Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Henrik Zhang"'
Autor:
Henrik Zhang, Thomas Tu
Publikováno v:
Clinical and Molecular Hepatology, Vol 28, Iss 2, Pp 135-149 (2022)
Chronic hepatitis B is a major cause of liver disease worldwide and is currently incurable. Hepatitis B virus (HBV) covalently closed circular (ccc) DNA is a key form of the virus responsible for its persistence and is the transcriptional template fo
Externí odkaz:
https://doaj.org/article/8990a3bfddab4b2c94fa30de2e6bfbd8
Autor:
Thomas Tu, Benno Zehnder, Jochen M. Wettengel, Henrik Zhang, Sally Coulter, Vikki Ho, Mark W. Douglas, Ulrike Protzer, Jacob George, Stephan Urban
Publikováno v:
JHEP Reports, Vol 4, Iss 9, Pp 100514- (2022)
Background & Aims: The chronicity of HBV (and resultant liver disease) is determined by intrahepatic persistence of the HBV covalently closed circular DNA (cccDNA), an episomal form that encodes all viral transcripts. Therefore, cccDNA is a key targe
Externí odkaz:
https://doaj.org/article/e6b22226bbb1439cb89ce9fb34e77cf5
Autor:
Henrik Zhang, Thomas Tu
Publikováno v:
Hepatology Communications, Vol 5, Iss 10, Pp 1629-1631 (2021)
Externí odkaz:
https://doaj.org/article/74c3e55407b6434eacc0fee26f7456c1
Publikováno v:
Viruses, Vol 13, Iss 2, p 180 (2021)
Hepatitis B virus (HBV) is a globally-distributed pathogen and is a major cause of liver disease. HBV (or closely-related animal hepadnaviruses) can integrate into the host genome, but (unlike retroviruses) this integrated form is replication-defecti
Externí odkaz:
https://doaj.org/article/c10cc7f3255743ffa41b634d978918c3
Autor:
Thomas Tu, Henrik Zhang
Publikováno v:
Hepatology. 76:15-17
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4edfff691f48146b03ab41c223ad5ce6
https://doi.org/10.1002/hep.32388
https://doi.org/10.1002/hep.32388
Autor:
Thomas Tu, Henrik Zhang
Publikováno v:
Hepatology Communications, Vol 5, Iss 10, Pp 1629-1631 (2021)
Hepatology Communications
Hepatology Communications
Integrated hepatitis B virus (HBV) DNA, found in more than 85% of HBV-associated hepatocellular carcinomas (HBV-HCCs), can play a significant role in HBV-related liver disease progression. HBV-host junction sequences (HBV-JSs), created through integr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8ee32a755325bbe22bb1824ab1045cdd
https://doaj.org/article/74c3e55407b6434eacc0fee26f7456c1
https://doaj.org/article/74c3e55407b6434eacc0fee26f7456c1
Autor:
Thomas Tu, Benno Zehnder, Jochen Wettengel, Henrik Zhang, Sally Coulter, Vikki Ho, Mark Douglas, Ulrike Protzer, Jacob George, Stephan Urban
Chronic hepatitis B is a major cause of liver failure and liver cancer, resulting in 600,000 annual deaths globally. Infection chronicity (and resultant liver disease) is determined by intrahepatic persistence of the viral covalently closed circular
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::755f50da7dee198717b584d09ab6ddaa
https://doi.org/10.21203/rs.3.rs-1325894/v1
https://doi.org/10.21203/rs.3.rs-1325894/v1
Autor:
Thomas Tu, Henrik Zhang
Publikováno v:
Clinical and molecular hepatology. 28(2)
Chronic hepatitis B is a major cause of liver disease worldwide and is currently incurable. Hepatitis B virus (HBV) covalently closed circular (ccc) DNA is a key form of the virus responsible for its persistence and is the transcriptional template fo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::43994cedc02c4b5b44a42b0f03e07799
https://pubmed.ncbi.nlm.nih.gov/34674513
https://pubmed.ncbi.nlm.nih.gov/34674513
Publikováno v:
Viruses, Vol 13, Iss 180, p 180 (2021)
Viruses
Viruses
Hepatitis B virus (HBV) is a globally-distributed pathogen and is a major cause of liver disease. HBV (or closely-related animal hepadnaviruses) can integrate into the host genome, but (unlike retroviruses) this integrated form is replication-defecti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::788c161d484399467c8e2284e02358aa
https://www.mdpi.com/1999-4915/13/2/180
https://www.mdpi.com/1999-4915/13/2/180