Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Hongchu Zha"'
Autor:
Zhixia Song, Yao Xia, Lang Shi, Hongchu Zha, Jing Huang, Xiaohong Xiang, Huiming Li, Hua Huang, Ruchi Yue, Hongtao Wang, Jiefu Zhu
Publikováno v:
Cellular & Molecular Biology Letters, Vol 29, Iss 1, Pp 1-23 (2024)
Abstract Background Acute kidney injury (AKI) is a common clinical disorder with complex etiology and poor prognosis, and currently lacks specific and effective treatment options. Mitochondrial dynamics dysfunction is a prominent feature in AKI, and
Externí odkaz:
https://doaj.org/article/ffc7cd57f8f943378dbd5017610ac902
Autor:
Lang Shi, Hongchu Zha, Zhou Pan, Jiayi Wang, Yao Xia, Huimin Li, Hua Huang, Ruchi Yue, Zhixia Song, Jiefu Zhu
Publikováno v:
Cell Death and Disease, Vol 14, Iss 11, Pp 1-16 (2023)
Abstract The mechanism underlying acute kidney injury (AKI) and AKI-to-Chronic kidney disease (CKD) transition remains unclear, but mitochondrial dysfunction may be a key driving factor. Literature reports suggest that dual-specificity phosphatase 1
Externí odkaz:
https://doaj.org/article/6aa7cbf26c994fa481b1ef066fe9c7eb
Publikováno v:
Renal Failure, Vol 44, Iss 1, Pp 694-705 (2022)
Objective To investigate the effect of vitamin D/vitamin D receptor (VDR)/Atg16L1 signaling on podocyte autophagy and survival in diabetic nephropathy.Methods Diabetic rat models were induced by intraperitoneal injection of streptozotocin (STZ) (60 m
Externí odkaz:
https://doaj.org/article/ba2307373e1d43fabcde73eb41471094
Publikováno v:
International Journal of Molecular Medicine; Nov2023, Vol. 52 Issue 5, p1-13, 13p
Publikováno v:
International immunopharmacology. 109
Acute kidney injury (AKI) is a critical and severe clinical disease caused by a variety of factors. Toll-like receptors (TLRs) play a crucial role in pathogenesis of AKI. Radioprotective 105 kDa protein (RP105) is a member of the TLR family, but the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0894c20db423563dacb563fe67518312
https://pubmed.ncbi.nlm.nih.gov/35696803
https://pubmed.ncbi.nlm.nih.gov/35696803