimiao Xu, Ram Kumar Manthari, Xiaofang Cheng, Jundong Wang, Jinming Wang
Publikováno v:
Ecotoxicology and Environmental Safety, Vol 226, Iss , Pp 112851- (2021)
Long-term excessive intake of fluoride (F) can cause osseous and non-osseous damage. The kidney is the main fluoride excretion organ of the body. This study aimed to explore whether dietary calcium (Ca) supplementation can alleviate kidney damage cau
Externí odkaz:
https://doaj.org/article/f87ffb9468db40f09eb29e91fcb6d352
Autor:
Jundong Wang, Xiaofang Cheng, Huimiao Xu, Zipeng Yan, Jiarong Yang, Haili Ma, Ram Kumar Manthari, Mohammad Mehdi Ommati, Jinming Wang, Yangfei Zhao
Publikováno v:
Food & Function. 11:1155-1164
Bone is the main target of fluorosis, and it has been perfectly elaborated that a moderate dosage of calcium (Ca) can alleviate bone fluorosis. However, whether Ca can alleviate fluorosis through the phosphatidylinositol 3 kinase (PI3K)/protein kinas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::158873cad7c551c3c03b1679695d3de4
https://doi.org/10.1039/c9fo02491c
https://doi.org/10.1039/c9fo02491c
Calcium alleviates fluoride-induced kidney damage via FAS/FASL, TNFR/TNF, DR5/TRAIL pathways in rats
Autor:
Xiaofang Cheng, Ram Kumar Manthari, Jinming Wang, Huimiao Xu, Yangfei Zhao, Jiarong Yang, Junjiang Fan, Haojie Li, Jundong Wang
Publikováno v:
Ecotoxicology and Environmental Safety, Vol 226, Iss, Pp 112851-(2021)
Long-term excessive intake of fluoride (F) can cause osseous and non-osseous damage. The kidney is the main fluoride excretion organ of the body. This study aimed to explore whether dietary calcium (Ca) supplementation can alleviate kidney damage cau
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::63de18245c26efa1a58e24699e566936
http://www.sciencedirect.com/science/article/pii/S0147651321009635
http://www.sciencedirect.com/science/article/pii/S0147651321009635
Autor:
Jundong Wang, Huimiao Xu, Ram Kumar Manthari, Xiaofang Cheng, Jinming Wang, Yanyan Li, Yangfei Zhao
Publikováno v:
Food and Chemical Toxicology. 116:189-195
In reviewing the literature, the cellular mechanism of fluoride F-induced osteoblast OB cells apoptosis is diverse and perplexing, but detailed regulatory pathway, targets and role of extracellular Ca2+ remains still unclear. Hence, in the present st
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::db40f7e88d7bf148799f0346b07b6f89
https://doi.org/10.1016/j.fct.2018.04.013
https://doi.org/10.1016/j.fct.2018.04.013
Autor:
Huimiao Xu, Ram Kumar Manthari, Zipeng Yan, Jundong Wang, Jinming Wang, Yangfei Zhao, Mohammad Mehdi Ommati, Yaya Zhu, Xiaofang Cheng, Jiarong Yang, Rui Xiao
Publikováno v:
Journal of agricultural and food chemistry. 67(39)
Excessive fluoride mainly causes skeletal lesions. Recently, it has been reported that an appropriate level of calcium can alleviate fluorosis. However, the appropriate concentration and mechanism of calcium addition is unclear. Hence, we evaluated t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::51d801c815c1b1a37e2dc6c5d8139747
https://pubmed.ncbi.nlm.nih.gov/31464433
https://pubmed.ncbi.nlm.nih.gov/31464433
Autor:
Jinming, Wang, Yangfei, Zhao, Xiaofang, Cheng, Yanyan, Li, Huimiao, Xu, Ram Kumar, Manthari, Jundong, Wang
Publikováno v:
Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association. 116
In reviewing the literature, the cellular mechanism of fluoride F-induced osteoblast OB cells apoptosis is diverse and perplexing, but detailed regulatory pathway, targets and role of extracellular Ca
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::7e956e89d5ebc3f293389a4d34e1ec85
https://pubmed.ncbi.nlm.nih.gov/29642045
https://pubmed.ncbi.nlm.nih.gov/29642045