Zobrazeno 1 - 10 of 23 pro vyhledávání: '"Hua-Sheng Ding"'
1
Akademický článek
Abrogation of CC Chemokine Receptor 9 Ameliorates Ventricular Electrical Remodeling in Mice After Myocardial Infarction
Autor: Yan Huang, Hua-Sheng Ding, Tao Song, Yu-Ting Chen, Teng Wang, Yan-Hong Tang, Hector Barajas-Martinez, Cong-Xin Huang, Dan Hu
Publikováno v: Frontiers in Cardiovascular Medicine, Vol 8 (2021)
Introduction: Myocardial infarction (MI) triggers structural and electrical remodeling. CC chemokine receptor 9 (CCR9) mediates chemotaxis of inflammatory cells in MI. In our previous study, CCR9 knockout has been found to improve structural remodeli
Externí odkaz: https://doaj.org/article/9b4a6ad32a614b9a9bb5f85c151713bd
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2
Akademický článek
S9, a novel anticancer agent, exerts its anti-proliferative activity by interfering with both PI3K-Akt-mTOR signaling and microtubule cytoskeleton.
Autor: Chao Zhang, Na Yang, Chun-Hao Yang, Hua-Sheng Ding, Cheng Luo, Yu Zhang, Mao-Jiang Wu, Xiong-Wen Zhang, Xu Shen, Hua-Liang Jiang, Ling-Hua Meng, Jian Ding
Publikováno v: PLoS ONE, Vol 4, Iss 3, p e4881 (2009)
BACKGROUND: Deregulation of the phosphatidylinositol 3-kinases (PI3K)/Akt/mammalian target of rapamycin (mTOR) pathway plays a central role in tumor formation and progression, providing validated targets for cancer therapy. S9, a hybrid of alpha-meth
Externí odkaz: https://doaj.org/article/f10535cd57594174ba42b5099b64f719
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3
AAV9-Mediated Cardiac CNTF Overexpression Exacerbated Adverse Cardiac Remodeling in Streptozotocin-Induced Type 1 Diabetic Models
Autor: Hua-sheng Ding, Jianye Peng, Tao Liu, Peng Zhong
Publikováno v: Cardiovascular Toxicology. 22:88-96
Ciliary neurotrophic factor (CNTF), which is a neural peptide, has been reported to confer cardioprotective effects. However, whether CNTF-based gene delivery could prevent cardiac remodeling in diabetes mellitus remains unknown. In this study, we us
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::e27d25236db7dd1a7f8a7dcbf549cf09
https://doi.org/10.1007/s12012-021-09706-6
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5
Sodium tanshinone IIA sulfonate protects Myocardium from Ischemia/ Reperfusion Injury in Mice via AKT- and SIRT1/TLR4-related Mechanisms
Autor: Hua-Sheng Ding, Fu-Ji Qu, Qiu-Jian Li, Zhi-Hui Luo, Yi Peng, Feng-Yuan Wang, Peng Zhong, Yan Huang, Xiao-Xiong Liu
Background Cardiac ischemia-reperfusion (I/R) injury is highly related to ischemic heart disease which is the main cause of death worldwide. However, no standard therapy for I/R is available. Sodium tanshinone IIA sulfonate (STS) has been demonstrate
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::b83ca406d4dd6261a859abd4371c93a4
https://doi.org/10.21203/rs.3.rs-1875759/v1
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6
AAV9-Mediated Cardiac CNTF Overexpression Exacerbated Adverse Cardiac Remodeling in Streptozotocin-Induced Type 1 Diabetic Models
Autor: Peng, Zhong, Jianye, Peng, Tao, Liu, Hua-Sheng, Ding
Publikováno v: Cardiovascular toxicology. 22(1)
Ciliary neurotrophic factor (CNTF), which is a neural peptide, has been reported to confer cardioprotective effects. However, whether CNTF-based gene delivery could prevent cardiac remodeling in diabetes mellitus remains unknown. In this study, we us
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::69e87ca20163fbe48467772740423f21
https://pubmed.ncbi.nlm.nih.gov/34878621
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7
Fluvastatin attenuated ischemia/reperfusion-induced autophagy and apoptosis in cardiomyocytes through down-regulation HMGB1/TLR4 signaling pathway
Autor: Jian Yang, Jun Yang, Congxin Huang, Yan Huang, Yanhong Tang, Zhen Chen, Zhi-Xing Fan, Xin Guo, Hua-Sheng Ding
Publikováno v: Molecular biology reports. 48(5)
Fluvastatin, a traditional fat-decreasing drug, is widely used for curing cardiovascular disease. Previous reports demonstrated that fluvastatin pretreatment protected against myocardial ischemia/reperfusion (I/R) by inhibiting TLR4 signaling pathway
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f66c59c8584f5efeba1919bb066cacb8
https://pubmed.ncbi.nlm.nih.gov/34032975
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8
MiR-33 promotes myocardial fibrosis by inhibiting MMP16 and stimulating p38 MAPK signaling
Autor: Yan Huang, Congxin Huang, Di Fan, Xin Guo, Zhen Chen, Hua-Sheng Ding, Jing-Jing Shen
Publikováno v: Oncotarget
Myocardial fibrosis occurs in the late stages of many cardiovascular diseases, and appears to be stimulated by various microRNAs (miRNAs). We previously found that miR-33 may stimulate cardiac remodeling. Here, we examined the involvement of miR-33 i
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d8092037b1e3c59eb6ea8e3d08bef892
https://doi.org/10.18632/oncotarget.25173
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10
Regulator of G-protein signalling 5 deficiency impairs ventricular remodelling after myocardial infarction by promoting NF-κB and MAPK signalling in mice
Autor: Di Fan, Hua-Sheng Ding, Dandan Wang, Congxin Huang, Yanhong Tang, Yan Huang, Zhen Chen
Publikováno v: Biochemical and biophysical research communications. 499(2)
Regulator of G-protein signalling 5 (RGS5) is, highly expressed in different cell types of the adult human heart, and it is a negative regulator of G protein-mediated signalling that inactivates Gα(q) and Gα(i) and thereby inhibits many signalling
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cd66f8658e7f18048fa1f86ea3d7dc3c
https://pubmed.ncbi.nlm.nih.gov/29534968
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