Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Hualu, Cui"'
Autor:
Zhuo Wang, Xiaowan Yin, Meichen Ma, Hongchi Ge, Bin Lang, Hong Sun, Sijia He, Yajing Fu, Yu Sun, Xiaowen Yu, Zining Zhang, Hualu Cui, Xiaoxu Han, Junjie Xu, Haibo Ding, Zhenxing Chu, Hong Shang, Yuntao Wu, Yongjun Jiang
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
A major barrier to HIV eradication is the persistence of viral reservoirs. Resting CD4+ T cells are thought to be one of the major viral reservoirs, However, the underlying mechanism regulating HIV infection and the establishment of viral reservoir i
Externí odkaz:
https://doaj.org/article/68c60685b58c4bd9aa75b83007bf450d
Autor:
Hualu Cui, Yongjun Jiang, Yajing Fu, Zining Zhang, Junjie Xu, Wenqing Geng, Guoxin Liang, Haibo Ding, Hong Sun, Hong Shang
Publikováno v:
Cellular Immunology. 327:47-53
The profound deficiency of Th17 cells contributes to HIV disease progression. The mechanisms of their perturbation remain unclear. Recently, CCR6+CD95+CD4+ naive T cells (CCR6+CD95+CD4+ TNA), identified as pre-committed Th17 precursors, were recogniz
Autor:
Gefei Guan, Hualu Cui, Wenqing Geng, Haibo Ding, Hong Shang, Bin Zhao, Ying Qiao, Xiaoxu Han, Guoxin Liang, Yuntao Wu, Xiaowei Zhang, Ruixuan Wang
Publikováno v:
FEBS Letters. 592:284-294
Activation-induced cytidine deaminase (AID), a member of the APOBEC family that induces antibody diversification, has been shown to inhibit the replication of hepatitis B virus, Kaposi's sarcoma-associated herpesvirus, and retro-transposons. However,
Autor:
Haibo Ding, Hong Shang, Huihui Chen, Hualu Cui, Yue Zhang, Zining Zhang, Xi Chen, Tao Dong, Yongjun Jiang, Junjie Xu, Xiaoxu Han, Yajing Fu
Publikováno v:
The Journal of infectious diseases. 220(12)
Background Natural killer (NK) cells are an important type of effector cell in the innate immune response, and also have a role in regulation of the adaptive immune response. Several studies have indicated that NK cells may influence CD4+ T cells dur
Autor:
Hong, Sun, Wenqing, Geng, Hualu, Cui, Guoxin, Liang, Yajing, Fu, Zining, Zhang, Yongjun, Jiang, Haibo, Ding, Junjie, Xu, Hong, Shang
Publikováno v:
Cellular immunology. 327
The profound deficiency of Th17 cells contributes to HIV disease progression. The mechanisms of their perturbation remain unclear. Recently, CCR6
Autor:
Ruixuan, Wang, Xiaowei, Zhang, Haibo, Ding, Ying, Qiao, Xiaoxu, Han, Wenqing, Geng, Gefei, Guan, Hualu, Cui, Bin, Zhao, Yuntao, Wu, Guoxin, Liang, Hong, Shang
Publikováno v:
FEBS letters. 592(2)
Activation-induced cytidine deaminase (AID), a member of the APOBEC family that induces antibody diversification, has been shown to inhibit the replication of hepatitis B virus, Kaposi's sarcoma-associated herpesvirus, and retro-transposons. However,
Autor:
Hualu Cui, Xuejie Fan, Zining Zhang, Wenqing Geng, Yongjun Jiang, Hong Sun, Hong Shang, Fang Gao, Sha Li
Publikováno v:
Journal of AIDS & Clinical Research.
Background: Deficient interferon (IFN)-α production of plasmacytoid DCs (pDCs) from HIV-1-infected individuals in response to virus or toll-like receptor (TLR) stimulation has been reported. pDCs express TLR7 and TLR9, but whether and how the TLR pa
Autor:
Yanan Wang, Yongjun Jiang, Wenqing Geng, Hong Shang, Hong Sun, Hualu Cui, Yingying Diao, Amy Sun, Xuejie Fan
Publikováno v:
BMC Infectious Diseases
Background During early HIV-1 infection (EHI), the interaction between the immune response and the virus determines disease progression. Although CD1c + myeloid dendritic cells (mDCs) can trigger the immune response, the relationship between CD1c + m
Autor:
Yingying Diao, Wenqing Geng, Xuejie Fan, Hualu Cui, Hong Sun, Yongjun Jiang, Yanan Wang, Sun, Amy, Hong Shang
Publikováno v:
BMC Infectious Diseases; Aug2015, Vol. 15 Issue 1, p1-8, 8p, 1 Illustration, 2 Charts, 2 Graphs
Autor:
Min Zhao, Wenqing Geng, Yongjun Jiang, Xiaoxu Han, Hualu Cui, Di Dai, Mingjia Bao, Ying Pan, Yating Wang, Xiaoli Zhang, Min Zhang, Guan Qi, Hong Shang
Publikováno v:
JAIDS: Journal of Acquired Immune Deficiency Syndromes; 2/1/2010 Supplement 1, Vol. 53, pS4-S9, 6p, 3 Charts, 3 Graphs