Zobrazeno 1 - 10 of 133 pro vyhledávání: '"Hing TC"'
2
Akademický článek
CSA13 inhibits colitis-associated intestinal fibrosis via a formyl peptide receptor like-1 mediated HMG-CoA reductase pathway.
Autor: Xu C; Center for Inflammatory Bowel Diseases, Vatche and Tamar Manoukian Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, 90095, USA.; The Key Laboratory for Space Bioscience and Biotechnology, School of Life Science, Northwestern Polytechnical University, Xian, Shaanxi Province, China., Ghali S; Center for Inflammatory Bowel Diseases, Vatche and Tamar Manoukian Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, 90095, USA., Wang J; Center for Inflammatory Bowel Diseases, Vatche and Tamar Manoukian Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, 90095, USA.; Department of Gastroenterology, First Affiliated Hospital, China Medical University, Shenyang City, Liaoning Province, China., Shih DQ; F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA, 90048, USA., Ortiz C; Center for Inflammatory Bowel Diseases, Vatche and Tamar Manoukian Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, 90095, USA., Mussatto CC; Center for Inflammatory Bowel Diseases, Vatche and Tamar Manoukian Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, 90095, USA., Lee EC; Center for Inflammatory Bowel Diseases, Vatche and Tamar Manoukian Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, 90095, USA., Tran DH; Center for Inflammatory Bowel Diseases, Vatche and Tamar Manoukian Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, 90095, USA., Jacobs JP; Center for Inflammatory Bowel Diseases, Vatche and Tamar Manoukian Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, 90095, USA., Lagishetty V; Center for Inflammatory Bowel Diseases, Vatche and Tamar Manoukian Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, 90095, USA., Fleshner P; F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA, 90048, USA., Robbins L; F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA, 90048, USA., Vu M; F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA, 90048, USA., Hing TC; F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA, 90048, USA., McGovern DPB; F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA, 90048, USA., Koon HW; Center for Inflammatory Bowel Diseases, Vatche and Tamar Manoukian Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, 90095, USA. hkoon@mednet.ucla.edu.
Publikováno v: Scientific reports [Sci Rep] 2017 Nov 27; Vol. 7 (1), pp. 16351. Date of Electronic Publication: 2017 Nov 27.
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3
Akademický článek
Cathelicidin suppresses lipid accumulation and hepatic steatosis by inhibition of the CD36 receptor.
Autor: Hoang-Yen Tran D; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Hoang-Ngoc Tran D; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Mattai SA; Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Sallam T; Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Ortiz C; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Lee EC; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Robbins L; Department of Medicine, Cedars-Sinai Medical Center, F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Los Angeles, CA, USA., Ho S; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Lee JE; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Fisseha E; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Shieh C; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Sideri A; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Shih DQ; Department of Medicine, Cedars-Sinai Medical Center, F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Los Angeles, CA, USA., Fleshner P; Department of Medicine, Cedars-Sinai Medical Center, F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Los Angeles, CA, USA., McGovern DP; Department of Medicine, Cedars-Sinai Medical Center, F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Los Angeles, CA, USA., Vu M; Department of Medicine, Cedars-Sinai Medical Center, F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Los Angeles, CA, USA., Hing TC; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Bakirtzi K; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Cheng M; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Su B; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Law I; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Karagiannides I; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Targan SR; Department of Medicine, Cedars-Sinai Medical Center, F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Los Angeles, CA, USA., Gallo RL; Division of Dermatology, Department of Medicine, the University of California San Diego, San Diego, CA, USA., Li Z; Division of Clinical Nutrition, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA., Koon HW; Division of Digestive Diseases, Department of Medicine, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA, USA.
Publikováno v: International journal of obesity (2005) [Int J Obes (Lond)] 2016 Sep; Vol. 40 (9), pp. 1424-34. Date of Electronic Publication: 2016 May 10.
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4
Akademický článek
Anti-fibrogenic effects of the anti-microbial peptide cathelicidin in murine colitis-associated fibrosis.
Autor: Yoo JH; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA ; Digestive Disease Center, CHA University Bundang Medical Center, Seongnam 463-712, Republic of Korea., Ho S; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA., Tran DH; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA., Cheng M; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA., Bakirtzi K; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA., Kukota Y; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA., Ichikawa R; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA., Su B; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA., Tran DH; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA., Hing TC; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA., Chang I; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA., Shih DQ; F. Widjaja Foundation, Inflammatory Bowel & Immunobiology Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA 90048, USA., Issacson RE; Department of Veterinary and Biomedical Sciences, University of Minnesota, St. Paul, MN 55108, USA., Gallo RL; Division of Dermatology, University of California San Diego, San Diego, CA 92093, USA., Fiocchi C; Department of Pathobiology, The Cleveland Clinic Foundation, Cleveland, OH 44195, USA., Pothoulakis C; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA., Koon HW; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, CA 90095, USA.
Publikováno v: Cellular and molecular gastroenterology and hepatology [Cell Mol Gastroenterol Hepatol] 2015 Jan; Vol. 1 (1), pp. 55-74.e1.
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5
Akademický článek
Fidaxomicin inhibits Clostridium difficile toxin A-mediated enteritis in the mouse ileum.
Autor: Koon HW; Center for Inflammatory Bowel Diseases, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, California, USA., Ho S; Center for Inflammatory Bowel Diseases, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, California, USA., Hing TC; Center for Inflammatory Bowel Diseases, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, California, USA., Cheng M; Center for Inflammatory Bowel Diseases, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, California, USA., Chen X; Division of Gastroenterology, Department of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA., Ichikawa Y; Cubist Pharmaceuticals, Inc., Lexington, Massachusetts, USA., Kelly CP; Division of Gastroenterology, Department of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA., Pothoulakis C; Center for Inflammatory Bowel Diseases, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, California, USA cpothoulakis@mednet.ucla.edu.
Publikováno v: Antimicrobial agents and chemotherapy [Antimicrob Agents Chemother] 2014 Aug; Vol. 58 (8), pp. 4642-50. Date of Electronic Publication: 2014 Jun 02.
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6
Akademický článek
Development of an antibody fused with an antimicrobial peptide targeting Pseudomonas aeruginosa: A new approach to prevent and treat bacterial infections.
Autor: Johnson, Kenneth1 (AUTHOR), Delaney, James C.1 (AUTHOR), Guillard, Thomas2 (AUTHOR), Reffuveille, Fany2 (AUTHOR), Varin-Simon, Jennifer2 (AUTHOR), Li, Kai1 (AUTHOR), Wollacott, Andrew1 (AUTHOR), Frapy, Eric3 (AUTHOR), Mong, Surin4 (AUTHOR), Tissire, Hamid1 (AUTHOR), Viswanathan, Karthik1 (AUTHOR), Touti, Faycal4 (AUTHOR), Babcock, Gregory J.1 (AUTHOR), Shriver, Zachary1 (AUTHOR) zshriver@visterrainc.com, Pentelute, Bradley L.4 (AUTHOR), Plante, Obadiah1 (AUTHOR), Skurnik, David3,5,6 (AUTHOR) zshriver@visterrainc.com
Publikováno v: PLoS Pathogens. 9/7/2023, Vol. 19 Issue 9, p1-24. 24p.
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7
Akademický článek
The antimicrobial peptide cathelicidin modulates Clostridium difficile-associated colitis and toxin A-mediated enteritis in mice.
Autor: Hing TC; Inflammatory Bowel Disease Center, Division of Digestive Diseases, the University of California Los Angeles, Los Angeles, California, USA., Ho S, Shih DQ, Ichikawa R, Cheng M, Chen J, Chen X, Law I, Najarian R, Kelly CP, Gallo RL, Targan SR, Pothoulakis C, Koon HW
Publikováno v: Gut [Gut] 2013 Sep; Vol. 62 (9), pp. 1295-305. Date of Electronic Publication: 2012 Jul 03.
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8
Akademický článek
Human monoclonal antibodies against Clostridium difficile toxins A and B inhibit inflammatory and histologic responses to the toxins in human colon and peripheral blood monocytes.
Autor: Koon HW; Center for Inflammatory Bowel Diseases, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, California, USA., Shih DQ, Hing TC, Yoo JH, Ho S, Chen X, Kelly CP, Targan SR, Pothoulakis C
Publikováno v: Antimicrobial agents and chemotherapy [Antimicrob Agents Chemother] 2013 Jul; Vol. 57 (7), pp. 3214-23. Date of Electronic Publication: 2013 Apr 29.
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9
Akademický článek
Cathelicidin signaling via the Toll-like receptor protects against colitis in mice.
Autor: Koon HW; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at University of California Los Angeles, Los Angeles, California 90095, USA. hkoon@mednet.ucla.edu, Shih DQ, Chen J, Bakirtzi K, Hing TC, Law I, Ho S, Ichikawa R, Zhao D, Xu H, Gallo R, Dempsey P, Cheng G, Targan SR, Pothoulakis C
Publikováno v: Gastroenterology [Gastroenterology] 2011 Nov; Vol. 141 (5), pp. 1852-63.e1-3. Date of Electronic Publication: 2011 Jul 14.
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10
Akademický článek
Substance P induces CCN1 expression via histone deacetylase activity in human colonic epithelial cells.
Autor: Koon HW; Inflammatory Bowel Disease Center, Division of Digestive Diseases, David Geffen School of Medicine at the University of California Los Angeles, Los Angeles, California, USA., Shih DQ, Hing TC, Chen J, Ho S, Zhao D, Targan SR, Pothoulakis C
Publikováno v: The American journal of pathology [Am J Pathol] 2011 Nov; Vol. 179 (5), pp. 2315-26. Date of Electronic Publication: 2011 Sep 23.
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