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Absence of ULK1 decreases AMPK activity in the kidney, leading to chronic kidney disease progression
Autor:
Tomoki Yanagi, Hiroaki Kikuchi, Koichiro Susa, Naohiro Takahashi, Hiroki Bamba, Takefumi Suzuki, Yuta Nakano, Tamami Fujiki, Yutaro Mori, Fumiaki Ando, Shintaro Mandai, Takayasu Mori, Koh Takeuchi, Shinya Honda, Satoru Torii, Shigeomi Shimizu, Tatemitsu Rai, Shinichi Uchida, Eisei Sohara
Publikováno v:
Genes to cells : devoted to molecularcellular mechanismsREFERENCES.
AMP-activated protein kinase (AMPK) inactivation in chronic kidney disease (CKD) leads to energy status deterioration in the kidney, constituting the vicious cycle of CKD exacerbation. Unc-51-like kinase 1 (ULK1) is considered a downstream molecule o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::aadaba469d69d45dc5f7b6727b547b9a
https://pubmed.ncbi.nlm.nih.gov/36318474
https://pubmed.ncbi.nlm.nih.gov/36318474
