A glial circadian gene expression atlas reveals cell type and disease-specific reprogramming in response to amyloid pathology or aging.

Autor: Sheehan PW; Department of Neurology, Washington University School of Medicine, Saint Louis MO, USA., Fass S; Department of Genetics, Washington University School of Medicine, Saint Louis MO, USA.; Department of Psychiatry, Washington University School of Medicine, Saint Louis MO, USA., Sapkota D; Department of Genetics, Washington University School of Medicine, Saint Louis MO, USA.; Department of Psychiatry, Washington University School of Medicine, Saint Louis MO, USA.; Department of Biological Sciences and Department of Neuroscience, University of Texas at Dallas, Richardson, TX, USA., Kang S; Department of Neuroscience, Mayo Clinic, Jacksonville, FL, USA., Hollis HC; School of Biomedical Engineering and Health Systems, Drexel University, Philadelphia, PA, USA., Lawrence JH; Department of Neurology, Washington University School of Medicine, Saint Louis MO, USA., Anafi RC; Department of Medicine, Chronobiology, and Sleep Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA., Dougherty JD; Department of Genetics, Washington University School of Medicine, Saint Louis MO, USA.; Department of Psychiatry, Washington University School of Medicine, Saint Louis MO, USA.; Intellectual and Developmental Disabilities Research Center, Washington University School of Medicine, St. Louis, MO, USA., Fryer JD; Department of Neuroscience, Mayo Clinic, Scottsdale, AZ, USA., Musiek ES; Department of Neurology, Washington University School of Medicine, Saint Louis MO, USA.; Center on Biological Rhythms and Sleep, Washington University School of Medicine, St. Louis, MO, USA.; Lead contact.

Publikováno v: BioRxiv : the preprint server for biology [bioRxiv] 2024 Jun 01. Date of Electronic Publication: 2024 Jun 01.