Zobrazeno 1 - 10
of 32
pro vyhledávání: '"Hidetoshi Akimoto"'
Reality and possibility of smoking cessation support and collaborative approach by dental hygienists
Publikováno v:
Tobacco Induced Diseases, Vol 17, Iss 1 (2019)
Objective We examined how dental hygienists support smoking cessation in their routine regular check-up sessions. In Japan, where primary medical care is provided by universal health insurance (UHI), smoking cessation support is not included in denta
Externí odkaz:
https://doaj.org/article/55de79868f974a988ff915f52102540f
Publikováno v:
Journal of Diabetes Research, Vol 2019 (2019)
Type 1 diabetes (T1D) is an autoimmune disease caused by the destruction of pancreatic β cells by autoantigen-reactive diabetogenic cells. Antigen-specific therapies using islet autoantigens for restoring immune tolerance have emerged as promising a
Externí odkaz:
https://doaj.org/article/3a33de59e5914dbeb4b7ad6ad7280e04
Publikováno v:
Journal of Diabetes Research
Journal of Diabetes Research, Vol 2019 (2019)
Journal of Diabetes Research, Vol 2019 (2019)
Type 1 diabetes (T1D) is an autoimmune disease caused by the destruction of pancreatic β cells by autoantigen-reactive diabetogenic cells. Antigen-specific therapies using islet autoantigens for restoring immune tolerance have emerged as promising a
Reality and possibility of smoking cessation support and collaborative approach by dental hygienists
Publikováno v:
Tobacco Induced Diseases, Vol 17, Iss 1 (2019)
Objective We examined how dental hygienists support smoking cessation in their routine regular check-up sessions. In Japan, where primary medical care is provided by universal health insurance (UHI), smoking cessation support is not included in denta
Autor:
Hideo Kawahara, Jun-ichi Masuda, Akira Uehama, null Henry H Takei, null Perry R Klokkevold, null Anthony Rowley, Hiroshi Kawadu, Takashi Watanabe, Yoshio Ishizaka, Kayoko Yui, Hidetoshi Akimoto, Yoshihiro Natsumi
Publikováno v:
THE JOURNAL OF THE ACADEMY OF CLINICAL DENTISTRY. 35:249-258
Autor:
Eiko Yamamoto, Fumitaka Kikkawa, Akihiro Nawa, Kiyosumi Shibata, Tetsuro Nagasaka, Tomoko Inaba, Osamu Takikawa, Hiroaki Kajiyama, Hidetoshi Akimoto, Kazuhiko Ino
Publikováno v:
Gynecologic Oncology. 115:185-192
Objective Indoleamine 2,3-dioxygenase (IDO) is a tryptophan-catabolizing enzyme that induces immune tolerance. The purpose of the present study is to investigate IDO expression and its functional role in ovarian cancer cells in vitro and in vivo . Me
Publikováno v:
Journal of Neurochemistry. 110:791-800
Indoleamine 2,3-dioxygenase (IDO) is the rate-limiting enzyme of the kynurenine pathway of tryptophan metabolism, ultimately leading to production of the excitotoxin quinolinic acid (QUIN) by monocytic cells. In the Tg2576 mouse model of Alzheimer's
Publikováno v:
Seibutsu Butsuri. 49:070-074
Imaging is an essential approach to understanding the physiological systems of organism. Luminescence imaging based on luciferase technology reveals biological phenomena from molecule to body by light. In this manuscript, we describe the characterist
Autor:
Hidetoshi Akimoto, Osamu Takikawa, Kazuhiko Ino, Hiroaki Kajiyama, Mikio Terauchi, Akihiro Nawa, Yoshiyuki Ishida, Fumitaka Kikkawa, Norio Yoshida, Ken-ichi Isobe, Kiyosumi Shibata, Eiko Yamamoto
Publikováno v:
Clinical Cancer Research. 14:7251-7259
Purpose: Indoleamine 2,3-dioxygenase (IDO) is a tryptophan-catabolizing enzyme that induces immune tolerance in mice. Our prior study showed that high tumoral IDO expression in endometrial cancer tissues correlates with disease progression and impair
Autor:
Fumihiko Nagase, Nanako Ogasawara, Hidetoshi Akimoto, Rie Hiramatsu, Tsutomu Kawabe, Ken-ichi Isobe, Toshiaki Hara, Osamu Takikawa
Publikováno v:
Immunology Letters. 116:95-102
Indoleamine 2,3-dioxygenase (IDO)-initiated tryptophan metabolism along the kynurenine (Kyn) pathway in some dendritic cells (DC) such as plasmacytoid DC (pDC) regulates T-cell responses. It is unclear whether bone marrow-derived myeloid DC (BMDC) ex