Zobrazeno 1 - 10
of 68
pro vyhledávání: '"Hofer Wong"'
Publikováno v:
The journal of allergy and clinical immunology. Global, vol 1, iss 4
BackgroundExposure to O3 has been associated with increased risk of exacerbations of asthma, but the underlying mechanisms are not well studied. We hypothesized that O3 exposure would enhance airway inflammatory responses to allergen and the GSTM1 nu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3769ac3d78858f3ee0a22442effd0d8a
https://escholarship.org/uc/item/7wf534rm
https://escholarship.org/uc/item/7wf534rm
Autor:
Pascale Leroy, Andrea Tham, Hofer Wong, Rachel Tenney, Chun Chen, Rachel Stiner, John R Balmes, Agnès C Paquet, Mehrdad Arjomandi
Publikováno v:
PLoS ONE, Vol 10, Iss 6, p e0127283 (2015)
Inhalation of ambient levels of ozone causes airway inflammation and epithelial injury.To examine the responses of airway cells to ozone-induced oxidative injury, 19 subjects (7 with asthma) were exposed to clean air (0ppb), medium (100ppb), and high
Externí odkaz:
https://doaj.org/article/bfb8d0b7232c45d1b91afc95acc7feca
Autor:
Mehrdad Arjomandi, Jessica Frelinger, Aneesh Donde, Hofer Wong, Amritha Yellamilli, Wilfred Raymond
Publikováno v:
PLoS ONE, Vol 6, Iss 10, p e25678 (2011)
Osteopontin (OPN) is a member of the small integrin-binding ligand N-linked glycoprotein (SIBLING) family and a cytokine with diverse biologic roles. OPN undergoes extensive post-translational modifications, including polymerization and proteolytic f
Externí odkaz:
https://doaj.org/article/3c2492ff16a845989cdbe21ac43a2b29
Publikováno v:
Airway Cell Biology and Immunopathology.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e20afc3948108b83f42f1adb2e5f1245
https://doi.org/10.1183/13993003.congress-2018.pa4970
https://doi.org/10.1183/13993003.congress-2018.pa4970
Autor:
Sarah Dalton, Jessica Frelinger, Aneesh Donde, Mehrdad Arjomandi, Karron Power, John R. Balmes, Hofer Wong, Wendy Ching
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 308:H1499-H1509
Epidemiological evidence suggests that exposure to ozone increases cardiovascular morbidity. However, the specific biological mechanisms mediating ozone-associated cardiovascular effects are unknown. To determine whether short-term exposure to ambien
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c483fb25bee31acb7e5cbd5c9df1ac15
https://doi.org/10.1152/ajpheart.00849.2014
https://doi.org/10.1152/ajpheart.00849.2014
Autor:
Subha Venkat, Veronica Davé, Mehrdad Arjomandi, Hofer Wong, Aneesh Donde, Nina Holland, John R. Balmes
Publikováno v:
Environmental and Molecular Mutagenesis. 56:378-387
Ozone is an important constituent of ambient air pollution and represents a major public health concern. Oxidative injury due to ozone inhalation causes the generation of reactive oxygen species and can be genotoxic. To determine whether ozone exposu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::91d9e080b26ef99bad7744ec26fe921b
https://doi.org/10.1002/em.21921
https://doi.org/10.1002/em.21921
Autor:
Sarah Nasir, Suzaynn F. Schick, D. Ken Giles, Hofer Wong, Donald Lucas, Justin Kim, John R. Balmes, Jiaxi Fang, Bryan M. Jenkins, Kathryn F. Farraro
Publikováno v:
Aerosol Science and Technology. 46:1246-1255
Research has shown that cigarette smoke changes chemically and physically after it is released into indoor air, that these changes can increase secondhand smoke (SHS) toxicity, and that acute exposures to even low levels of SHS increase the risk of c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c245d74451a528f836fa31331dae6885
https://doi.org/10.1080/02786826.2012.708947
https://doi.org/10.1080/02786826.2012.708947
Inflammatory and Repair Pathways Induced in Human Bronchoalveolar Lavage Cells with Ozone Inhalation
Autor:
Andrea Tham, Mehrdad Arjomandi, Chun Chen, Rachel K Stiner, Pascale Leroy, Agnès Paquet, Hofer Wong, John R. Balmes, Rachel Tenney
Publikováno v:
PLoS ONE
PLoS ONE, Public Library of Science, 2015, 10 (6), pp.e0127283. ⟨10.1371/journal.pone.0127283⟩
PLoS ONE, Vol 10, Iss 6, p e0127283 (2015)
PLoS ONE, Public Library of Science, 2015, 10 (6), pp.e0127283. ⟨10.1371/journal.pone.0127283⟩
PLoS ONE, Vol 10, Iss 6, p e0127283 (2015)
Background Inhalation of ambient levels of ozone causes airway inflammation and epithelial injury. Methods To examine the responses of airway cells to ozone-induced oxidative injury, 19 subjects (7 with asthma) were exposed to clean air (0ppb), mediu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e58d2d89a2b7992844b4492972cf2912
http://europepmc.org/articles/PMC4452717
http://europepmc.org/articles/PMC4452717
Autor:
John V. Fahy, Colin Solomon, Jeffrey Ratto, Hofer Wong, Jane Liu, Homer A. Boushey, John R. Balmes
Publikováno v:
Environmental Health Perspectives
Environmental health perspectives, vol 114, iss 2
Environmental health perspectives, vol 114, iss 2
Single short-term exposures to ozone are known to cause acute changes in pulmonary function and neutrophilic airway inflammation. The respiratory health effects of repeated exposures are not as well studied. Pulmonary function decrements are known to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dab1ebe66aef65777ca7ba8b4fa1fe0c
http://europepmc.org/articles/PMC1367833
http://europepmc.org/articles/PMC1367833
Autor:
Owen D. Solberg, Roderick Carter, Prescott G. Woodruff, Hofer Wong, Ronald E. Ferrando, Gregory Dolganov, Peggy S. Cadbury, John V. Fahy, Samantha Donnelly, Steven R. Hays
Publikováno v:
American Journal of Respiratory and Critical Care Medicine. 169:1001-1006
Bronchial hyperresponsiveness in mild to moderate asthma may result from airway smooth muscle cell proliferation or acquisition of a hypercontractile phenotype. Because these cells have not been well characterized in mild to moderate asthma, we exami
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b0885772b919d00c38a26b3f01953ffa
https://doi.org/10.1164/rccm.200311-1529oc
https://doi.org/10.1164/rccm.200311-1529oc