Zobrazeno 1 - 10
of 115
pro vyhledávání: '"Heras‐Garvin, A."'
Autor:
Nicholson, L., Piras, I.S., DeBoth, M.D., Siniard, A., Heras-Garvin, A., Stefanova, N., Huentelman, M.J.
Publikováno v:
In Brain Research 1 July 2024 1834
Publikováno v:
In Parkinsonism and Related Disorders April 2020 73:94-104
Autor:
Miguel Lemos, Serena Venezia, Violetta Refolo, Antonio Heras-Garvin, Sabine Schmidhuber, Armin Giese, Andrei Leonov, Sergey Ryazanov, Christian Griesinger, Gergana Galabova, Guenther Staffler, Gregor Karl Wenning, Nadia Stefanova
Publikováno v:
Translational Neurodegeneration, Vol 9, Iss 1, Pp 1-14 (2020)
Abstract Background Misfolded oligomeric α-synuclein plays a pivotal role in the pathogenesis of α-synucleinopathies including Parkinson’s disease and multiple system atrophy, and its detection parallels activation of microglia and a loss of neur
Externí odkaz:
https://doaj.org/article/ef88796be9ef4ed0aba14c1021ca1753
Autor:
Sabine Eschlböck, Margarete Delazer, Florian Krismer, Thomas Bodner, Alessandra Fanciulli, Beatrice Heim, Antonio Heras Garvin, Christine Kaindlstorfer, Elfriede Karner, Katherina Mair, Christoph Rabensteiner, Cecilia Raccagni, Klaus Seppi, Werner Poewe, Gregor K. Wenning
Publikováno v:
Annals of Clinical and Translational Neurology, Vol 7, Iss 2, Pp 219-228 (2020)
Abstract Objective Cognitive impairment in multiple system atrophy (MSA) is common, but remain poorly characterized. We evaluated cognitive and behavioral features in MSA patients and assessed between‐group differences for MSA subtypes and the effe
Externí odkaz:
https://doaj.org/article/1dae767cd9a640999f2f8d7c16b483e9
Autor:
Herrera-Vaquero, Marcos, Bouquio, Danielle, Kallab, Martin, Biggs, Karl, Nair, Gayatri, Ochoa, Jessica, Heras-Garvin, Antonio, Heid, Christian, Hadrovic, Inesa, Poewe, Werner, Wenning, Gregor K., Klärner, Frank-Gerrit, Schrader, Thomas, Bitan, Gal, Stefanova, Nadia
Publikováno v:
In BBA - Molecular Basis of Disease 1 November 2019 1865(11)
Publikováno v:
Frontiers in Neurology, Vol 12 (2021)
In the past few years, an increasing amount of studies primarily based on experimental models have investigated the existence of distinct α-synuclein strains and their different pathological effects. This novel concept could shed light on the hetero
Externí odkaz:
https://doaj.org/article/66edead2209e433cba733f7531c03ae0
Publikováno v:
Journal of Neuroinflammation, Vol 17, Iss 1, Pp 1-14 (2020)
Abstract Aim Pre-clinical studies in models of multiple sclerosis and other inflammatory disorders suggest that high-salt diet may induce activation of the immune system and potentiate inflammation. However, high-salt diet constitutes a common non-ph
Externí odkaz:
https://doaj.org/article/746635ac98db42eba76fb2a77d87e000
Autor:
Antonio Heras-Garvin, Nadia Stefanova
Publikováno v:
Frontiers in Synaptic Neuroscience, Vol 12 (2020)
Since its discovery 30 years ago, α-synuclein (α-syn) has been one of the most studied proteins in the field of neuroscience. Dozens of groups worldwide have tried to reveal not only its role in the CNS but also in other organs. α-syn has been lin
Externí odkaz:
https://doaj.org/article/929043c815964775b124f4ce468f4304
Autor:
Herrera‐Vaquero, M.1 (AUTHOR), Heras‐Garvin, A.1 (AUTHOR), Krismer, F.1 (AUTHOR), Deleanu, R.2 (AUTHOR), Boesch, S.1 (AUTHOR), Wenning, G. K.1 (AUTHOR), Stefanova, N.1 (AUTHOR) Nadia.Stefanova@i-med.ac.at
Publikováno v:
Neuropathology & Applied Neurobiology. Feb2021, Vol. 47 Issue 2, p268-282. 15p.
Autor:
Adrian Viehweger, Almudena Gerpe, Manuel A. Sanchez-Garcia, Maria V. Sanchez-Mico, Alberto Pascual, Angela Gomez-Arboledas, José López-Barneo, Jose Carlos Davila, Javier Vitorica, Alicia E. Rosales-Nieves, Marisa Vizuete, Victoria Navarro, Edurne Berra, Antonio Heras-Garvin, Nieves Lara-Ureña, Rosana March-Díaz, Carmen Romero-Molina, Cristina Forja, Elisabeth Sanchez-Mejias, Antonia Gutierrez, Maria I. Alvarez-Vergara, Clara Ortega-de San Luis, Tammie Bishop, Emma J. Hodson, Alberto Serrano-Pozo
Publikováno v:
Digital.CSIC. Repositorio Institucional del CSIC
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Genetic Alzheimer’s disease (AD) risk factors associate with reduced defensive amyloid β plaque-associated microglia (AβAM), but the contribution of modifiable AD risk factors to microglial dysfunction is unknown. In AD mouse models, we observe c