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Stress-experienced monocytes/macrophages lose anti-inflammatory function via β2-adrenergic receptor in skin allergic inflammation.

Autor: Urakami H; Department of Cellular Physiology, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan; Department of Dermatology, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan., Yoshikawa S; Department of Cellular Physiology, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan; Department of Immunology, School of Medicine, Juntendo University, Tokyo, Japan; Juntendo Itch Research Center (JIRC), Institute for Environmental and Gender Specific Medicine, Juntendo University Graduate school of Medicine, Chiba, Japan. Electronic address: s.yoshikawa.jm@juntendo.ac.jp., Nagao K; Department of Cellular Physiology, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan; Department of Immunology, School of Medicine, Juntendo University, Tokyo, Japan; Juntendo Itch Research Center (JIRC), Institute for Environmental and Gender Specific Medicine, Juntendo University Graduate school of Medicine, Chiba, Japan., Miyake K; Inflammation, Infection & Immunity Laboratory, Advanced Research Institute, Tokyo Medical and Dental University (TMDU), Tokyo, Japan., Fujita Y; Department of Cellular Physiology, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan; Department of Immunology, School of Medicine, Juntendo University, Tokyo, Japan., Komura A; Department of Cellular Physiology, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan., Nakashima M; Department of Cellular Physiology, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan., Umene R; Department of Physiology of Visceral Function and Body Fluid, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan., Sano S; Department of Immunology, School of Medicine, Juntendo University, Tokyo, Japan., Hu Z; Department of Immunology, School of Medicine, Juntendo University, Tokyo, Japan., Nishii E; Department of Immunology, School of Medicine, Juntendo University, Tokyo, Japan., Fujimura A; Department of Cellular Physiology, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan., Hiyama TY; Department of Integrative Physiology, Tottori University Graduate School and Faculty of Medicine, Yonago, Japan; International Platform for Dryland Research and Education, Tottori University, Tottori, Japan., Naruse K; Department of Cellular Physiology, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan; Department of Cardiovascular Physiology, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan., Karasuyama H; Inflammation, Infection & Immunity Laboratory, Advanced Research Institute, Tokyo Medical and Dental University (TMDU), Tokyo, Japan., Inoue T; Department of Physiology of Visceral Function and Body Fluid, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan., Tominaga M; Juntendo Itch Research Center (JIRC), Institute for Environmental and Gender Specific Medicine, Juntendo University Graduate school of Medicine, Chiba, Japan., Takamori K; Juntendo Itch Research Center (JIRC), Institute for Environmental and Gender Specific Medicine, Juntendo University Graduate school of Medicine, Chiba, Japan., Morizane S; Department of Dermatology, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan. Electronic address: zanemori@cc.okayama-u.ac.jp., Miyake S; Department of Immunology, School of Medicine, Juntendo University, Tokyo, Japan. Electronic address: s-miyake@juntendo.ac.jp.

Publikováno v: The Journal of allergy and clinical immunology [J Allergy Clin Immunol] 2024 Nov 18. Date of Electronic Publication: 2024 Nov 18.

P2X4 receptor mediates macrophage infiltration leading to endometriotic cyst epithelium proliferation and hyperalgesia in mouse model.

Autor: Nagata H; Division of Obstetrics and Gynecology, Tottori University Graduate School and Faculty of Medicine, Yonago, Tottori, Japan., Hiyama TY; Department of Integrative Physiology, Tottori University Graduate School and Faculty of Medicine, Yonago, Tottori, Japan; International Platform for Dryland Research and Education, Tottori University, Tottori, Tottori, Japan. Electronic address: hiyama@tottori-u.ac.jp., Inoue M; Department of Integrative Physiology, Tottori University Graduate School and Faculty of Medicine, Yonago, Tottori, Japan., Xu S; Department of Integrative Physiology, Tottori University Graduate School and Faculty of Medicine, Yonago, Tottori, Japan., Wada I; Division of Obstetrics and Gynecology, Tottori University Graduate School and Faculty of Medicine, Yonago, Tottori, Japan., Yoshimura Y; Department of Integrative Physiology, Tottori University Graduate School and Faculty of Medicine, Yonago, Tottori, Japan., Nakamura K; Advanced Medicine, Innovation and Clinical Research Center, Tottori University Hospital, Yonago, Tottori, Japan., Azuma Y; Division of Obstetrics and Gynecology, Tottori University Graduate School and Faculty of Medicine, Yonago, Tottori, Japan., Harada T; Division of Obstetrics and Gynecology, Tottori University Graduate School and Faculty of Medicine, Yonago, Tottori, Japan., Taniguchi F; Division of Obstetrics and Gynecology, Tottori University Graduate School and Faculty of Medicine, Yonago, Tottori, Japan.

Publikováno v: F&S science [F S Sci] 2024 Oct 22. Date of Electronic Publication: 2024 Oct 22.