Zobrazeno 1 - 10
of 29
pro vyhledávání: '"Hernan Concha Quezada"'
Autor:
Rui-Sheng Duan, Zhiguo Chen, Lei Bao, Hernan Concha Quezada, Inger Nennesmo, Bengt Winblad, Jie Zhu
Publikováno v:
Neurobiology of Disease, Vol 16, Iss 3, Pp 630-637 (2004)
Experimental autoimmune neuritis (EAN) is an inflammatory autoimmune demyelinating disease of peripheral nervous system (PNS) and represents an animal model of Guillain–Barré syndrome (GBS) in man. The inflammatory cell infiltrating into the PNS i
Externí odkaz:
https://doaj.org/article/bcf494bddf324272ba445a58837acbbb
Publikováno v:
Mediators of Inflammation, Vol 2014 (2014)
The Guillain-Barré syndrome (GBS) is considered a T helper 1 (Th1) cells-mediated acute inflammatory peripheral neuropathy. However, some changes in GBS could not be explained completely by Th1 cells pathogenic role. Recently, Th17 cells have been i
Externí odkaz:
https://doaj.org/article/88be69908a15463ca3a766e5d117d01e
Autor:
Hong-Liang Zhang, Mohammed Y Hassan, Xiang-Yu Zheng, Sheikh Azimullah, Hernan Concha Quezada, Naheed Amir, Mohamed Elwasila, Eilhard Mix, Abdu Adem, Jie Zhu
Publikováno v:
PLoS ONE, Vol 7, Iss 5, p e38157 (2012)
The role of tumor necrosis factor (TNF)-α and its receptors in neuroautoimmune and neuroinflammatory diseases has been controversial. On the basis of our previous studies, we hereby aimed to further clarify TNF-α's mechanism of action and to explor
Externí odkaz:
https://doaj.org/article/7ddbbb8321b942e1954c847271607e2a
Autor:
Hernán Concha Quezada
Este libro nos da una visión acabada de la privilegiada y enaltecida vida profesional e intelectual del renombrado fisiólogo y médico cardiólogo Georg Friedrich Nicolai (1874-1964) antes de la Primera Guerra Mundial, previo a su emigración oblig
Autor:
Aino Fianu Jonasson, Cecilia Götherström, Outi Hovatta, Hernan Concha Quezada, Åsa Ekblad, Mohammed Saliem, Shahla Hamza Al-Saqi
Publikováno v:
Cell and tissue banking. 20(2)
In the original article, Fig. 1A was by mistakenly duplicated. The corrected image is provided in this correction article.
Autor:
Anna Kats, Hernan Concha Quezada, Göran Andersson, Zenebech Wondimu, Maria Norgård, Tülay Yucel-Lindberg, Catalin Koro
Publikováno v:
Journal of Cellular and Molecular Medicine. 20:1128-1138
Periodontitis is characterized by chronic inflammation and osteoclast-mediated bone loss regulated by the receptor activator of nuclear factor-κB (RANK), RANK ligand (RANKL) and osteoprotegerin (OPG). The aim of this study was to investigate the eff
Autor:
Jie Zhu, Hong-Liang Zhang, Dan Wang, Hernan Concha Quezada, Bo Zhang, Tao Jin, Wei Zhu, Hong Yu
Publikováno v:
Immunology Letters. 171:15-25
Background In the present study, we investigated the immuno-regulatory and therapeutic effects of Bowman–Birk inhibitor concentrate (BBIC) on experimental autoimmune neuritis (EAN), an animal model of Guillain–Barre syndrome (GBS) in human. Metho
Publikováno v:
Mediators of Inflammation
Mediators of Inflammation, Vol 2014 (2014)
Mediators of Inflammation, Vol 2014 (2014)
The Guillain-Barré syndrome (GBS) is considered a T helper 1 (Th1) cells-mediated acute inflammatory peripheral neuropathy. However, some changes in GBS could not be explained completely by Th1 cells pathogenic role. Recently, Th17 cells have been i
Autor:
Claes Lindberg, Leif Jansson, Anders Gustafsson, Karin Näsström, Pierre Georgsson, Jörgen Jönsson, Anna Kats, Hernan Concha Quezada, Tülay Yucel-Lindberg, Tove Båge
Publikováno v:
The FASEB Journal
The potent inflammatory mediator prostaglandin E2 (PGE2) is implicated in the pathogenesis of several chronic inflammatory conditions, including periodontitis. The inducible enzyme microsomal prostaglandin E synthase-1 (mPGES-1), catalyzing the termi
Autor:
Ke-Jia Zhang, Xing-Mei Zhang, Xiang-Yu Zheng, Duo Zhang, Jie Zhu, Hong-Liang Zhang, Hernan Concha Quezada
Publikováno v:
Neuroscience Letters. 499:175-180
Previously, we reported that apolipoprotein E (apoE) deficiency increased the susceptibility to experimental autoimmune neuritis (EAN), an inflammatory autoimmune disorder of the peripheral nervous system (PNS) and an animal model for human Guillain-