Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Hongquan Qin"'
Autor:
Hongquan Qin, Jiali Chen, Katia Bouchekioua-Bouzaghou, Ya-Ming Meng, Jordi Bach Griera, Xue Jiang, Xiangzhan Kong, Minghui Wang, Qiuping Xu, Ping-Pui Wong
Publikováno v:
Journal of Translational Medicine, Vol 21, Iss 1, Pp 1-22 (2023)
Abstract Background Pancreatic ductal adenocarcinoma (PDAC) is characterised by limited responses to chemoimmunotherapy attributed to highly desmoplastic tumor microenvironment. Disrupting the tumor-stromal cell crosstalk is considered as an improved
Externí odkaz:
https://doaj.org/article/57f8cb11ec2f4207954cfdc131aa223a
Autor:
Manna Wang, Zubiao Niu, Hongquan Qin, Banzhan Ruan, You Zheng, Xiangkai Ning, Songzhi Gu, Lihua Gao, Zhaolie Chen, Xiaoning Wang, Hongyan Huang, Li Ma, Qiang Sun
Publikováno v:
Cell Reports, Vol 32, Iss 8, Pp 108071- (2020)
Summary: Entosis is a cell-in-cell (CIC)-mediated death program. Contractile actomyosin (CA) and the adherens junction (AJ) are two core elements essential for entotic CIC formation, but the molecular structures interfacing them remain poorly underst
Externí odkaz:
https://doaj.org/article/05d9a2de96ec4ea2aab350e35ade724e
Autor:
Chenxi Wang, Ang Chen, Banzhan Ruan, Zubiao Niu, Yan Su, Hongquan Qin, You Zheng, Bo Zhang, Lihua Gao, Zhaolie Chen, Hongyan Huang, Xiaoning Wang, Qiang Sun
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 8 (2020)
Though homotypic cell-in-cell (hoCIC) structures are implicated in the development and progression of multiple human tumors, the molecular mechanisms underlying their formation remain poorly understood. We found that the expression of Protocadherin-7
Externí odkaz:
https://doaj.org/article/0e3ad9d64c88461cb8c0611d96a253c4
Autor:
Xin Zhang, Zubiao Niu, Hongquan Qin, Jie Fan, Manna Wang, Bo Zhang, You Zheng, Lihua Gao, Zhaolie Chen, Yanhong Tai, Mo Yang, Hongyan Huang, Qiang Sun
Publikováno v:
Frontiers in Oncology, Vol 9 (2019)
Though current pathological methods are greatly improved, they provide rather limited functional information. Cell-in-cell structures (CICs), arising from active cell–cell interaction, are functional surrogates of complicated cell behaviors within
Externí odkaz:
https://doaj.org/article/ff218b57ea334da3aedb4b46f4c4adad
Autor:
Cheng Huang, Liangping Su, Yitian Chen, Sangqing Wu, Ruipu Sun, Qiuping Xu, Xiaoyi Qiu, Ciqiu Yang, Xiangzhan Kong, Hongquan Qin, Xinbao Zhao, Xue Jiang, Kun Wang, Yinghua Zhu, Ping-Pui Wong
Publikováno v:
Pharmacological research. 187
Dysregulated sphingolipid metabolism contributes to ER+ breast cancer progression and therapeutic response, whereas its underlying mechanism and contribution to tamoxifen resistance (TAMR) is unknown. Here, we establish sphingolipid metabolic enzyme
Autor:
Li Ma, Xiaoyong Sai, Xiaoning Wang, Manna Wang, Qiang Sun, Jianqing Liang, Hongquan Qin, He Ren, Hongyan Huang, Zubiao Niu, Xin Zhang, Zhaolie Chen, Lihua Gao, Songzhi Gu, Banzhan Ruan, Yanhong Tai, You Zheng, Xiaochen Yu, Chenxi Wang, Bo Zhang
Publikováno v:
Cell Death Differ
Entosis was proposed to promote aneuploidy and genome instability by cell-in-cell mediated engulfment in tumor cells. We reported here, in epithelial cells, that entosis coupled with mitotic arrest functions to counteract genome instability by target
Autor:
Xiangkai Ning, Xiaoning Wang, Manna Wang, Hongquan Qin, Qiang Sun, Songzhi Gu, Li Ma, Zubiao Niu, You Zheng, Banzhan Ruan, Hongyan Huang, Lihua Gao, Zhaolie Chen
Publikováno v:
Cell Reports, Vol 32, Iss 8, Pp 108071-(2020)
Summary: Entosis is a cell-in-cell (CIC)-mediated death program. Contractile actomyosin (CA) and the adherens junction (AJ) are two core elements essential for entotic CIC formation, but the molecular structures interfacing them remain poorly underst
Autor:
Jianqing Liang, Xiaoyong Sai, Li Ma, Xiaochen Yu, Yanhong Tai, You Zheng, Zhaolie Chen, Lihua Gao, Manna Wang, Qiang Sun, Banzhan Ruan, Bo Zhang, Hongquan Qin, Zubiao Niu, Songzhi Gu, Xiaoning Wang, Hongyan Huang, Xin Zhang
Entosis was proposed to promote aneuploidy and genome instability by cell-in-cell mediated engulfment in tumor cells. We reported here, in epithelial cells, that entosis coupled with mitotic arrest functions to counteract genome instability by target
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ecc7c13f50f10777624d8bec4ad0aebc