Zobrazeno 1 - 10
of 34
pro vyhledávání: '"Honoka, Kiso"'
Autor:
Sayaka Mishima, Katsu Takahashi, Honoka Kiso, Akiko Murashima-Suginami, Yoshihito Tokita, Jun-Ichiro Jo, Ryuji Uozumi, Yukiko Nambu, Boyen Huang, Hidemitsu Harada, Toshihisa Komori, Manabu Sugai, Yasuhiko Tabata, Kazuhisa Bessho
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-9 (2021)
Abstract Runt-related transcription factor 2 (Runx2)-deficient mice can be used to model congenital tooth agenesis in humans. Conversely, uterine sensitization-associated gene-1 (Usag-1)-deficient mice exhibit supernumerary tooth formation. Arrested
Externí odkaz:
https://doaj.org/article/b69b2500ef85490687eba3c4e049a2f7
Autor:
Katsu Takahashi, Honoka Kiso, Akiko Murashima-Suginami, Yoshihito Tokita, Manabu Sugai, Yasuhiko Tabata, Kazuhisa Bessho
Publikováno v:
Inflammation and Regeneration, Vol 40, Iss 1, Pp 1-9 (2020)
Abstract Analysis of various genetically modified mice, with supernumerary teeth, has revealed the following two intrinsic molecular mechanisms that increase the number of teeth. One plausible explanation for supernumerary tooth formation is the resc
Externí odkaz:
https://doaj.org/article/6df984da5359472497a0e5d6d7596fe5
Autor:
Yumiko Togo, Katsu Takahashi, Kazuyuki Saito, Honoka Kiso, Hiroko Tsukamoto, Boyen Huang, Motoko Yanagita, Manabu Sugai, Hidemitsu Harada, Toshihisa Komori, Akira Shimizu, Mary MacDougall, Kazuhisa Bessho
Publikováno v:
PLoS ONE, Vol 11, Iss 8, p e0161067 (2016)
Supernumerary teeth and tooth agenesis are common morphological anomalies in humans. We previously obtained evidence that supernumerary maxillary incisors form as a result of the successive development of the rudimentary maxillary incisor tooth germ
Externí odkaz:
https://doaj.org/article/076c04ac79814de298e9e3c3c430d045
Autor:
Yoshihito Tokita, Yasuhiko Tabata, Kazuhisa Bessho, Akiko Murashima-Suginami, Manabu Sugai, Honoka Kiso, Katsu Takahashi
Publikováno v:
Inflammation and Regeneration, Vol 40, Iss 1, Pp 1-9 (2020)
Inflammation and Regeneration
Inflammation and Regeneration
Analysis of various genetically modified mice, with supernumerary teeth, has revealed the following two intrinsic molecular mechanisms that increase the number of teeth. One plausible explanation for supernumerary tooth formation is the rescue of too
Autor:
Honoka Kiso, Katsu Takahashi, Kazuyuki Saito, Yumiko Togo, Hiroko Tsukamoto, Boyen Huang, Manabu Sugai, Akira Shimizu, Yasuhiko Tabata, Aris N Economides, Harold C Slavkin, Kazuhisa Bessho
Publikováno v:
PLoS ONE, Vol 9, Iss 5, p e96938 (2014)
Bone morphogenetic proteins (BMPs) are highly conserved signaling molecules that are part of the transforming growth factor (TGF)-beta superfamily, and function in the patterning and morphogenesis of many organs including development of the dentition
Externí odkaz:
https://doaj.org/article/0c57dd312ca5439da3d15ee07a03feff
Autor:
Honoka Kiso, Junichi Takagi, Katsu Takahashi, Yukiko Nambu, Yasuhiko Tabata, Kazuhisa Bessho, Ryuji Uozumi, Yoshihito Tokita, E. Mihara, Akiko Murashima-Suginami, Manabu Sugai
Publikováno v:
Science Advances
Uterine sensitization–associated gene-1 (USAG-1) deficiency leads to enhanced bone morphogenetic protein (BMP) signaling, leading to supernumerary teeth formation. Furthermore, antibodies interfering with binding of USAG-1 to BMP, but not lipoprote
Autor:
Yumiko Togo, Katsu Takahashi, Kazuyuki Saito, Honoka Kiso, Boyen Huang, Hiroko Tsukamoto, Suong-Hyu Hyon, Kazuhisa Bessho
Publikováno v:
Stem Cells International, Vol 2013 (2013)
Background. The expression term of the gene transfected in cells needs to belong enough inorder to make a gene therapy clinically effective. The controlled release of the transfected gene can be utilized. The new biodegradable hydrogel material creat
Externí odkaz:
https://doaj.org/article/d07baf04b21b4ecab8618411d5775cf9
Autor:
Kazuhisa Bessho, Katsu Takahashi, Hironori Haga, Manabu Sugai, Honoka Kiso, Kazumasa Nakao, Akira Shimizu, Masayuki Ikeno, Noriaki Koyama, Yu Isobe
Publikováno v:
Archives of Oral Biology. 93:133-140
Objective Fibrous dysplasia (FD) is a benign bone disease characterized by fibro-osseous lesions. FD is caused by somatic mutations in the gene, guanine nucleotide-binding protein, alpha stimulating activity polypeptide 1 (GNAS), which encodes the G
Autor:
Katsu Takahashi, Masaki Nagata, Toshihisa Komori, Honoka Kiso, Masato Asai, Akira Shimizu, Yoshihito Tokita, Sayaka Mishima, Mary MacDougall, Hiroko Tsukamoto, Kazuhisa Bessho, Manabu Sugai, Boyen Huang, Machiko Iida, Yumiko Togo, Kazuyuki Saito, Masakazu Asahara, Hidemitsu Harada
Publikováno v:
Scientific Reports, Vol 8, Iss 1, Pp 1-13 (2018)
Charles Sturt University
Charles Sturt University
Adult Cebpb KO mice incisors present amelogenin-positive epithelium pearls, enamel and dentin allopathic hyperplasia, fewer Sox2-positive cells in labial cervical loop epitheliums, and reduced Sox2 expression in enamel epithelial stem cells. Thus, Ce
Autor:
Boyen Huang, Katsu Takahashi, Tomoko Sakata, Honoka Kiso, Manabu Sugai, Kazuma Fujimura, Akira Shimizu, Shinji Kosugi, Tosiya Sato, Kazuhisa Bessho
Publikováno v:
PLoS ONE, Vol 6, Iss 10, p e25503 (2011)
OBJECTIVES: This study aimed to carry out a histological examination of the temporomandibular joint (TMJ) in ank mutant mice and to identify polymorphisms of the human ANKH gene in order to establish the relationship between the type of temporomandib
Externí odkaz:
https://doaj.org/article/2d6e1fad1ffa488faa85eae97c7c7b88