Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Henriette, Undeutsch"'
Autor:
Ralf Paschke, Fu-Ping Zhang, Christoffer Löf, Holger Jaeschke, Jukka Kero, Matti Poutanen, Jorma Toppari, Meeri Jannari, Kristiina Makkonen, Henriette Undeutsch, Moosa Khalil, Konrad Patyra, Markus Eszlinger
Publikováno v:
Thyroid : official journal of the American Thyroid Association. 28(10)
Constitutively active thyrotropin receptor (TSHR) mutations are the most common etiology of non-autoimmune hyperthyroidism (NAH). Thus far, the functionality of these mutations has been tested in vitro, but the in vivo models are lacking.To understan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d4e272cef38dc54402eff8bb958b3b0d
https://pubmed.ncbi.nlm.nih.gov/30132406
https://pubmed.ncbi.nlm.nih.gov/30132406
Autor:
Konrad, Patyra, Holger, Jaeschke, Christoffer, Löf, Meeri, Jännäri, Suvi T, Ruohonen, Henriette, Undeutsch, Moosa, Khalil, Andreina, Kero, Matti, Poutanen, Jorma, Toppari, Min, Chen, Lee S, Weinstein, Ralf, Paschke, Jukka, Kero
Publikováno v:
FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
Thyroid function is controlled by thyroid-stimulating hormone (TSH), which binds to its G protein-coupled receptor [thyroid-stimulating hormone receptor (TSHR)] on thyrocytes. TSHR can potentially couple to all G protein families, but it mainly activ
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::ad08e5853b1a9b8582e0ca9763263be1
https://pubmed.ncbi.nlm.nih.gov/29799790
https://pubmed.ncbi.nlm.nih.gov/29799790
Autor:
Meeri Jannari, Holger Jaeschke, Christoffer Löf, Suvi T. Ruohonen, Jorma Toppari, Min Chen, Lee S. Weinstein, Ralf Paschke, Konrad Patyra, Jukka Kero, Henriette Undeutsch
Publikováno v:
Endocrine Abstracts.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2096a0291a2168c2d17e87cfab192327
https://doi.org/10.1530/endoabs.49.oc4.2
https://doi.org/10.1530/endoabs.49.oc4.2
Publikováno v:
genesis. 52:333-340
We have created a mouse model expressing tamoxifen-inducible Cre recombinase (CreER(T2) ) under the control of the thyroglobulin (Tg) gene promoter to be able to study the role of defined genetic modifications in the regulation of thyroid function. W
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::18a9df03611094ee062e922544025a2d
https://doi.org/10.1002/dvg.22740
https://doi.org/10.1002/dvg.22740
Autor:
Marja Ojaniemi, Teemu Kuulasmaa, Jukka Kero, Krista Rantakari, Jaakko Ignatius, Juha-Pekka Pursiheimo, Jorma Toppari, Christoffer Löf, Henriette Undeutsch, Jarmo Jääskeläinen, Markku Laakso, Konrad Patyra, Heike Biebermann, Tuulia Pajunen, Gunnar Kleinau, Taina Mustila, Heiko Krude, Jagadish Vangipurapu, Andreina Kero, Päivi J. Miettinen, Turkka Kirjavainen, Peter Kühnen, Holger Jaeschke
Publikováno v:
Thyroid
Background: Congenital hypothyroidism (CH) is defined as the lack of thyroid hormones at birth. Mutations in at least 15 different genes have been associated with this disease. While up to 20% of CH cases are hereditary, the majority of cases are spo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::178eb13c0d0bc3c4ff9ae436f667f551
https://pubmed.ncbi.nlm.nih.gov/27373559
https://pubmed.ncbi.nlm.nih.gov/27373559
Publikováno v:
Experimental and Clinical Endocrinology & Diabetes. 122
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::098c4c4876d9cfef5a2687fd704d78d6
https://doi.org/10.1055/s-0035-1547750
https://doi.org/10.1055/s-0035-1547750
Publikováno v:
Genesis (New York, N.Y. : 2000). 52(4)
We have created a mouse model expressing tamoxifen-inducible Cre recombinase (CreER(T2) ) under the control of the thyroglobulin (Tg) gene promoter to be able to study the role of defined genetic modifications in the regulation of thyroid function. W
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::77895eda9cfacc062e86a81a80ec39c6
https://pubmed.ncbi.nlm.nih.gov/24395757
https://pubmed.ncbi.nlm.nih.gov/24395757