Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Hitomi Yamamoto‐Imoto"'
Publikováno v:
STAR Protocols, Vol 3, Iss 3, Pp 101539- (2022)
Summary: Senescent cells accumulation is associated with aging and age-related diseases, and recent findings suggest that autophagy, the activity of the intracellular degradation system, decreases during senescence. In this protocol, we detail steps
Externí odkaz:
https://doaj.org/article/898d9cf009e3410dac5c44839b9ad729
Autor:
Georgette L. Suidan, Pradeep K. Singh, Sunita Patel-Hett, Zu-Lin Chen, Dmitri Volfson, Hitomi Yamamoto-Imoto, Erin H. Norris, Robert D. Bell, Sidney Strickland
Publikováno v:
Blood Advances, Vol 2, Iss 9, Pp 954-963 (2018)
Abstract: Alzheimer disease (AD) is a neurodegenerative disorder characterized by extracellular β-amyloid (Aβ) deposition. Although peripheral inflammation and cerebrovascular pathology are reported in AD, there is a lack of plasma biomarkers in th
Externí odkaz:
https://doaj.org/article/0e4f33cbf5d94a24b3f2a05f21603c2b
Autor:
Hitomi Yamamoto‐Imoto, Daria Zamolodchikov, Zu‐Lin Chen, S. Lloyd Bourne, Syeda Rizvi, Pradeep Singh, Erin H. Norris, Frances Weis‐Garcia, Sidney Strickland
Publikováno v:
Alzheimer’s & Dementia: Diagnosis, Assessment & Disease Monitoring, Vol 10, Iss 1, Pp 480-489 (2018)
Abstract Introduction Accumulation of β‐amyloid is a pathological hallmark of Alzheimer's disease (AD). β‐Amyloid activates the plasma contact system leading to kallikrein‐mediated cleavage of intact high‐molecular‐weight kininogen (HKi)
Externí odkaz:
https://doaj.org/article/736a60c21d2c42abb28c56a2c65bc95c
Publikováno v:
Progress in Biophysics and Molecular Biology. 179:10-15
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::6903b6e21e9e041b779c41658e3f6806
https://doi.org/10.1016/j.pbiomolbio.2023.02.005
https://doi.org/10.1016/j.pbiomolbio.2023.02.005
Autor:
Mengying Cui, Koji Yamano, Kenichi Yamamoto, Hitomi Yamamoto-Imoto, Satoshi Minami, Takeshi Yamamoto, Sho Matsui, Tatsuya Kaminishi, Takayuki Shima, Monami Ogura, Megumi Tsuchiya, Kohei Nishino, Layden, Brian T., Hisakazu Kato, Hidesato Ogawa, Shinya Oki, Yukinori Okada, Yoshitaka Isaka, Hidetaka Kosako, Noriyuki Matsuda
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America; 1/9/2024, Vol. 121 Issue 2, p1-12, 42p
Autor:
Keiko Yamato, Hiroji Yanamoto, Jun C. Takahashi, Hitomi Yamamoto-Imoto, Toshiyuki Miyata, Yukako Nakajo, Hiroharu Kataoka, Koichi Kokame
Publikováno v:
Neurosurgery Open. 1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::772e4e70e3f667db2ee9ccc9bbe3cb73
https://doi.org/10.1093/neuopn/okaa014
https://doi.org/10.1093/neuopn/okaa014
Autor:
Hitomi Yamamoto-Imoto, Satoshi Minami, Tatsuya Shioda, Yurina Yamashita, Shinsuke Sakai, Shihomi Maeda, Takeshi Yamamoto, Shinya Oki, Mizuki Takashima, Tadashi Yamamuro, Kyosuke Yanagawa, Ryuya Edahiro, Miki Iwatani, Mizue So, Ayaka Tokumura, Toyofumi Abe, Ryoichi Imamura, Norio Nonomura, Yukinori Okada, Donald E. Ayer, Hidesato Ogawa, Eiji Hara, Yoshitsugu Takabatake, Yoshitaka Isaka, Shuhei Nakamura, Tamotsu Yoshimori
Publikováno v:
Cell Reports. 38:110444
Accumulation of senescent cells affects organismal aging and the prevalence of age-associated disease. Emerging evidence suggests that activation of autophagy protects against age-associated diseases and promotes longevity, but the roles and regulato
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4032ba94a04b9e59cadf556c264cea78
https://doi.org/10.1016/j.celrep.2022.110444
https://doi.org/10.1016/j.celrep.2022.110444
Autor:
Pradeep K. Singh, Sidney Strickland, Daria Zamolodchikov, Frances Weis-Garcia, Syeda Rizvi, Zu-Lin Chen, S. Lloyd Bourne, Erin H. Norris, Hitomi Yamamoto-Imoto
Publikováno v:
Alzheimer’s & Dementia: Diagnosis, Assessment & Disease Monitoring, Vol 10, Iss 1, Pp 480-489 (2018)
Alzheimer's & Dementia : Diagnosis, Assessment & Disease Monitoring
Alzheimer's & Dementia : Diagnosis, Assessment & Disease Monitoring
Introduction Accumulation of β-amyloid is a pathological hallmark of Alzheimer's disease (AD). β-Amyloid activates the plasma contact system leading to kallikrein-mediated cleavage of intact high-molecular-weight kininogen (HKi) to cleaved high-mol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b303a91680fc691fff9123175c8db9b0
https://doi.org/10.1016/j.dadm.2018.06.008
https://doi.org/10.1016/j.dadm.2018.06.008
Autor:
Zu-Lin Chen, Sidney Strickland, Robert D. Bell, Georgette L. Suidan, Hitomi Yamamoto-Imoto, Sunita Patel-Hett, Erin H. Norris, Pradeep K. Singh, Dmitri Volfson
Alzheimer disease (AD) is a neurodegenerative disorder characterized by extracellular β-amyloid (Aβ) deposition. Although peripheral inflammation and cerebrovascular pathology are reported in AD, there is a lack of plasma biomarkers in this field.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3c7cee5eddd92aca28cd6ea92bcae8f0
https://europepmc.org/articles/PMC5942003/
https://europepmc.org/articles/PMC5942003/
Autor:
Yukako Oe, Shuhei Nakamura, Tamotsu Yoshimori, Kentaro Yamamoto, Kyle L. Morris, Goran Stjepanovic, James H. Hurley, Lindsey N. Young, Gerhard Hummer, Hitomi Yamamoto-Imoto, Chunmei Chang, Sören von Bülow, Johannes Schöneberg
Publikováno v:
Molecular Cell. 73:339-353.e6
Membrane targeting of the BECN1-containing class III PI 3-kinase (PI3KC3) complexes is pivotal to the regulation of autophagy. The interaction of PI3KC3 complex II and its ubiquitously expressed inhibitor, Rubicon, was mapped to the first β sheet of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::fff6cbc6852f36ba9d4e03311d8dfb36
https://doi.org/10.1016/j.molcel.2018.10.035
https://doi.org/10.1016/j.molcel.2018.10.035