Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Howard T. J. Mount"'
Autor:
Shreya H. Dhume, Kayode Balogun, Ambalika Sarkar, Sebastian Acosta, Howard T. J. Mount, Lindsay S. Cahill, John G. Sled, Lena Serghides
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 17 (2024)
BackgroundCombination antiretroviral therapy (ART) use in pregnancy has been pivotal in improving maternal health and reducing perinatal HIV transmission. However, children born HIV-exposed uninfected fall behind their unexposed peers in several area
Externí odkaz:
https://doaj.org/article/bce873f96d344bb199afb41b22cba351
Autor:
Lena Serghides, Chloe R McDonald, Ziyue Lu, Miriam Friedel, Cheryl Cui, Keith T Ho, Howard T J Mount, John G Sled, Kevin C Kain
Publikováno v:
PLoS Pathogens, Vol 10, Iss 3, p e1003980 (2014)
Cerebral malaria (CM) is associated with a high mortality rate, and long-term neurocognitive impairment in approximately one third of survivors. Adjunctive therapies that modify the pathophysiological processes involved in CM may improve outcome over
Externí odkaz:
https://doaj.org/article/2c2aeaa0ad1a4561bcd0a02d50ccfbe5
Autor:
Bettina, Drisaldi, Janaky, Coomaraswamy, Peter, Mastrangelo, Bob, Strome, Jing, Yang, Joel C, Watts, M Azhar, Chishti, Melissa, Marvi, Otto, Windl, Rosemary, Ahrens, François, Major, Man-Sun, Sy, Hans, Kretzschmar, Paul E, Fraser, Howard T J, Mount, David, Westaway
Publikováno v:
The Journal of biological chemistry. 279(53)
The PrP-like Doppel (Dpl) protein causes apoptotic death of cerebellar neurons in transgenic mice, a process prevented by expression of the wild type (wt) cellular prion protein, PrP(C). Internally deleted forms of PrP(C) resembling Dpl such as PrPDe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c9612748cd09234ebe1ae6dc821905df
https://pubmed.ncbi.nlm.nih.gov/15459186
https://pubmed.ncbi.nlm.nih.gov/15459186
Autor:
Howard T J, Mount, Jean-Claude, Martel, Paul, Fluit, Yingji, Wu, Eleanor, Gallo-Hendrikx, Cristina, Cosi, Marc R, Marien
Publikováno v:
Journal of neurochemistry. 88(6)
Ataxia-telangiectasia (A-T) is a genetic disease, associated with progressive motor impairment and a lack of functional ATM protein. It has been reported that immunoreactive tyrosine hydroxylase and dopamine transporter are reduced in an Atm-/- mouse
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::9cc629be73fac93969d2e7bc0bc5a511
https://pubmed.ncbi.nlm.nih.gov/15009646
https://pubmed.ncbi.nlm.nih.gov/15009646