Zobrazeno 1 - 10
of 3 578
pro vyhledávání: '"Hepatic ischemia"'
Autor:
Yi Xiong, Jiawen Chen, Wei Liang, Kun Li, Yingqi Huang, Jingwen Song, Baoyu Zhang, Xiusheng Qiu, Dongbo Qiu, Qi Zhang, Yunfei Qin
Publikováno v:
Journal of Translational Medicine, Vol 22, Iss 1, Pp 1-16 (2024)
Abstract Background Liver surgery during the perioperative period often leads to a significant complication known as hepatic ischemia-reperfusion (I/R) injury. Hepatic I/R injury is linked to the innate immune response. The cGAS-STING pathway trigger
Externí odkaz:
https://doaj.org/article/036a65c91ca34544a116b3ba95110ba0
Publikováno v:
Iranian Journal of Basic Medical Sciences, Vol 27, Iss 10, Pp 1323-1330 (2024)
Objective(s): Hepatic ischemia-reperfusion (HIR) is a severe process in pathophysiology that occurs clinically in hepatectomy, and hepatic transplantations. The present study aimed to investigate the effect of PKC θ deletion against HIR injury and e
Externí odkaz:
https://doaj.org/article/61518b881be149e89fec08374aecf8f7
Autor:
ZHANG Xiangxin, LI Wen
Publikováno v:
Jichu yixue yu linchuang, Vol 44, Iss 6, Pp 877-881 (2024)
In case of hepatic ischemia-reperfusion injury (HIRI), mitophagy is induced by multiple pathways such as PINK1/Parkin and mitophagy receptor, which is closely related to mitochondrial dynamic related protein. Mitophagy is a double-edged sword. Modera
Externí odkaz:
https://doaj.org/article/bfb844eafda54c42b8e1f2e698f81e9e
Publikováno v:
Molecular Biomedicine, Vol 5, Iss 1, Pp 1-17 (2024)
Abstract Hepatic ischemia–reperfusion injury (HIRI) is a critical pathophysiological process during liver transplantation (LT). Multiple genes and signal pathways are dysregulated during HIRI. This study aims to identify genes as potential therapeu
Externí odkaz:
https://doaj.org/article/3ebd386c312c4a66add634bc85c6e4e3
Publikováno v:
Molecular Medicine, Vol 30, Iss 1, Pp 1-14 (2024)
Abstract Background Programmed cell death is an important mechanism for the development of hepatic ischemia and reperfusion (IR) injury, and multiple novel forms of programmed cell death are involved in the pathological process of hepatic IR. ERRFI1
Externí odkaz:
https://doaj.org/article/4511074bd11c4c1892009c23b54c4e9f
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-13 (2024)
Abstract 6-Gingerol, the main bioactive compound of ginger, has antioxidant, anti-inflammatory, anti-cancer and neuroprotective effects. However, it is unclear whether 6-Gingerol has protective effects against hepatic ischemia/reperfusion (I/R) injur
Externí odkaz:
https://doaj.org/article/b2095ab35f3b453b98fb348024ec64b3
Autor:
Peng Jiang, Xinqiang Li, Yuntai Shen, Lijian Luo, Bin Wu, Dahong Teng, Jinshan Wang, Imran Muhammad, Qingguo Xu, Shipeng Li, Bin Zhang, Jinzhen Cai
Publikováno v:
Heliyon, Vol 10, Iss 16, Pp e34385- (2024)
Hepatic ischemia‒reperfusion injury is a common injury in liver surgery and liver transplantation that can lead to liver function damage, including oxidative stress, apoptosis, autophagy and inflammatory reactions. Pyroptosis is a type of inflammat
Externí odkaz:
https://doaj.org/article/e1a4a10bb933443daa923cfc1c75c643
Publikováno v:
BMC Anesthesiology, Vol 24, Iss 1, Pp 1-13 (2024)
Abstract Background Animal experiments have confirmed that remote ischemic preconditioning (RIPC) can reduce hepatic ischemia-reperfusion injuries (HIRIs), significantly improving early tissue perfusion and oxygenation of the residual liver after res
Externí odkaz:
https://doaj.org/article/139e214b10324d59a41607b42bb8b055
Autor:
Leiyi Zhang, Xiaoxiang Gong, Juan Tan, Rongsen Zhang, Mingxia Li, Cong Liu, Chenhao Wu, Xiaojing Li
Publikováno v:
Biology Direct, Vol 19, Iss 1, Pp 1-13 (2024)
Abstract Background This study seeks to investigate the impacts of Lactobacillus reuteri (L. reuteri) on hepatic ischemia-reperfusion (I/R) injury and uncover the mechanisms involved. Methods Mice in the I/R groups were orally administered low and hi
Externí odkaz:
https://doaj.org/article/ff0395ea685f4264a1ce1c6185885db6
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-15 (2024)
Abstract Hepatic ischemia–reperfusion injury (HIRI) elicits an immune-inflammatory response that may result in hepatocyte necrosis and apoptosis, ultimately culminating in postoperative hepatic dysfunction and hepatic failure. The precise mechanism
Externí odkaz:
https://doaj.org/article/88f5e7d84dec46bdaf6a0eb60a069123