Výsledky vyhledávání - "Hillary Maillet"

Akademický článek

A method for the efficient evaluation of substrate-based cholinesterase imaging probes for Alzheimer’s disease

Autor: Sultan Darvesh, Scott Banfield, Maeve Dufour, Katrina L. Forrestall, Hillary Maillet, G. Andrew Reid, Dane Sands, Ian R. Pottie

Publikováno v: Journal of Enzyme Inhibition and Medicinal Chemistry, Vol 38, Iss 1 (2023)

Cholinesterase (ChE) enzymes have been identified as diagnostic markers for Alzheimer disease (AD). Substrate-based probes have been synthesised to detect ChEs but they have not detected changes in ChE distribution associated with AD pathology. Probe

Externí odkaz: https://doaj.org/article/c958e74950c54e83ac55b4fa6fec5a01

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Microarray decitabine-induced gene expression changes from Decitabine Response in Breast Cancer Requires Efficient Drug Processing and Is Not Limited by Multidrug Resistance

Autor: Paola Marcato, Ian C.G. Weaver, Carman A. Giacomantonio, Kerry B. Goralski, David M. Waisman, Hillary Maillet, Krysta M. Coyle, Steven R. Hall, Ryan W. Holloway, Cheryl Dean, Ainsleigh J. Jackson, Brianne M. Cruickshank, Margaret L. Dahn

Microarray gene expression analyses for MDA-MB-468, MDA-MB-231, and SUM159 cells after decitabine treatment

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::665472946697f97513d3da2193889282
https://doi.org/10.1158/1535-7163.22521439

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GSEA for decitabine up-regulated genes from Decitabine Response in Breast Cancer Requires Efficient Drug Processing and Is Not Limited by Multidrug Resistance

Gene Set Enrichment Analysis for genes upregulated by decitabine treatment in MDA-MB-468, MDA-MB-231, or SUM159

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::692e93d3cd5ee79e2f56d7879754dd43
https://doi.org/10.1158/1535-7163.22521445

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HM450 for genes with altered expression from Decitabine Response in Breast Cancer Requires Efficient Drug Processing and Is Not Limited by Multidrug Resistance

HM450 DNA methylation B-values for genes up- or down-regulated by decitabine in MDA-MB-468, MDA-MB-231, or SUM159

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8322c2e62101bf8066dce9e13b6508c8
https://doi.org/10.1158/1535-7163.22521442.v1

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Supplementary Materials from Decitabine Response in Breast Cancer Requires Efficient Drug Processing and Is Not Limited by Multidrug Resistance

Additional figures, tables, and methods

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::16ee0ce418e51f19b6dcef9a90694f58
https://doi.org/10.1158/1535-7163.22521436

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Up-regulated Hypermethylated genes from Decitabine Response in Breast Cancer Requires Efficient Drug Processing and Is Not Limited by Multidrug Resistance

Genes up-regulated by decitabine treatment in MDA-MB-468, MDA-MB-231, or SUM159 with hypermethylated promoters (TSS200)

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0f824b0643fe23b65ae050975a3b15c9
https://doi.org/10.1158/1535-7163.22521433.v1

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Decitabine Response in Breast Cancer Requires Efficient Drug Processing and Is Not Limited by Multidrug Resistance

Autor: David M. Waisman, Hillary Maillet, Carman A. Giacomantonio, Paola Marcato, Steven R. Hall, Ian C. G. Weaver, Brianne M. Cruickshank, Margaret L. Dahn, Krysta M. Coyle, Cheryl A. Dean, Ryan W. Holloway, Kerry B. Goralski, Ainsleigh J. Jackson

Publikováno v: Molecular cancer therapeutics. 19(5)

Dysregulation of DNA methylation is an established feature of breast cancers. DNA demethylating therapies like decitabine are proposed for the treatment of triple-negative breast cancers (TNBC) and indicators of response need to be identified. For th

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::88df59f2c5e406c4fe2f51622e9d2ef0
https://pubmed.ncbi.nlm.nih.gov/32156786

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